CHAP 45 - GI Flashcards

(52 cards)

1
Q
  • Normally obtained by the body through oral ingestion of fluid and by the water formed in the metabolic breakdownof food.
  • It is lost from the body in urine and feces
A

FLUID

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2
Q

3 FLUID IMBALANCES

A
  1. ISOTONIC DEHYDRATION
  2. HYPERTONIC D
  3. HYPOTONIC D
  4. OVERHYDRATION
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3
Q

Occurs when a child’s body loses more water than it absorbs (as with diarrhea) or absorbs less fluid than it excretes (aswitnausea and vomiting)

A

ISOTONIC DEHYDRATION

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4
Q

When water is lost in a greater proportion than electrolytes

A

HYPERTONIC DEHYDRATION

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5
Q

There is a disproportionately high loss of electrolytes in diabetic proportion to fluid loss

A

HYPOTONIC D

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6
Q

Excessive body fluid intake; generally occurs in children who are receiving IV fluid

A

OVERHYDRATION

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7
Q

PH OF BLOOD

A

7.35-7.45

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8
Q
  • Results from diarrhea because a great deal of sodium is lost with stool
  • This excessive loss of Na+ causes the body to conserve H+ ions in an attempt to keep the number of positive andnegativeionsin serum balanced
A

METABOLIC ACIDOSIS

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9
Q
  • With vomiting, a great deal of hydrochloric acid is lost. - Number of H+ ions becomes proportionately lower than the number of OH- ions present
  • The serum HCO3 will invariably be high
A

METABOLLIC ALKALOSIS

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10
Q

Most children with _________ are suffering from an acute episode of gastroenteritis (infection) due to a viral or bacterial
organism.

A

VOMITING

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11
Q

is often associated with infectious, chronic illnesses, or malabsorption issues

A

DIARRHEA

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12
Q

 Causative agent: one of the Salmonella bacteria.

 Incubation period: 6 to 72 hours for intraluminal type; 7 to 14 days for extraluminal type

 Period of communicability: while the organisms are being excreted (may be as long as 3 months)

 Mode of transmission: ingestion of contaminated food, especially chicken and uncooked eggs

A

SALMONELLOSIS

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13
Q

Causative agent: Listeria monocytogenes.

 Incubation period: variable, ranging from 1 day to more than 3 weeks

 Mode of transmission: ingestion of unpasteurized milk or cheeses or vegetables grown in contaminated soil. Theinfectionis particularly important to avoid during pregnancy because infections during pregnancy can lead to miscarriageor stillbirth,prematurity, or infection of the newborn.

A

LISTERIOSIS

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14
Q

 Causative agent: organisms of the genus Shigella
 Incubation period: 1 to 7 days
 Period of communicability: approximately 1 to 4 weeks
 Mode of transmission: contaminated food, water, or milk products

A

SHIGELLOSIS (DYSENTERY)

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15
Q

 Causative agent: staphylococcal enterotoxin produced by some strains of Staphylococcus aureus
 Incubation period: 1 to 7 hours
 Period of communicability: carriers may contaminate food as long as they harbor the organism
 Mode of transmission: ingestion of contaminated food such as poultry, creamed foods (e.g., potato salad), andinadequatecooking

A

STAPHYLOCOCCAL FOOD POISONING

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16
Q

a results in loose, watery stools. The Chief therapy for these is ORS. Childrenwhoarecultured with G. Lamblia may be prescribed metronidazole.

A

PROTOZOAN / VIRAL DIARRHEA

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17
Q

COMMON DISORDERS OF THE STOMACH AND DUODENUM

A

 Inadequate function of the gastroesophageal valve
 Infections
 Gastroesophageal Reflux (GER)

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18
Q

in infants occurs due to the imma-turity of the lower esophageal sphincter, which allows easyregurgitation of gastric contents into the esophagus. It is very common during infancy, with about 70% of infants affected, anditusually requires no treatment. It is more common in preterm infants. It usually starts within 1 week of birth and may beassociated with a hiatal hernia. The emesis occurs after eating, is effortless, and most often consists of 1 to 2 oz of undigestedmilk.

A

GASTROESOPHAGEAL REFLUX

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19
Q

When infants develop complications from reflux such as:
 Irritability
 Failure to thrive
 Esophagitis
 Aspiration pneumonia
 Wheezing
 Apnea
Children with cerebral palsy or other neurologic involvement are at particular risk
Reflux is occasionally due to cow’s milk intolerance`

A

GASTROESOPHAGEAL REFLUX DISEASE

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20
Q
  • The pyloric sphincter is the opening between the lower portion of the stomach and the beginning portion of the intestine(theduodenum). If hypertrophy or hyperplasia of the muscle surrounding the sphincter occurs, it is difficult for the stomachtoempty,a condition called pyloric stenosis. The incidence is high, approximately 1:150 in males and 1:750 in females. It tends tooccurmost frequently in first-born White male infants. The exact cause is unknown, but multifactorial inheritance is a presumedetiology.
A

PYLORIC STENOSIS

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21
Q

a shallow excavation formed in the mucosal wall of the stomach, the pylorus, or the duodenum. Theyarerare,occurring in only 1% to 2% of children and more fre-quently in males than females.

