Cardiac Rhythm Abnormalities - EKG Flashcards

(43 cards)

1
Q

what does atrial flutter appear as on an EKG

A

saw tooth appearance
- repeating P waves

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2
Q

how are atrial rhythm abnormalities treated

A

cardioversion
ablation
antiarrhythmic
antithrombotic

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3
Q

for those with atrial rhythm what does exercise tolerance depend upon

A

rate control due to loss of atrial kick

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4
Q

explain atrial fibrilliation

A

rate varies and is always irregular
= due to SA node not setting the pace

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5
Q

how to treat atrial fibrillation

A

rate control digoxin, beta blockers, or calcium channel blockers

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6
Q

if atrial fibrillation is found to be new, what needs to be done / why?

A

notify medical provider immediately

cardiac stroke

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7
Q

what medications are indicated for A-Fib? what can this affect in regard to PT

A

cardioversion / anticoagulants
CO and decrease exercise tolerance

if new, may be accompanied in BP drop with lightheadedness

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8
Q

if the rate is controlled during A-Fib, what is the implication of PT? what may the patient report?

A

low-moderate exercise with slow progression

  • may report palpitations, angina, SOB and fatigue
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9
Q

explain HR associated with supraventricular tachycardia

A

<150 bpm but normal sinus rhyhtm

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10
Q

how is supraventricular tachycardia treated

A

self-limitations typically

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11
Q

what do patients typically report with supraventricular tachycardia

A

palpitations
dizziness
fatigue
SOB
chest discomfort
possible syncope

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12
Q

what is the treatment for premature ventricular contraction

A

none required if asymptomatic
- if >6-12 in one minute = underlying cardiac pathology may be indicated

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13
Q

if premature ventricular contractions occur in a run, what is the indication

A

may precede ventricular tachycardia
then V-fib

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14
Q

patterned preventricular contractions effect on SV

A

decreased –> then CO

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15
Q

for those with patterned PVCs, what is the PT indication?

A

low to moderate range
- monitor closely

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16
Q

what red flags are associated with PVCs

A

increased frequency of PVS
increased pattern changes
PVCs accompanied by symptoms
– maybe BP and/or syncope

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17
Q

what is the defining feature of ventricular tachycardia

A

three or more PVCs in a row

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18
Q

explain what ventricular tachycardia is

A

ventricles being in a constant state of contraction-relaxation

19
Q

what to do if you notice ventricular tachycardia?

A

stop exercising
CPR/defibrillation
call a code

20
Q

ventricular fibrillation is described as

A

chaotic and asynchronous electrical activity
– no CO

21
Q

what do to if ventricular fibrillation is noticed

A

life threatening, stop call code and do CPR/defibrillation

22
Q

asystole is described as ____? what is done in response?

A

cardiac standstill

CPR

23
Q

first degree atrioventricular blocks are described as

A

PT interval is prolonged by >0.2 seconds
then normal conduction occurs

24
Q

explain why 1st degree AV blocks occur

A

conduction through AV node is affected (slowed)

25
how do 1st degree AV blocks impair a pt
generally do not produce significant CO or exercise tolerance impairments
26
how are second degree AV blocks described
when not all P waves are followed by a QRS PR Interval is progressively longer and longer until there is one without a QRS
27
for those with 2nd degree AV blocks, what is the PT indication?
submaximal testing with gradual progression to set exercise response baseline - moderate intensity - do what makes sense with symptoms (ie more symptomatic, less intervention)
28
what are second degree AV blocks also called?
Mobitz 1 and 2
29
how are second degree (mobitz 2) AV blocks described
P waves that appear normal - end up being more P waves than QRS complexes PT interval normal or prolonged, but consistent
30
comparing rate of ventricle and atria, what does a mobitz 2 AV block suggest
atrial rate > ventricle rate
31
explain QRS complexes in Mobitz 2 AV block
QRS is often wider than normal
32
explain CO in those with Mobitz 2 - what is often associated with Mobitz 2 AV blocks/what is done to treat this?
CO is significantly compromised, patient will be symptomatic structural heart disease / higher risk of negative events pacemaker placement
33
explain atria / ventricle relationship in 3rd degree AV block
completely disassociated
34
explain P wave and QRS complex relationship in 3rd degree AV block
if activated by P wave, QRS looks normal if not, wide and bizarre QRS complex
35
what is atrial and ventricular rate
atria = 60-100 bpm ventricle = 20-40 bpm
36
explain PT intervention for those with 3rd degree AV block
need pacemaker, do not do anything until then
37
what is a bundle branch block
block of conduction in the right or left bundle branch
38
explain the differnce between RBBB and LBBB
RBBB = typically benign LBBB = structural heart disease
39
ST elevation is described as _____? this indicates _____?
ST segment higher than normal as assessed from J point STEMI = requires immediate medical attention
40
ST depression is described as __? it indicates _____?
ST segment lower than normal Ischemia
41
T wave changes indicate
ischemia ventricular hypertrophy non-coronary/cardiac conditions
42
are T wave changes ever normal
yea, in children
43
explain the evolution of STEMI in regard to EKG changes and times associated
hyperacute T wave = within minutes ST elevation = minutes to hours ST elevation & Q wave = 1-12 hrs ST elevation & T wave inversion = days T-wave recovery & Q-wave = weeks/months