brain lab briefing Flashcards

1
Q

describe the layers of the scalp

A

Skin: where hair grows, nutrients for hair follicles are found here

Connective tissue: dense subcutaneous layer of fat and fibrous tissue. contains nerves and vessels of scalp.

epicranial Aponeurosis: tough layer of dense connective tissue which runs from frontalis muscle anteriorly to occipitalis posteriorly

Loose connective tissue: areolar connective tissue, made up of random collagen bundles, separates the upper 3 layers of scalp of pericranium, and allows them to move in realign to pericranium.

Periosteum: provides nutrition to the cranium capacity to repair

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2
Q

if there is an infected scalp wound, how is there potential for it to spread intracranially?

A

via emissary veins into cranial cavity

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3
Q

why is the convex shape of the skull important?

A

it dissipates injury by distributing and minimising the effects fo a blow to the head so the head is able to take more force without fracturing

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4
Q

what important structures are found the base of the skull?

A

cranial nerves and blood supply

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5
Q

what bones make up the base of the skull?

A

orbital plate of frontal bone, sphenoid bone, squamous and petrous temporal bones, occipital bone

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6
Q

describe a depressed skull fracture

A

bone fragments depress inwards with damage/compression of brain

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7
Q

describe a linear skull fracture

A

a break in cranial bone resembling a thin line. it occurs at site of impact but the fracture lines radiate away

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8
Q

what is battle’s sign?

A

bruising over the mastoid process (purplish bruising)

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9
Q

what is periorbital ecchymosis also known as?

A

raccoon or panda eyes

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10
Q

what causes panda eyes?

A

basal skull fracture that ruptures meninges and causes venous sinuses to bleed into arachnoid vill and cranial sinuses.

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11
Q

what clinical features are seen in panda eyes beside bruising around the eyes?

A

blood or csf coming out of the ear or nose which indicates breach of the meninges

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12
Q

what diagnostic test should be carried out if panda eyes are present?

A

CT scan

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13
Q

what is the pterion?

A

region of skull behind the temple, where the frontal, parietal, temporal, and sphenoid bones join together.

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14
Q

why is there a high chance of fracture of the pterion and what consequences would a fracture have?

A

It is translucent and very thin

There are branches of meningeal arteries underneath so fracture could lead to intracranial haematoma.

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15
Q

what are the 2 layers of the dura mater and where are they attached and separated?

A

endosteal and meningeal layers

they are closely attached and only separate to form venous sinuses and at reflections

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16
Q

what are the 2 main dural reflections and what do reflections do?

A

falx cerebri and tentroium cerebellum

They divide the cranial cavity into compartments by forming partitions

17
Q

list 3 types of intracranial haematomas

A

epidural/extradural
subdural
intracerebral

18
Q

describe an epidural haematoma including causes and shape

A

When blood collects between periosteal layer of dura and skull, the blood strips the dura away from the periosteum.

Related to a skull fracture, caused by arterial bleeding usually of middle meningeal artery. It forms a lens shaped swelling because it cannot swell along suture lines

19
Q

describe a subdural haematoma

A

when blood collects between dura and arachnoid

Blood is of venous origin = slower bleeding

20
Q

what features can be seen in an acute extradural/epidural haematoma CT scan?

A

lens shaped haematoma
“white” area = fresh blood in context of head injury
ventricular shift - swelling of brain closes the ventricles and shifts the ventricle away from midline

21
Q

what features can be seen in an acute subdural haematoma CT scan?

A

crescent shaped haematoma as there are no suture limitations so blood can spread all the way around the brain
middling shift of ventricles

22
Q

what re the 3 components of intracranial volume?

A

blood, csf, brain

23
Q

what happens in response to raised cranial pressure due to presence of a mass? what happens I this mass keeps increasing?

A

compensatory mechanisms eg removing venous blood and csf

after a critical point, there is decompensation which causes ICP to rise rapidly

24
Q

what is the equation for cerebral perfusion pressure?

A

CPP = MAP - ICP

25
how does a normal brain auto regulate its blood flow to maintain perfusion pressure?
by altering the resistance of cerebral blood vessels
26
if homeostatic mechanisms fail after a head injury, what effect does a rising ICP have?
can lead to reduced CPP and ischaemic damage
27
what are signs of increasing ICP?
decreasing Glasgow coma scale diminished pupil response to light lateralising signs eg weakness on one side
28
if there is uncontrolled rise in ICP, what are the types of herniation that occur? describe them
1st: cingulate herniation occurs cingulate gyrus slips under the flax cerebri and this causes a midline shift which is seen on CT scan 2nd: uncal herniation occurs uncal of temporal lobe slips under tentorium cerebellum and this ipsilaterally compresses oculomotor nerve (CN3) which affect pupils reflexes = fixed and dilated pupil if it progresses further: brainstem could be compressed