Chapter 15 Flashcards

(49 cards)

1
Q

shock/hypoperfusion

A

inadequate perfusion of cells, tissue, and organs with oxygen and other nutrients resulting in cell, tissue, and organ dysfunction.

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2
Q

etiology

A

causation of disease/condition

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3
Q

etiologies of shock

A

inadequate volume
inadequate pump
inadequate vessel

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4
Q

4 categories of shock

A

hypovolemic, distributive, cardiogenic, obstructive

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5
Q

5th category of shock

A

metabolic

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6
Q

hypovolemic shock

A

low blood volume
most common form of shock
types:
hemorrhagic, nonhemorrhagic, burn

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7
Q

distributive shock

A

decrease in intravascular volume caused by massive systemic vasodilation and an increase in capillary permeability
types:
neurogenic, septic, anaphylaxis

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8
Q

obstructive shock

A

results from a condition that obstructs forward blood flow. The volume is adequate, the heart is not damaged, and the vessels are of a normal size with adequate resistance.

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9
Q

cardiogenic shock

A

caused by ineffective pump function of the heart

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10
Q

metabolic shock

A

described as a dysfunction in the ability of oxygen to diffuse into the blood, be carried by hemoglobin, off-load at the cell, or be used effectively by the cell for metabolism.

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11
Q

hemorrhagic shock

A

whole blood from the intravascular space. The term hemorrhagic specifically indicates whole blood loss that can occur from a traumatic injury or a medical illness

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12
Q

nonhemorrhagic hypovolemic shock

A

caused by the shift of fluid out of the intravascular space; however, red blood cells and hemoglobin remain within the vessels. It is primarily water, plasma proteins, and electrolytes that are lost.

examples:severe diarrhea, vomiting, excessive sweating, and excessive urination.

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13
Q

burn shock

A

specific form of nonhemorrhagic hypovolemic shock resulting from a burn injury. Burns can interrupt the integrity of the capillaries and vessels and allow them to leak plasma and plasma proteins.

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14
Q

anaphylactic shock

A

Chemical mediators that are released in the anaphylactic reaction cause massive and systemic vasodilation. These chemical mediators also cause the capillaries to become permeable and to leak.

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15
Q

septic shock

A

results from sepsis, which is an exaggerated inflammatory response to an infection that causes the vessels throughout the body to dilate and become permeable. Fluid leaks out of the vessels into the interstitial space.

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16
Q

sepsis

A

the body’s exaggerated inflammatory response to an infection, typically fungal, viral, or bacterial, that overwhelms the body’s normal defense and regulatory systems causing a disruption in cell and organ function.

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17
Q

sepsis pathophsyiology

A

Increased capillary permeability

Vasodilation

Myocardial depression

Alveolar/capillary membrane damage allowing fluid to accumulate in the alveoli

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18
Q

sepsis s/s

A

Tachycardia

Tachypnea

Hyperthermia or hypothermia

Hypotension

flushed warm skin (early)
cyanotic (late)

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19
Q

respiratory based sepsis s/s

A

Productive cough

Fever

Chills

Upper respiratory symptoms (runny or stuffy nose, sneezing)

Throat or ear pain

Crackles upon auscultation (may indicate pneumonia)

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20
Q

GI Based Sepsis s/s

A

Abdominal pain or flank tenderness

Nausea and vomiting

Diarrhea

Dysuria

Polyuria

21
Q

robson screening tool

for suspected sepsis

A

Sepsis is suspected if two of the following findings are present

temp <96.88 or >100.98
hr > 90
RR > 20/min
acute ams
BGL < 120

22
Q

BAS 90-30-90 scale

for susptected sepsis

A

Sepsis is suspected if one or more of the following findings are present

systolic bp < 90
RR > 30/min
spo2 < 90

23
Q

qsofa scale

A

Sepsis is suspected if two or more of the following findings are present

RR >= 22/min
AMS GCS<13
systolic bp <100

24
Q

neurogenic shock

A

Spinal cord injury is a cause of neurogenic shock. A spinal cord injury may damage the sympathetic nerve fibers that control vessel tone below the level of injury. Loss of sympathetic tone causes the vessels to dilate. If the injury is high in the thoracic spinal cord or in the cervical region, enough vessel tone may be lost to cause a drop in systemic vascular resistance, blood pressure, and perfusion.

