Feeding Specific Groups Flashcards

1
Q

Describe the energy requirements for maintenance.

A

DE (Mcal) = 0.03 x BW(kg) + 1.4
-Mcal = 1 mil cal

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2
Q

Describe factors affecting maintenance DE.

A
  1. Individual activity
    -racing, retired, restless energy
  2. Physiologic state
    -healthy, diseased, pregnant/lactating
  3. Thermal stress
    -extreme cold or heat
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3
Q

Describe energy requirements for work horses.

A

-inc maintenance (M) based on work load
-more cal based on inc M

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4
Q

Describe specific energy requirements.

A
  1. Breeding stallions
    -energy = maintenance (M) + 25%
    >protein 10%
  2. Broodmares
    -gestation
    >energy & protein inc as gestation cont
    -lactation
    >energy = M + 44%M
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5
Q

Describe performance horses.

A

-type of performance: age, temperament, intensity, climate
-inc grain ratio to meet energy needs + low roughage
-fat supplement
>1 cup oil replaces 3-4 cups of conc & 5-10lbs of grass hay
-3-4 meals per day to limit boredom
-Na, Cl, K, Mg = lost in sweat
>salt licks
>water

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6
Q

Describe aged horses.

A

-age not equal to disease
-maintain BCS 4-9
-protein, fiber, phos digestion & absorption dec w age
-common health issues: dental, parasites, arthritis
-rec: palatable, easy digested, easy masticated, fat, remove competition

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7
Q

Describe starved horses.

A

-avoid starting on a high qual diet
>refeeding syndrome
>start low, go slow
>too rapid intro to conc cal -> fatal inc in blood insulin -> cardiac & respiratory failure in 3-5d
>start low glycemic diet
>roughage only (alfalfa = high DE, protein, minerals)
>introduce at 50% of maintenance based on current BW & gradually inc for 10 d -> can inc to 125% after 10d & can add fats)
-reduce nutrient drains
>dewormer
>blanket
>stable = reduce exercise
>no competition for food
-dental, hoof, underlying disease

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8
Q

Describe equine metabolic syndrome (EMS).

A

-adult <15yrs
-obesity (BCS >7/9)
-easy keeper -> little feed
-intermittent laminitis
-PPID neg
-metabolic dysregulation
>persistent hyper insulinemia -> insulin resistance
-treatment: low glycemic diet, late cut hay, no grass pasture, grazing muzzle, avoid high NSC, exercise, levothyroxine

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9
Q

Describe pituitary pars intermedia dysfunction.

A

‘Equine cushings disease’
-pituitary adenoma
-most common disease >15yrs (85%)
>avg age is 20yrs
-all breeds
-Morgan’s & ponies
-CS
>hirsutism = long hair (doesnt shed)
>chronic intermittent laminitis
>lethargy
>abnormal fat distribution
>PU/PD

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10
Q

Describe the diagnosis & treatment of pituitary pars intermedia dysfunction.

A

DX:
-CS
-endogenous ACTH
-insulin
-dex suppression
-TRH response ACTH
TX:
-pergolide mesylate
>mimics inhibitory effect of dopamine
GOAL:
-control symptoms
-control output of hormones caused by tumor
-avoid laminitis

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11
Q

Describe feeding & concentrate of pituitary pars intermedia dysfunction.

A
  1. Feeding
    -low glycemic index
    -forage diet
    -avoid pasture high in NSC
    -feed hay low in NSC
  2. Concentrate
    -maintain BCS 4-6
    -small freq meals (0.5% of BW)
    -avoid insulin & glu deviation
    -high fiber & high fat
    -oil
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12
Q

Describe hyperlipemia.

A

-life threatening
-ponies, donkeys, mini horses
-sudden & severe breakdown of body fat stores
-56% survival rate

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13
Q

Describe hyperlipemia predisposing & precipitating factors.

A
  1. Predisposing
    -obesity
    -insulin resistant
    -female (preg, lactating)
  2. Precipitating
    -inadequate feed intake
    -stress (feed change, transport)
    -pain
    -disease
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14
Q

Describe hyperlipemia clinical signs.

A

-anorexia
-dysphagia
-colic
-Pyrexia
-encephalopathy
-depression
-weakness
-abortion
-rapid weight loss

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15
Q

Describe hyperlipemia characteristics.

