EKG - Textbook Flashcards

(59 cards)

1
Q

what may cause sinus bradycardia

A

beta blockers
decreased automaticity of SA node
increased vagal stimulation

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2
Q

what is the most common form of sinus arrhythmia? if not this, what can cause the other form?

A

related to normal respiratory cycle
- rate increasing with inspiration/decreasing with expiration

infection, medication administration, and fever

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3
Q

explain what a sinus block is

A

SA node fails to initiate an impulse - typically only for one cycle

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4
Q

sinus pause includes

A

PR interval is 0.12-0.20
QRS complexes are identical and last 0.06-0.10 seconds
RR interval is regular with occasional pauses
HR between 60-100

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5
Q

what can cause sinus pause

A

sudden increase in PSNS activity
sick sinus syndrome
infection
rheumatic disease
severe ischemia or infarction of SA node

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6
Q

if a sinus pause is prolonged or occurs frequently what happens to CO

A

compromised - pt may complain of dizziness / syncope episodes

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7
Q

what are the characteristics of wandering atrial pacemakers

A

P waves present, but may look different
- P before QRS
PR intervals vary, but normal width
RR intervals vary
– <100 bpm

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8
Q

what populations are wandering atrial pacemaker arrhythmias often seen in

A

young/elderly
– ischemia or injury to SA node
– CHF
– increased vagal firing

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9
Q

what is a premature atrial complex defined as

A

ectopic focus in either atrium that initiates an impulse before the next impulse is initiated by the SA Node

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10
Q

defining feature of atrial tachycardia

A

three or more premature atrial complexes in a row

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10
Q

explain EKG findings of premature atrial complexes

A

the normal complexes look normal, but P wave of the early beats is noticeably different

P wave of the early beat may be buried within the T wave

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11
Q

what EKG findings are associated with atrial tachycardia

A

P waves may not be present before every QRS
QRS complexes are normal
RR intervals vary
HR is rapid, beating greater than 100 (maybe up to 200)

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12
Q

what may cause an atrial tachycardia

A

pulmonary disease with hypoxemia
pulmonary HTN
altered pH

– often found in those with COPD

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13
Q

define paroxysmal atrial tachycardia

A

atrial tachycardia or repetitive firing from an atrial focus
– normal sinus rhythm followed by an episodic burst of atrial tachycardia that then returns to normal

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14
Q

EKG finding associated with paroxysmal atrial tachycardia

A

rapid HR, often >160 bpm
P waves may be present or merged with T

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15
Q

if paryoxysmal atrial tachycardia remains for >24 hrs, what is it considered?

A

sustained atrial tachycardia

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16
Q

what can atrial flutter be caused by

A

rheumatic heart disease
mitral valve disease
coronary artery disease or infarction
renal failure
hypoxemia
pericarditis

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17
Q

what can cause atrial fibrillation

A

advanced age
CHF
ischemia/infarction
cardiomyopathy
rheumatic heart disease
renal failure

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18
Q

what is lost during atrial fibrillation

A

atrial kick
- up to 30% of CO is lost

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19
Q

explain the severity of A-Fib in patients with ventricular response greater/less than 100 bpm at rest

A

if less than, atrial fib is relatively benign

if more than, can begin to show signs of decomposition and need constant monitoring

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20
Q

explain the commonality of A-Fib

A

very common in older population
- will take anticoagulants

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21
Q

explain secondary issues related to a-fib and what can be done to prevent these issues

A

potential for developing mural thrombi
– due to coagulation of blood in fibrillating atria

