D & N OF THE NOSE, PARANASAL SINUSES & FACE 1.1 (AB) Flashcards

(67 cards)

1
Q

What is choanal atresia?

A

Embryologic failure of the bucconasal membrane to rupture before birth

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2
Q

What connects the nasal cavities with the nasopharynx?

A

Choanae

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3
Q

When do the choanae develop?

A

Between the 3rd and 7th embryonic weeks

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4
Q

How often does choanal atresia occur?

A

1 in 5000 to 8000 live births

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5
Q

What is the female to male ratio in choanal atresia?

A

2 to 1

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6
Q

What is the treatment for choanal atresia?

A

Surgery. Puncture or remove the uncultured bucconasal membrane

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7
Q

When do nasal dermoid cysts usually present?

A

At birth but may remain unrecognized until later childhood or adult life

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8
Q

What are nasal dermoid cysts composed of?

A

Epithelium-lined cavities or sinus tracts filled with keratin debris. hair follicles. sweat glands. and sebaceous glands

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9
Q

What causes nasal dermoid cysts?

A

Failure of ectodermal elements of the fetal trilaminar septum to degenerate

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10
Q

What is a nasal glioma?

A

Rare congenital lesion composed of dysplastic glial cells that have lost intracranial connections

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11
Q

What causes nasal glioma?

A

Abnormal closure of the fonticulus frontalis

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12
Q

Where are nasal gliomas most commonly located?

A

60 percent extranasal. 30 percent paranasal. 10 percent both

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13
Q

What is encephalocele?

A

Congenital extracranial herniation of CNS tissues through a skull base defect

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14
Q

What is the cause of encephalocele?

A

Arrested closure of normal confining tissue and early outgrowth of neural tissue

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15
Q

What conditions are associated with encephalocele?

A

Midline fusion defects and CNS anomalies

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16
Q

What is a mucocoele?

A

Cyst-like. mucus-containing sac within a sinus

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17
Q

What is a pyocoele?

A

Mucocoele containing pus due to superinfection

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18
Q

What causes mucocoeles?

A

Adhesions. mass lesions. and obstruction of drainage pathways

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19
Q

What is the most common site for mucocoeles?

A

Frontal sinus

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20
Q

What is the least common site for mucocoeles?

A

Sphenoid sinus

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21
Q

How does a maxillary sinus mucocoele present?

A

Cheek swelling with proptosis due to orbital floor displacement

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22
Q

How does a frontal sinus mucocoele present?

A

Tense swelling over frontal sinus. inferolateral displacement of orbit

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23
Q

How does an ethmoid sinus mucocoele present?

A

Proptosis or diplopia due to lateral displacement of medial orbital wall

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24
Q

How does a sphenoid sinus mucocoele present?

A

Headache radiating to vertex and occiput

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25
How are mucocoeles diagnosed?
History. CT scan. and MRI
26
What is the treatment for mucocoeles?
Surgical removal and re-establishment of drainage or sinus obliteration
27
What is nasal septum deviation?
Congenital or traumatic bending or bowing of the nasal septum
28
What are symptoms of nasal septum deviation?
Nasal obstruction. olfactory impairment. sinusitis. epistaxis
29
How is nasal septum deviation diagnosed?
Anterior rhinoscopy. endoscopy. rhinomanometry
30
What are treatments for nasal septum deviation?
Septoplasty. scoring. or submucous resection
31
What is anterior epistaxis?
Bleeding in front of the maxillary sinus ostium. usually mild
32
What is posterior epistaxis?
Bleeding behind the maxillary sinus ostium. often severe
33
What is the most common cause of anterior epistaxis?
Trauma
34
What is the typical age for posterior epistaxis?
Over 40 years old
35
How is epistaxis diagnosed?
BP. Hgb. platelet count. coagulation profile. anterior rhinoscopy and endoscopy
36
What are general measures for treating epistaxis?
Nasal compression. upright posture. avoid swallowing blood. ice bag
37
What is Trotter’s method for epistaxis?
Patient sits and leans forward while pinching the nostril and breathing through mouth
38
What is silver nitrate cautery used for?
Mild epistaxis from Kiesselbach’s area
39
When is nasal packing used?
For severe epistaxis
40
When is vascular ligation indicated?
Persistent bleeding despite nasal packing
41
What arteries may be ligated in epistaxis?
Sphenopalatine artery. internal maxillary artery. ethmoid artery. external carotid artery
42
When is embolization used in epistaxis?
Posterior epistaxis refractory to standard treatments
43
Q
A
44
What are the signs of inflammation?
Hotness. Redness. Swelling. Pain. Loss of function.
45
What is the cardinal sign of inflammation?
Pain.
46
What is the sequence of inflammation?
Injury. Vascular response. Cellular response. Healing.
47
What is the vascular response in inflammation?
Vasodilation. Increased vascular permeability.
48
What causes vasodilation in inflammation?
Histamine. Prostaglandins. Bradykinin.
49
What causes increased vascular permeability?
Histamine. Bradykinin. Leukotrienes.
50
What are the main cells involved in acute inflammation?
Neutrophils. Macrophages.
51
What are the main cells involved in chronic inflammation?
Lymphocytes. Plasma cells. Macrophages.
52
What are the types of exudate?
Serous. Fibrinous. Purulent. Hemorrhagic.
53
What is serous exudate?
Watery fluid with low protein content.
54
What is fibrinous exudate?
Thick
55
What is purulent exudate?
Thick yellow-green fluid with many neutrophils. Dead cells. Microorganisms.
56
What is hemorrhagic exudate?
Contains RBCs due to severe vessel damage.
57
What is an abscess?
Localized collection of pus.
58
What is cellulitis?
Diffuse
59
What is an ulcer?
Localized area of tissue necrosis on skin or mucous membrane.
60
What are systemic effects of inflammation?
Fever. Leukocytosis. Malaise. Anorexia.
61
What is leukocytosis?
Increased white blood cells in the blood.
62
What causes fever in inflammation?
Pyrogens. Cytokines like IL-1 and TNF.
63
What is granulomatous inflammation?
Chronic inflammation with granuloma formation.
64
What are examples of granulomatous diseases?
Tuberculosis. Sarcoidosis. Leprosy.
65
What is a granuloma?
Collection of macrophages. Giant cells. Lymphocytes.
66
What are the outcomes of acute inflammation?
Resolution. Abscess formation. Chronic inflammation.
67
What are the phases of wound healing?
Inflammation. Proliferation. Maturation.