altitude Flashcards

1
Q

low altitude

A

500-2000m leth, calg, denver

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2
Q

mod altitude

A

2000-3000 mt temple, right outside lake louise

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3
Q

high altitude

A

3000-5500m pikes peak

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4
Q

extreme altitude

A

> 5500, mt everest

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5
Q

why do you get mountain sickness in planes

A

because they keep it at 2000m

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6
Q

Partial pressure of oxygen and altitude

A

decrease as latter increases

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7
Q

mt everest

1921, 1924, 1953, 1978

A

mallory - reconnaissance (7020)
mallory and irving - dont know
hillary and tensing - first successful summit
messner - first summit w/o o2

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8
Q

arterial blood on mt everest

A

little oxygenation - 30mmhg in femoral blood

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9
Q

death zone

A

8000 metres - wont be able to survive

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10
Q

rob hall

A

commercialization - competition, survived the night in death zone till the next day till 530 pm

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11
Q

hypobaric hypoxia

A

lower total pressure - go to everest without going to everest - expensive and challenging because of the pressure diff inside and outside of the chamber

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12
Q

normobaric hypoxia

A

environment made hypoxia by reducing oxygen in the air (% of gases)

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13
Q

Haig Glacier Becky Scott High performance training centre

A

2700m

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14
Q

acute changes at altitude of arterial blood

A

reduced oxygenation of arterial blood
decreased PAO2 - decreased SaO2
decreased PAO2 increases time for PAO2 to equilibriate with PaO2

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15
Q

why are highly trained ind not protected from the effects of altitude

A

reduced transit time with exercise further reduces the O2 saturation of arterial blood

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16
Q

how to increase O2 in arterial blood

A

increase minute ventilation

  • decreased SaO2 will lead to an increase in minute ventilation (increased frequency) at rest and during submaximal exercise in order to increase PA O2 and SaO2%
  • hyper ventilation leads to decreased PAcos which decreases ventilatory drive and induces alkalosis,
17
Q

how to increase O2 delivery to tissues

A

increase HR and MAP activation of the SNS
- increase HR at rest and during submax,
-vasoconstricion -increases TPR which contributes to overall increase in MAP-
predisposes to high altitude pulmonary edema and high altitude cerebral edema
- increased production of lactate - more fatigue

18
Q

acute changes at altitude

A

reduced oxygenation of arterial blood
increase VE in order to increase O2 in arterial blood
increased HR and MAP in order to increase O2 delivery to tissues

19
Q

effects of altitude on VO2 max

A

declines by 1% for every 100m increase in altitude (when over 1500m) - pikes peak - lowered by 27%
- everest - lowered by 75%

20
Q

what else does altitude do to aerobic endurance exercise

A

earlier onset of fatigue

21
Q

does altitude affect sprint, strength, and power exercise performace?

A

yes but not to the same extent

22
Q

why might anaerobic exercise performance improve at high altitude

A

they dont require oxygen and theres less pressure/air resistance

23
Q

acclimatization

A

adaptive physiological responses

24
Q

rapid altitude acclimatization

A

INCREASED ARTERIAL OXYGEN CONTENT

INCREASED DELIVERY OF OXYGEN

25
how is the arterial oxygen content brought up
- reduced plasma volme (increased urination) - increasd hematocrit (more ability for O2) and decreased Q - erythropoietin release - stimulates production of RBCs
26
how is the delivery of oxygen increased -2
reduced bicarbonate which increases CO2 (decreased pH) which causes a right shift in the oxyhemoglobin dissociation curve and increases VE increaese in 2,3 DPG (diphosphoglycerate) which is a metabolic by-product of RBCs, causes a right shift, and its beneficial up to 5000m, beyond that SaO2% is impaired
27
4 chronic adaptation to altitude acclimatization
increased VE(VT) increased blood volume increased hematocrit (increased RBC from EPO) reduced diffusion diff
28
how does chronic adaptation work with acclimatization
``` increased myoglobin (increased oxygen carrying capacity of tissues) stimulation of angiogenesis in lungs and muscles ```
29
traditional altitude training
live and train at high altitude benefits: increase in RBC, HGB, Hct, vascularization negative: decrease in plasma volume, @, buffering capacity, increased viscosity of blood - fatigue more readily
30
Live high, train low altitude training
sleep in normobaric hypoxic chamber benefits: no decrease in plasma volume and some athletes benefit from increased hematocrit negatives: less altitude stimulus
31
train high, live low altitude training
rationale - increased training intensity | no clear evidence of increased VO2max
32
overall effect of altitude training depends on 4
ind response, type of training, elevation, duration etc
33
acute mountain sickness
mild altitude sickness that leads to headache, nausea, irritability, weakness, poor appetite, vomiting, tachycardia, disturbed breathing above 2500m: 10-25% between 4500-5500: 50-85% takes 1-3 days to resolve
34
6 risk factors for AMS
history less than 46 yrs of age - brain atrophy - more room for edema female- women have more migraines history for migraines genetics - hypoxic ventilatory response - CV fitness is not protective - only more efficient rapid ascent
35
how do you adapt to AMS
you cant, you go down to recover
36
high altitude cerebral edema - 3 symptoms and compromise of?
severe altitude sickness characterized by swelling in the brain due to increased BP and permeability of the barrier - non-responsive headache and vomiting - confusion, ataxia, cognitive issues - drowsiness, unconsciousness, brain herniation, death endothelial permeability
37
high altitude pulmonary edema | 2 symptoms and possible?
severe altitude sickness characterized by edema and fluid leakage in the lungs due to increased pulmonary BP and permeability of the vascular endothelium - low exercise capacity, labored breathing, coughing, low SaO2%, cyanosis (turning blue) - gurgling, pink, frothy sputum - can lead to death if not treated
38
gamow bag
used to treat altitude sickness