4: Spinal cord compression Flashcards

1
Q

Which

a) ascending pathways
b) descending pathways

are found in the spinal cord?

A

Important ones (for me anyway) are:

a) DC/ML pathway (fine touch, proprioception, vibration), Spinothalamic tract (firm touch, temperature, pain)

b) Corticospinal tracts (voluntary movement)

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2
Q

Which two tracts does the corticospinal tract split into?

A

Anterior corticospinal tract (15% of fibres, cross at spinal cord level of muscle)

Lateral corticospinal tract (85% of fibres, have already crossed over at medulla)

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3
Q

Which structures are connected by the upper motor neurons of the corticospinal tract?

A

Motor cortex TO Anterior horn of spinal cord

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4
Q

Which structures are connected by the lower motor neurons of the corticospinal tract?

A

Anterior horn of spinal cord TO Skeletal muscle

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5
Q

Where do 85% of corticospinal tract fibres decussate?

A

Pyramids of the medulla

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6
Q

Compare muscle tone in UMN versus LMN problems.

A

UMN - hypertonia

LMN - hypotonia

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7
Q

Does atrophy tend to occur in UMN or LMN problems?

A

LMN

muscle can’t contract, so atrophies due to underuse

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8
Q

In which type of motor neuron pathology are fasciculations seen?

A

LMN

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9
Q

Describe reflexes in

a) UMN
b) LMN problems.

A

a) Brisk

b) Slow / absent

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10
Q

In which type of motor neuron problem do you see the Babinski reflex?

What does this look like?

A

UMN disease

When plantar surface of the foot is stroked, toes extend and fan out (abnormal, they should flex)

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11
Q

Describe upper motor neuron disease in terms of

a) reflexes
b) atrophy
c) fasciculations
d) muscle tone?

A

In UMN disease:

a) brisk reflexes
b) little / no atrophy
c) no fasciculations
d) increased muscle tone

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12
Q

Describe lower motor neuron disease in terms of

a) reflexes
b) atrophy
c) fasciculations
d) muscle tone?

A

In LMN disease:

a) Slow / absent reflexes
b) Marked atrophy
c) Fasciculations present
d) Decreased muscle tone

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13
Q

Which sensations are carried by the spinothalamic tract?

A

Firm touch

Temperature

Pain

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14
Q

Where does the spinothalamic tract decussate?

A

Spinal cord

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15
Q

Where does the DC/ML pathway decussate?

A

Medulla

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16
Q

What can happen to the spinal cord to cause disease?

A

Compression

Transection

17
Q

What are some acute causes of spinal cord compression?

A

Trauma

Disc prolapse

Tumour

Infection

Haemorrhage into cord / subarachnoid space

18
Q

What are some chronic causes of spinal cord compression?

A

Spondylosis - degenerative spinal diseases

Tumour

Arthritis (especially RA)

19
Q

Why is it important to know where the motor and sensory pathways cross over in terms of spinal cord injuries?

A

If you know where the tract is in the spinal cord

i.e DC/ML pathway and corticospinal tracts will be ipsilateral, spinothalamic tract will already have crossed to be contralateral

You can predict what will be affected by spinal cord injuries

20
Q

Which pathways are affected by a cord transection?

A

All of them

Sensory and motor symptoms

21
Q

Sensory AND motor innervations are totally disrupted bilaterally, below the lesion by which type of spinal cord injury?

A

Complete cord transection

22
Q

What occurs acutely below the level of a spinal cord injury?

A

Spinal shock

23
Q

Describe spinal shock.

A

Loss of reflexes below the level of an acute spinal cord injury

can occur in injuries like cord transection

24
Q

Spinal shock is a loss of ___ below the level of a spinal cord injury.

Is it permanent?

A

reflexes

usually not

25
What type of **spinal injury** causes **Brown-Sequard syndrome?**
**Spinal cord hemisection** Damage to one side of the spinal cord
26
Think about where the **crossing over** of the **DC/ML, spinothalamic** and **corticospinal tracts** occur. If a patient suffers a **cord hemisection**, what **symptoms** will they have and what **side** will each symptom be felt on relative to the lesion?
**Brown-Sequard syndrome:** **IPSILATERAL loss of fine touch, proprioception and vibration** (DC/ML runs ipsilateral to skin it supplies and decussates at medulla) **CONTRALATERAL loss of firm touch, temperature and pain** (spinothalamic tract decussates at spinal level and runs contralaterally to skin it supplies, upwards) **IPSILATERAL** **loss of motor function** (corticospinal tracts decussate at medulla and run down the spinal cord, ipsilateral to the muscles they control)
27
What type of injury causes **central cord syndrome**?
**Hyperflexion** **Hyperextension** neck injuries
28
Which pathway is primarily affected by **central cord syndrome**?
**Spinothalamic tract** Firm touch, temperature, pain
29
What sort of **distribution** does sensory disturbance often have in patients with **central cord syndrome**? Which tract is involved, and which senses are lost?
**Cape** distribution **Spinothalamic tract** (firm touch, temperature, pain)
30
What is the difference between the **clinical presentation** of **acute and chronic cord compression**?
**Chronic** is the same but also has **UMN signs** (increased tone, hyperreflexia, upgoing plantars, clonus)
31
High-\_\_\_ trauma can cause spinal cord compression.
**high-energy**
32
What disease causes **insidious cord compression?**
**CNS cancer** - e.g meningiomas, Schwannomas, astrocytomas, ependymomas
33
How may **CNS tumours** cause cord compression acutely?
**Haemorrhage** **?Collapse**
34
Which spinal disease, causing **neurogenic claudication**, can also cause **spinal cord compression**?
**Spinal stenosis**
35
Pus-forming bacteria can form ___ which compress the spinal cord.
**abscesses**
36
How is suspected **spinal cord compression** investigated?
**Imaging** X-ray, CT or MRI scan
37
How is **spinal cord compression** managed surgically?
**Decompression** (draining of fluid, removal of tumour, clearing up basically) +/- **Traction** (use of a device to straighten the bones involved) +/- **Fixation** (use of screws, rods etc. to keep broken bones held together, internally or externally) +/- Antibiotics, chemo/radiotherapy