Divalproex sodium Flashcards by Lucas Holanda

Q

Seizures:

A

Decreased

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Q

GABA:

A

Increased

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Q

Mitogen-activated protein kinase-extracellular signal-regulated kinase:

A

Activated

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Q

Histone deacetylase:

A

Inhibited

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Q

Mood:

A

Stabilized

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Q

Neuroprotection:

A

Increased

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Q

BDNF:

A

Increased

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Q

Melatonin MT1 receptor:

A

Increased

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Q

Synergistic with:

A

Melatonin, Nifedipine,

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Q

Dispepsia, Diarreia, Tremor, Seda-
ção, +TGP, -Leuco,
-Plaq, Perda de cabelo e +Peso:

A

Adverse effects

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Q

Neuropeptide Y:

A

Increased

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Q

Voltage-dependent Sodium channels:

A

Blocked

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Q

Conditioned fear:

A

Decreased

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Q

Alzheimer:

A

Controlled

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Q

Excitotoxicity:

A

Decreased

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Q

Oxidative stress:

A

Decreased

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Q

Dopaminergic neurons in midbrain:

A

Protected

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Q

Proteina-quinase C:

A

Decreased

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Q

Cognitive dysfunction induced by d-amphetamine:

A

Decreased

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Q

Glutamate receptor expressions:

A

Regulated

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Q

Hippocampal neurogenesis:

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A

Increased

Q

Axonal regeneration:

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A

Increased

Q

Inositol:

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A

Depleted

Q

Oxygen-glucose deprivation injury:

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A

Decreased

Lifespan of cortical neurons:

Increased

Neurons exposed to hemorrhage or ischemia:

Protected

Transcription factor CREB

Activated

CREB:

cAMP response element | binding protein

Pro-apoptotic molecules:

Inhibited

Microglia-mediated inflammation:

Decreased

Blood-Brain Barrier permeability:

Decreased

Cognitive dysfunction induced by TBI:

Decreased

Glycogen synthase kinase-3:

Activated

Bcl-2:

Increased

Pain:

Decreased

Migraine:

Decreased

Inflammation:

Decreased

Glycine:

Increased

Taurine:

Increased

Glutamic acid decarboxylase:

Enhanced

CSF:

Increased

Extracellular Serotonin:

Increased

Extracellular Dopamine:

Increased

CNS axons:

Regenerated

Grey matter volume:

Increased

Brain N-acetyl aspartate:

Increased

Neuritogenesis:

Increased

Neural cell adhesion molecules:

Increased

Learning:

Increased

Memory:

Improved

Microtubule formations:

Stabilized