Hemodynamic disorders Flashcards

1
Q

Mechanism of fluid accumulation in heart failure

A

increase in hyrdrostatic pressure

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2
Q

Mechanism of fluid accumulation in nephrotic syndrome

A

decrease in oncotic pressure

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3
Q

Mechanism of fluid accumulation in burns and infections

A

increase vascular permeability

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4
Q

Mechanism of fluid accumulation post MRM

A

lymphatic obstruction

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5
Q

During chronic congestion, macrophages phagocytose RBCs leading to this cell type (hemosiderin-laden)

A

heart failure cells

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6
Q

____ (acute, chronic) congestion wherein the central vein and sinusoids are distended with centrilobular ischemic necrosis and periportal fatty changes

A

acute congestion

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7
Q

Steps in hemostasis

A
  1. Arteriolar vasoconstriction
  2. Primary hemostasis
  3. Secondary hemostasis
  4. Clot stabilization and resorption
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8
Q

Deficiency in GPIIb-IIIa

A

Glanzman Thrombastenia

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9
Q

Deficiency in GP1B

A

Bernard-Soulier Syndrome

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10
Q

Most common cause of hypercoaguability due to resistance in Activated Protein C

A

Factor V Leiden

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11
Q

Presence of lines of zahn suggests ___ thrombosis

A

arterial

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12
Q

Condition that is characterized as thrombosis with thrombocytopenia

A

APAS

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13
Q

Most common source of pulmonary embolism

A

DVT

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14
Q

Most common source of systemic thromboembolism

A

mural thrombi arising from LV infarcts

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15
Q

___ mL of air can result in symptomatic air embolism

A

100cc

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16
Q

___ disease: ischemia of femoral head, tibia, and humerus; seen in decompression sickness

A

Caisson disease

17
Q

type of infarct seen in solid organs and end-arterial circulation like heart, spleen, kidney

A

white infarct

18
Q

[Stage of shock]

presence of tachycardia with peripheral vasoconstriction and renal fluid vasoconstriction

A

non-progressive

basically, reflex compensatory mechanisms are intact, perfusion of vital organs are maintained

19
Q

[stage of shock]

Onset of lactic acidosis due to tissue hypoperfusion

A

progressive

20
Q

[type of shock]

increased PCWP, low CO, high SVR

A

cardiogenic

21
Q

[type of shock]

decreased PCWP, increased CO, decreased SVR

A

distributive