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22
Q

what is a classic sign of a-fib that can be seen during intervention

A

very irregularly irregular pulse

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23
Q

define premature junctional/nodal complexes

A

premature impulses that arise from AV node or junctional tissues

24
EKG findings associated with premature junctional/nodal complexes
inverted, absent, retrograde P waves
25
what pathological conditions can cause premature junctional/nodal complexes
cardiac / mitral valve disease
26
what is junctional rhythm
when the AV junction takes over as pacemaker of the heart
27
EKG findings associated with junctional rhythm
absence of P waves before QRS - may be retrograde ventricular rate between 40-60 bpm
28
what can cause junctional rhythm
failure of SA Node -- sinus node disease, increased vagal tone, infarction or severe ischemia to conduction system
29
nodal / junctional tachycardia definition
AV junctional tissue acting as pacemaker but the rate of discharge becomes acccelerated
30
characteristics of nodal tachycardia in EKG
P waves absent, but retrograde P wave may be present HR typically < 100 bpm
31
common causes of junctional tachycardia include
coronary artery disease infarction postcardiac surgery digoxin toxicity myocarditis
32
what is the sequence of events that leads to a first degree AV block
impulses initiated at SA node is delayed - can be initiated in the AV node itself causes AV conduction time to be prolonged lengthening of PR interval only
33
what causes first degree AV blocks
coronary artery disease rheumatic heart disease infraction reactions to medications
34
what are the names of type one, 2nd degree AV block
Wenckebach or Mobitz 1
35
define a second degree AV block, type 1
transient disturbance that occurs high in the AV junction disrupts conduction of some of the impulses through the AV node
36
EKG characteristics of weckenbach / mobitz 1 AV block
P waves precede QRS -- progressive lengthening of PR interval until one P wave stands alone without QRS RR interval is irregular (regularly irregular)
37
causes of weckenbach AV block
right CAD / infarction digoxin toxicity excessive beta adrenergic blockade
38
define 2nd degree AV block, type 2
nonconduction of an impulse to the ventricles without a change in PR interval - site of blockage is typically below bundle of His and can be bilateral
39
EKG findings of mobitz 2 AV block
P wave to QRS complex ratio of 2-4:1 RR interval variance HR is typically below 100 and can be below 60
40
causes of second degree, type 2 AV block
myocardial infarction - especially if LAD is involved ischemia/infarction of AV node digoxin toxicity
41
define third degree AV block
impulses conducted above the ventricle are not conducted to the ventricle
42
characteristics of complete heart block (3rd degree AV block) on EKG
P wave present --- no relation to QRS complex QRS complexes and RR intervals are regular HR may range from 30-50 bpm
43
causes of 3rd degree AV block
myocardial infarction digoxin toxicity degeneration of conduction system
44
how do PVCs look on an EKG - yes the term, but describe it
wide and bizarre - without a P wave - followed by complete compensatory pause
45
define bigeminy PVCs
every other beat is a PVC
46
define trigeminy PVCs
every 3rd beat is a PVC
47
define multifocal PVCs
if more than one PVC is present and no two appear similar in configuration
48
define unifocal PVCs
PVC appear identical in configuration
49
explain paired / triplet PVCs -- any indication?
paired = two in a row together triplet = three in a row together, VTACH -- call code bro
50
what is an interpolated PVC
one that falls between two normal sinus beats that are separated by a normal RR interval
51
PVCs are often common in the presence of
ischemia cardiac disease overdistension of ventricle - CHF / cardiomyopathy acute infarction irritation of myocardium or its vessels chronic lung disease
52
what does a PVC feel like
pause or skip in regular rhythm that usually is followed by a stronger beat
53
why may PVCs be felt
decreased preload with PVC beat followed by long compensatory pause allow for increased filling time of ventricle an increased preload for the beat following the PVC and increased SV
54
PVCs are considered to be life threatening if
paired together multifocal >6 a min land directly on T wave triplets or more
55
what is the HR associated with V-Tach
100-250
56
causes of V-tach
ischemia acute infarction coronary artery disease HTN heart disease reaction to medication
57
what is the EKG reading that looks like a sound wave / sound cloud logo
tosade de pointes HR = 240-250
58
what is ventricular fibrillation
erratic quivering of ventricular muscle - no cardiac output will occur