25
sympathetic system immediate response to shock
Increase in heart rate Increase in force of ventricular contraction (stroke volume) Vasoconstriction Stimulation of the release of epinephrine and norepinephrine from the adrenal gland
26
Stages of shock
Compensatory, Decompensatory
27
Multiple organ dysfunction syndrome (mods)
stage in which multiple organs begin to fail throughout the body from extreme and prolonged hypoxia, altered metabolism, and elevated carbon dioxide and acid levels.
28
compensatory shock s/s
anxiety normal bp mild tachycardia pulse: thready in extremities mild-moderate tachypnea 20-30/min pale cool skin
29
decompensatory shock s/s
ams hypotension marked tachycardia >120 weak/absent pulse white/waxy skin
30
hypovelemic shock s/s
decrease bp narrowing pulse pressure tachycardia tachypnea pale cool clammy skin poor spo2 weak/absent peripheral pulse
31
cardiogenic shock s/s
decrease bp narrowing pulse pressure tachycardia or bradycardia tachypnea pale cool clammy skin jvd/peripheral edema (R side heart failure) crackle/rales (L side heart failure)
32
distributive shock s/s
decrease bp tachycardia (anaphylactic/septic) bradycardia (neurogenic) tachypnea Flushed skin (sept/anaphylactic) cyanosis(late sep/anaphy/neuro) weak/absent pulse fever (sepsis) loss of motor function (neuro) edema (anaphylactic)
33
obstructive shock s/s
decrease bp pulsus paradoxus narrowing pulse pressure tachycardia tachypnea pale cool clammy skin JVD (tension pneumo, pericardial tamponade) weak/absent pulse severely decreased to absent breath sound
34
Cardiac arrest three phases
electrical, circulatory, metabolic
35
electrical phase
0-4 min heart still has a good supply of oxygen and glucose; therefore, aerobic metabolism is maintained with continued energy production for cell function and prevention of mass production of acid.
36
circulatory phase
4-10 min the oxygen stores are reduced and the myocardial cells shift from aerobic to anaerobic metabolism. This results in very little energy production for cell function, in addition to the production of acid
37
metabolic phase
after 10 min heart is starved of oxygen and glucose and has a large amount of acid buildup. The tissues are ischemic and may begin to die. The chances of survival drop dramatically during this phase
38
return of spontaneous circulation (ROSC)
when the patient regains a spontaneous pulse during the resuscitation effort. The patient may not yet have begun to breathe on his own
39
two most common shockable rhythms
Ventricular fibrillation and pulseless ventricular tachycardia
40
fully automated aed vs semi aed
more buttons to push; semi is more preferred
41
ventricular fibrillation (v-fib)
a disorganized cardiac rhythm that produces no pulse or cardiac output
42
pulseless ventricular tachycardia (v-tac)
very fast rhythm that is generated in the ventricle instead of the sinoatrial node in the atrium. Because the pumping is so rapid, the heart does not refill properly and cardiac output is sharply reduced.
43
non shockable rhythms
asystole and pulseless electrical activity
44
asystole
absence of electrical activity and pumping action in the heart.
45
pulseless electrical activity (PEA)
heart has an organized rhythm, but either the heart muscle is so weakened that it fails to pump, or the heart muscle does not respond to the electrical activity, or the circulatory system has lost so much blood that there is nothing to pump.
46
lateral uterine displacement
relieve pressure off of vena cavae by displacing uterus women pregnant at 20 weeks or greater
47
pacemaker aed consideration
dont place pad directly over aed
48
automatic implantable cardioverter defibrillators (ICDs)
monitor the heart’s electrical activity and provide a shock to the heart if it detects a shockable dysrhythmia
49
ICD AED Consideration
place pad at least 1 inch away wait 30 to 60 seconds for the ICD to complete its cycle prior to attaching the AED