A

PEPTIC ULCER DISEASE

22
Q

include both congenital disorders, such as obstruction or atresia of the biliary duct, and acquireddisor-ders,such as hepatitis or cirrhosis.

A

HEPATIC DISORDER

23
Q

(inflammation and infection of the liver) is caused by invasion of the A, B, C, D, or E virus

24
Q

 Causative agent: a picornavirus, A virus (HAV)
 Incubation period: 25 days on average
 Period of communicability: highest during 2 weeks preceding onset of symptoms
 Mode of transmission: in children, ingestion of fecal-contaminated water or shellfish; day care center spreadfromcontaminated changing tables

25
 Causative agent: a hepadnavirus; B virus (HBV)  Incubation period: 120 days on average  Period of communicability: later part of incubation pe-riod and during the acute stage  Mode of transmission: transfusion of contaminated blood and plasma or semen; inoculation by a contami-natedsyringeorneedle through IV drug use; may be spread to fetus if birthing parent has infection in third trimester of pregnancy
HEPA B
26
is a single-strand RNA virus. Transmission, as with HBV, is primarily by bloodor bloodproducts, IV drug use, maternal-fetal transfer, or sexual contact. The virus produces mild symptoms of disease, but thereisa high incidence of chronic infection with the virus
HEPATITS C
27
is enterically transmitted simi-larly to hepatitis A (e.g., fecal-contaminated water). Diseasesymptoms from the E virus can range from asymptomatic to mild to chronic liver disease. Young children oftenhavenosymptoms, and most people recover fully from the disease (CDC, 2021b).
HEPA E
27
when it persists for longer than 6 months. It is most likely the result of a hepatitis B, D, orCinfection. Abnormal liver enzyme levels and a liver biopsy establish the diagnosis and can predict the severity. Withchronichepatitis, fatty infiltration and bile duct dam-age can occur. The disease may progress to cirrhosis and even-tually liver failure.Therapy is supportive to compensate for decreased liver function (Lin & Kao, 2020).
CHRONIC HEPATITS
28
or the delta form, is similar to HBV in transmission, although it apparently requires a coex-istingHBV infection to be activated. Disease symptoms are mild, but there is a high incidence of fulminant hepatitis after theinitial infection.
HEPA D
29
is present when acute, massive necrosis or sudden, severe impairment of liver function oc-curs, leading to liver failure and hepatic encephalopathy. It can be due to infection or toxicity. Acetaminophen overdoseis alikelyetiology. Hepatic encephalopathy, or invasion of brain cells by ammonia, occurs because of the inability of the liver todetoxifythe ammonia being constantly produced by the intestine in the process of digestion (Brumbaugh et al., 2020)
FULMINANT HEPATIC FAILURE
30
generally occurs from congenital biliary atresia, stenosis, or absence of theduct. Italsocan occur from the plugging of biliary secretions, although this is rare. When the bile duct is obstructed, bile, unabletoentertheintestinal tract, accumulates in the liver. Bile pigments (direct bilirubin) enter the bloodstream and jaundice occurs, increasinginintensity daily.
OBSTRUCTION OF THE BILE DUCT
31
s (distended veins in the esophagus) are a frequent complication of liver disorders such as cirrhosis. Theygenerally form at the distal end of the esophagus near the stomach because of back pressure on the veins resulting fromincreased portal circulation blood pressure. Varices may bleed if the child coughs vigorously or strains to pass stool. Gastricreflux into the distal esophagus may irritate and erode the fine covering of the distended vessels, causing rupture
ESOPHAGEAL VARICES
31
is the accumula-tion of fatty deposits in the liver and is usually associated with obesity. As obesity is increasing in school-agedchildren and adolescents, this condition is also increasing in incidence (Brumbaugh et al., 2020). NAFLD can progress tocirrhosisin rare cases.
NON-ALCHOLIC FATTY LIVER DISEASE (NAFLD)
31
Is fibrotic scarring of the liver. ___________ means "yellow," or the typical color of hepatic scar tissue. It rarely occurs inchildren, although it may be seen as a result of congenital biliary atresia or as a complication of chronic ill-nesses such as protractedhepatitis, sickle cell anemia, or cystic fibrosis.
Cirrhosis
32
the surgical replacement of a mal-functioning liver by a donor liver. Child-size donor livers are not readily available, sothewaiting time for surgery may be months. Adult livers can be reduced in size for transplantation or a lobe of a liver fromalivingdonor can be used. Often, the child is extremely ill with ascites, GI bleeding, extreme pruritus, hepatic encephalopathy, orrenaldysfunction before the surgery. This makes nursing care after liver transplantation in a child complex as it involves takingcareofa child who has had major surgery and who normally would be too ill to undergo surgery.
LIVER TRANSPLANTATION
33
