COPD Flashcards

(39 cards)

1
Q
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2
Q

What is COPD?

A

Chronic Obstructive Pulmonary Disease (COPD) is a progressive, chronic inflammatory lung disease characterized by airflow limitation that is not fully reversible.

The inflammation causes slowly an obstruction to bronchioles and thereby a pulmonary emphysema.

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3
Q

What are the main symptoms of COPD?

A

Chronic cough, sputum production (especially in the morning), dyspnea (shortness of breath), wheezing, chest tightness, decreased exercise tolerance, fatigue due to poor oxygenation, frequent respiratory infections, weight loss, and decreased Spo2 % levels (increased risk for Co2-retention).

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4
Q

What are examples of COPD?

A

emphysema and chronic bronchitis.

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5
Q

What are the stages of COPD according to GOLD Criteria?

A

Stages include:
- Mild (Stage I): FEV1 ≥80%, may have no symptoms, winded with moderate exercise or walking upstairs.

  • Moderate (Stage II): FEV1 50–79%, frequent stops to catch breath, coughing, wheezing and breathlessness.
  • Severe (Stage III): FEV1 30–49%, shortness of breath worsens, frequent flare-ups or exacerbations that leads to hospitalization. Worsening symptoms.
  • Very Severe (Stage IV): FEV1 <30% or <50% with respiratory failure, Low oxygen level, constant shortness of breath. Flare ups and exacerbation, can be life threatening.
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6
Q

What is the pathophysiology of Emphysema?

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Emphysema develops when the alveoli are destroyed, causing them to merge into one large air sac, leading to decreased surface area for gas exchange and air trapping.

Since it doesn’t absorb oxygen as well as healthy alveoli, the blood gets less oxygen.

Emphysema also causes the lungs to stretch out, losing their elasticity. As a result, air remains trapped in the lungs, making breathing difficult.

Alveoli are destroyed, leading to fewer, larger air sacs

Decreased surface area for gas exchange

Air trapping and hyperinflation

Loss of lung elasticity

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7
Q

What are the clinical presentations of Emphysema?

A

‘pink puffers’ (normal oxygen levels, pursed-lip breathing), severe dyspnea, barrel chest from difficulty exhaling, leading to air trapping., weight loss, and decline in functional ability.

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8
Q

What is the pathophysiology of Chronic Bronchitis?

A

The condition causes inflammation in the lining of bronchial tubes, which are responsible for carrying air to and from the alveoli. Chronic bronchitis damages the cilia (the hair-like fibers lining the bronchial tubes), which help eliminate mucus.

As a result, it becomes more challenging to remove mucus, making you cough more and obstructs the airflow. That’s why people with chronic bronchitis develop mucus that stays for an extended period, sometimes up to two years. Other symptoms of chronic bronchitis are shortness of breath and coughing.

Inflammation of the bronchi

Increased mucus production and thickening of airway walls

Ciliary dysfunction

Airflow obstruction

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9
Q

What are the clinical presentations of Chronic Bronchitis?

A

‘blue bloaters’ (low oxygen levels, cyanosis, fluid retention), persistent productive cough with thick mucus esp in morning, and edema.

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10
Q

What is the primary cause of COPD?

A

Smoking is the primary cause of COPD.

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11
Q

What are other risk factors for COPD?

A

Long-term exposure to air pollutants, second-hand smoke, indoor cooking fuels, occupational exposure to dust, fumes, and chemicals from workplace, genetic predisposition (Alpha-1 antitrypsin deficiency) a rare genetic disorder that can cause COPD even in nonsmokers, and respiratory infections.

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12
Q

What are the main risk factors for emphysema?

A

Long-term smoking, genetic deficiency (Alpha-1 antitrypsin), and chronic respiratory infections.

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13
Q

What are the main risk factors for chronic bronchitis?

A

Smoking, recurrent respiratory infections, air pollution, and occupational irritants.

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14
Q

What are the pharmacological treatments for COPD?

A

Bronchodilators (e.g., albuterol, salmeterol) - Relax airway muscles, inhaled corticosteroids (e.g., fluticasone) - Reduce airway inflammation, combination inhalers, oral corticosteroids (short-term), and antibiotics for infections.

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15
Q

What are the non-pharmacological interventions for COPD?

A

Smoking cessation, pulmonary rehabilitation, exercise training, education, and breathing techniques to improve quality of life, oxygen therapy (if SpO2 <88%), PAP at night in cases of sleep apnea and COPD or CPAP or BiPAP CPAP or BiPAP can be consider in palliative phase of the disease for ventilation support, Lung Transplantation. Lung transplantation is a viable option for definitive surgical treatment of end-stage emphysema, Surgery: Bullectomy

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16
Q

What is bullectomy?

A

Bullectomy is a surgical option for select patients with bullous emphysema and can help reduce dyspnea and improve lung function.

17
Q

What is lung volume reduction surgery?

A

Lung Volume Reduction Surgery. Lung volume reduction surgery is a palliative surgery in patients with homogenous disease or disease that is focused in one area and not widespread throughout the lungs.

18
Q

What surgical options are available for COPD?

A

Bullectomy, lung volume reduction surgery, and lung transplantation.

19
Q

What are the long-term treatments for COPD?

A

Maintenance bronchodilators, inhaled corticosteroids, two long-acting β2-agonists in the same inhaler, and lifestyle modifications.

20
Q

What are the short-term treatments for COPD?

A

Rescue inhalers (SABAs) and oral corticosteroids during exacerbations.

21
Q

What is CPAP?

