exam 5 part 2 Flashcards

(30 cards)

1
Q

barrett esophagus

A

long term acid reflux damages esophageal lining (and causes dysplasia) and increases risk of developing adenocarcinoma (cancer)

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2
Q

gerd treatment

A

antacids, H2-receptor antagonists (blocks histamine which stimulates release of gastric juice), proton pump inhibitors (block release of hydrochloric acid)

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3
Q

coffee grounds vomitus

A

caused by the partial digestion of blood protein by the stomach (hematemesis)/intestines

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4
Q

yellow vomitus

A

bile from the duodenum

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5
Q

deep brown vomitus

A

indicates contents from the lower intestine which is seen in patients with intestinal obstruction (ileum and large intestine)

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6
Q

Frank blood in vomit

A

Visible blood in the vomit - issues with esophagus

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7
Q

frequent vomiting after a meal

A

indicates a problem with gastric emptying (pyloric sphincter not opening or obstructed)

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8
Q

Frank blood in stool

A

appears red because it has not been digested and usually results from lesions in the rectum or anal canal

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9
Q

melena

A

dark stool resulting from bleeding that occurs higher in the intestinal tract and the blood has been digested
- Tarry - bleeding in ascending transverse or descending colon

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10
Q

occult blood

A

hidden blood that is not visible to the eye but can be detected upon testing the stool

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11
Q

dumping syndrome

A

May result from gastric resection in which the pyloric sphincter has been removed. Large amounts of food enters the small intestine at one time -> dumping into the SI

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12
Q

s/s dumping syndrome

A

cramps, nausea, diarrhea, hypovolemia, hyperglycemia followed by hypoglycemia (s/s of hypovolemia, hypoglycemia)

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13
Q

Why do people with dumping syndrome develop hyperglycemia followed by hypoglycemia

A

rapid delivery of nutrients to the small intestine triggers a rise in blood glucose, followed by overproduction of insulin

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14
Q

Mechanical obstruction

A
  • Intestinal Hernias — portions of intestine that protrude into another part of your body
  • Tumors in the small or large intestine
  • Inflammatory bowel diseases, such as Crohn’s disease
  • Twisting of the intestine (volvulus)
  • Telescoping of the intestine (intussusception)
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15
Q

Mechanical obstruction bowel sounds

A

results in high-pitched peristalsis (Longer the obstruction - less likely to hear high pitched bowel sound)

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16
Q

Paralytic obstruction

A
  • malfunction of nerves and muscle
  • electrolyte imbalances
  • gastroenteritis
  • Appendicitis
  • pancreatitis
  • surgical complications
  • Certain drugs and medications
17
Q

Paralytic obstruction bowel sounds

A

bowel sounds are inaudible (silent abdomen).

18
Q

liver role in digestion

A

produces bile and filters toxins from the blood
- Bilirubin excretion
- Drug and hormone metabolism (makes water-soluble forms)

19
Q

cirrhosis

A

represents the end stage of chronic liver disease -> normal liver cells replaced by fibrous tissue
- Alcohol
- Hepatitis
- Non-alcohol liver disease

20
Q

cirrhosis pathophysiology

A

liver has diffuse fibrosis and loss of lobular organization. Fibrosis results in decreased blood and bile flow. It is a progressive disease that results in liver failure

21
Q

cirrhosis s/s

A

Development of ascites - decreased plasma proteins and portal hypertension causes leakage from vessels into abdominal cavity
Weight loss
Weakness
bleeding

22
Q

jaundice

A

Excess unconjugated bilirubin in blood -> bilirubinemia

23
Q

prehepatic jaundice

A

excessive destruction of RBC (seen 2-3 days after birth and in people with hemolytic anemia
(not livers fault)

24
Q

intrahepatic jaundice

A

patients with liver disease (livers fault)

25
posthepatic jaundice
caused by obstruction of bile flow into gall bladder or duodenum (outflow problem) can cause kernicterus when bilirubin deposits in brain (lipophilic)
26
preicteric or prodromal period of hepatitis
insidious with fatigue, malaise, anorexia, nausea, muscle ache, fever/headache, with an elevation of serum liver enzymes (AST, ALT) if caught, liver can repair
27
Icteric phase of hepatitis
Jaundice -> obstruction urine -> darker (contains bile) feces -> lighter (lacks bile) Liver tenderness, pruritus
28
convalescent phase of hepatitis
reduction in signs/symptoms - recovery
29
damage to liver cells by hepatitis
a) direct action of the virus b) cell mediated immune response cell injury results in inflammation and necrosis
30
hepatitis
inflammation of the liver - viral or autoimmune