The invagination of one portion of the intestine into another, most frequently occurs in the second half of the first year of life(Mandeville, Chien, Willyerd, et al., 2012) with 90% of cases occurring by 2 years of age. About 75% of intussusceptionoccursforidiopathic reasons where there is no clear cause. In other cases, a “lead point” on the intestine likely cues the invagination. Sucha point might be a Meckel’s diverticulum, a polyp, hypertrophy of Peyer patches (lymphatic tissue of the bowel that increasesinsize with viral diseases), or bowel tumors. The point of the invagination is generally at the juncture of the distal ileumandproximal colon
INTUSSUSCEPTION
34
is the twisting of the intestine, which blocks stool passage and cuts off blood supply to the affected area. It oftenresultsfrom intestinal malrotation, a condition where the intestines do not rotate and attach properly during fetal development, usually between the 6th and 10th week of pregnancy. This incomplete rotation leads to an unstable mesentery, makingtwistingmore likely.
VOLVOLUS WITH MALNUTRITION
35
There is a lower incidence of the condition in infants who are fed breast milk than in those who are formula fed becauseintestinal organisms grow more profusely with cow’s milk than breast milk (cow’s milk lacks antibodies). A responsetotheforeign protein in cow’s milk may also be a mechanism that starts the necrotic process. Therefore, encouraging breastfeedingmay help prevent this disorder (Underwood, 2013) Signs of NEC usually appear in the first week of life. The infant’s abdomenbecomes distended and tense.
NECROTIZING ENTEROCOLITIS (NEC)
36
is an absorptive disorder in which there is not sufficient bowel surface area in the small intestine for proper nutrient absorption.The condition has several causes, including surgery for NEC, volvulus, and GI tract trauma, which resulted in a largeportionofthe intestine being removed.
SHORT BOWEL/SHORT GUT SYNDROME
37
(INFLAMMATION OF THE APPENDIX) is the most common cause of abdominal surgery in children. It occurs most frequently in school-age children and adolescents, although it can occur in preschoolers and even in newborns (Ladd, Neff, Becher, et al., 2012).
APPENDICITIS
38
During embryonic life, the intestine is connected to the umbilicus by the omphalomesenteric (vitelline) duct, whichtypicallyregresses by term. In about 2–3% of infants, a portion remains as Meckel’s diverticulum—located near the ileum. It maycontaingastric mucosa that secretes acid, causing intestinal irritation, ulcers, and painless, bloody or tarry stools. It can alsoleadtointussusception or bowel obstruction due to fibrous bands. Diagnosis is suggested by history and confirmed with a Meckel’sscan.Treatment involves laparoscopic removal.
MECKEL'S DIVERTICULUM
39
is an immune reaction to gluten found in wheat, rye, barley, and possibly oats, in genetically predisposedindividuals.
CELIAC DISEASE
40
is a common childhood issue, often starting in the first year of life, and occurs without an underlyingmedical condition. It’s usually defined as two or fewer painful bowel movements per week. Stools are hard, large, andpainful, sometimes causing anal fissures. This pain leads to stool holding, which worsens the problem as the rectumstretches andreduces the urge to defecate.
CONSTIPATION
41
is a protrusion of a section of the bowel into the inguinal ring. It usually occurs in boys (9:1) because, as the testes descendfromthe abdominal cavity into the scrotum late in fetal life, a fold of parietal peritoneum also descends, forming a tube fromtheabdomen to the scrotum (García-Hernández, Carvajal-Figueroa, Suarez- Gutiérrez, et al., 2012). In most infants, this tubeclosescompletely.
INGUINAL HERNIA
42
(instillation of carbon dioxide into the perineal cavity) during surgery may be performed to reveal thepresence of an enlarged inguinal ring on the opposite side
PNEUMOPERITONEUM
43
is an absence of ganglionic innervation to the muscle of a section of the bowel—in most instances, the lower portionof thesigmoid colon just above the anus
AGANGLIONIC MEGACOLON
44
develop crampy abdominal pain, urgency, tenesmus, and frequent bloody stools. Anemia and hypoalbuminemia due to losses in the stool may be present.
ULCERATIVE COLITIS
45
Common signs include abdominal pain, diarrhea (with or without blood), and weight loss. Inflammation may cause bowel narrowing (strictures) and, if untreated, bowelobstruction. Fistulae—abnormal tunnels between the bowel and other organs or skin—often affect the perianal area
CROHN DISEASE
46
is a functional bowel disorder marked by abdominal pain and altered bowel habits without anidentifiable organic cause
IRRITABLE BOWEL SYNDROME
47
The pain can be constant or come and go, usually centered around the belly button, with no link to meals or bowel habits.Physical exams often show no signs of tenderness or swelling, but stress-related issues like sleep problems or family tensionmaybe present. Though the pain is real, treatment focuses on symptom management through dietary changes (like addingfiberorprobiotics), medications, and therapies such as cognitive behavioral therapy, hypnosis, and parental support
RECURRENT ABDOMINAL PAIN
48
a severe protein deficiency disease
KWASHIORKOR
49
a severe form of malnutrition caused by a deficiency of all food groups, essentially a state of starvation.
NUTRITIONAL MARASMUS