A

Continuous Positive Airway Pressure (CPAP) is a device that pressurizes the air to a higher level than the air in the room,- delivering constant air pressure to - keep airways open, especially during sleep. - It prevents alveolar collapse and - improves oxygenation. Used in sleep apnea and acute exacerbations of COPD.

22
Q

What is BiPAP? It’s Functions?

A

Bilevel Positive Airway Pressure (BiPAP) provides two levels of pressure: higher during inhalation (IPAP) and lower during exhalation (EPAP). It supports both breathing in and out and enhances ventilation in severe COPD.

23
Q

What are the main aims for treating COPD?

A

To relieve symptoms, improve exercise tolerance and quality of life, prevent disease progression and exacerbations, and reduce mortality.

24
Q

What are the prevention strategies for COPD?

A

smoking cessation, avoiding occupational/environmental irritants, vaccinations (influenza, pneumococcal), early treatment of respiratory infections, and regular follow-ups with a healthcare provider.

25
What are the symptoms and clinical presentation of COPD?
Symptoms of COPD include cough, dyspnea, and sputum. A history of smoking or pollutant exposure is common, and physical signs may include barrel chest, wheezing, and prolonged expiration.
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What are the necessary examinations for diagnosing COPD?
Spirometry (FEV1/FVC <70%), chest X-ray or CT scan, arterial blood gas (ABG), sputum cultures, alpha-1 antitrypsin testing, Blood chemistry: alpha1-antitrypsin is measured to verify deficiency and diagnosis of primary emphysema. Complete blood count (CBC) and differential - Increased hemoglobin (advanced emphysema), increased eosinophils (asthma). Electrocardiogram (ECG) - Right axis deviation, peaked P waves (severe asthma); atrial dysrhythmias (bronchitis), tall, peaked P waves in leads II, III, AVF (bronchitis, emphysema); vertical QRS axis (emphysema).
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What to do in emergency response for COPD?
Assess airway, breathing, SpO2 Administer SABA (e.g., salbutamol) Provide supplemental oxygen Initiate corticosteroids and antibiotics if indicated Position patient upright.
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What should be assessed in a nursing assessment for COPD?
Assess patient’s exposure to risk factors, past and present medical history, signs and symptoms of COPD and their severity, patient’s knowledge of the disease, vital signs, breath sounds and pattern, respiratory rate and rhythm, lung sounds, SpO2 monitoring, skin color, and use of accessory muscles.
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What are some nursing interventions for COPD patients?
Administer medications as prescribed, educate on inhaler use, position for optimal breathing, encourage fluid intake and coughing, and teach breathing techniques.
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Nursing interventions
Administer medications as prescribed (bronchodilators and corticosteroids) stay alert for potential side effects. Educate on inhaler use. Positioning for optimal breathing (high fowler position) Encourage fluid intake and coughing Pursed lip breathing. Pursed lip breathing helps slow expiration, prevents collapse of small airways, and control the rate and depth of respiration. Diaphragmatic breathing. Diaphragmatic breathing reduces respiratory rate, increases alveolar ventilation, and sometimes helps expel as much air as possible during expiration. Inspiratory muscle training. This may help improve the breathing pattern. Monitor cognitive changes. The nurse should monitor for cognitive changes such as personality and behavior changes and memory impairment. Monitor pulse oximetry values. Pulse oximetry values are used to assess the patient’s need for oxygen and administer supplemental oxygen as prescribed. Prevent infection. The nurse should encourage the patient to be immunized against influenza and S. pneumonia because the patient is prone to respiratory infection.
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What is pursed lip breathing?
Pursed lip breathing helps slow expiration, prevents collapse of small airways, and control the rate and depth of respiration.
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What is diaphragmatic breathing?
Diaphragmatic breathing reduces respiratory rate, increases alveolar ventilation, and sometimes helps expel as much air as possible during expiration.
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Why is pulse oximetry important in COPD management?
Pulse oximetry values are used to assess the patient’s need for oxygen and administer supplemental oxygen as prescribed.
35
What are key points in patient education for COPD?
Proper inhaler technique, PEP blow guidance, peer support - rehabilitation groups especially for physical exercise, importance of medication adherence, smoking cessation, recognizing symptoms of exacerbation, energy conservation techniques, Manage daily activities. Daily activities must be paced throughout the day and support devices can be also used to decrease energy expenditure. Exercise training. Exercise training can help strengthen muscles of the upper and lower extremities and improve exercise tolerance and endurance.
36
What are common symptoms of COPD exacerbation?
Increased dyspnea, increased sputum volume or purulence, wheezing, chest tightness, fever, fatigue, functional capacity due to poor ventilation, and sleep difficulties.
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What treatments are indicated for COPD exacerbation?
Sputum sample, Chest X-ray if pneumonia is suspected, Blood samples (CBC, CRP, Krea and electrolytes), Medicines (Bronchodilators, inhaled corticosteroid, antibiotics), Oxygen supply carefully, remember the risk of CO2- retention… nasal prongs, Sometimes invasive or non-invasive (CPAP, BiPaP) ventilation is needed. Prophylaxis of vein thrombosis
38
What nursing considerations are important during COPD exacerbation management?
Monitor ABGs and vitals, assess skin color, auscultate heart and lungs, assess breathing, ensure medication compliance, patient positioning, prevent infections, follow ups and encourage vaccination (influenza and pneumococcal)
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Management of the exacerbation of COPD
SABA use Corticosteroids and antibiotics Increased oxygen (with caution due to CO2 retention)