A
  1. Serum conc
  2. Dehydration
  3. Anorexia
  4. Myopathy
  5. Stress leukogram
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16
Q

Describe hyperlipemia treatment.

A
  1. ID precipitating factors
    -terminate PG = stress
    -weaning = stress
  2. Secondary problems
    -dehydration = fluid therapy, electrolytes (K, Ca)
    -hypoglycemia (50% dextrose)
  3. Reduce LDLs
    -enteral nutrition (low cost)
    >fresh palatable feed (grass, apple, carrot, conc)
    -tube feeding
    >liquefied pelleted complete feeds, alfalfa pellets
    -parenteral nutrition (high cost)
    >lipid free PN effective
17
Q

Describe developmental orthopedic disease (DOD).

A

physitis, angular limb deformities, flexural deformities, osteochondorsis & OCD, cervical vertebral malformation
Key factors:
1. rapid growth
2. diet imbalance
>hyper nutrition -> excessive energy 130%
>Ca:P
>Cu & Zn deficiency
3. biomechanical stress or trauma
>too much exercise at young age
4. hormonal factors (hyperinsulinemia)
5. genetic predisposition (IMO) *
Etiology
1. High energy intake = rapid growth
2. Failure of vascularized ion
3. Irregularities of endochondral ossification
(Not proven)

18
Q

Quiz review.

A
19
Q

Describe physitis/epiphysitis.

A

-most common DOD in TB
-inflam of physis
-lead to angular limb deformities in severe cases
-distal radium & distal MCIII/MTIII most common
TREATMENT:
-dec nutritional plane - roughage only
>mineral supplement (Ca:P)
-rest
-self limiting when growth plate close
-NSAID

20
Q

Describe angular limb deformities.

A

-deviation of limb in sagittal plane
-distal radium, distal MCIII/MTIII most common
-carpal vagus & fetlock varus most common
TREATMENT (age & severity dependent)
-trimming/shoeing
-stall confinement
-surgery severe
>hemi circumferential periosteal transection & elevation
—periosteal stripping
>transphyseal bridging
—screwing

21
Q

Describe flexural limb deformities.

A

-distal interphalangeal joint (DIT)
-metacarpophalangeal joint (MCP)
-metatarsophalangeal joint (MTP)
-carpal region
1. Congenital
-ext environment factors affecting mare (inf)
-intrauterine position
2. Acquired
-rapidly growing foals
-excessive energy intake after prev inadequate energy may trigger
TREATMENT
-depends on severity, exercise, shoeing, oxytetracycline, surgery (desmotomy, tenotomy)

22
Q

Describe osteochondorsis & OCD.

A

-disturbances in endochondral differentiation, proliferation, maturation & ossification of fast growing animals
-horse, cattle, pig, dog, cat, rat
-high incidence in standardbred & warmblood breeds
CS:
-joint effusion, lameness
TREATMENT:
-arthroscopy
PREVENTION:
-maternal nutrition

23
Q

Describe cervical vertebral malformation.

A

-TB, QH, Morgan, male:female (3:1), rapid growing, ataxia, CS worse w neck extension, treatment is surgery (spinal fusion) ‘bagby basket’

24
Q

Describe orphan foal.

A

-high metabolic rate, low hepatic glycogen reserves
-freq ingestion of good qual colostrum then milk
>colostrum in first 12h
-normal foal nurse 5-7x per hr for 2 min
-foal <2mo need milk
-nurse mare ideal
-bottle/bucket feeding

25
Q

Describe orphan foal milk.

A
  1. Mare milk = best
  2. Goat milk = ok (close to mare but causes constipation)
  3. Cow milk = less fat & sugar
  4. Commercial mare milk replacer (powder/pellet)
26
Q

Describe how often to feed foals.

A

-feed hourly for 1st 48h
-then every 2h for 2 wk
-feed 10% bwt/day inc to 25% by day 10
-gastric ulcer tx
-2 wk start creep feeding
-monitory weight gain (1-2kg/d)

27
Q

Describe hand rearing VS nurse mare.

A
  1. Hand rearing
    -behavior issue
    -avoid humanization
    -dont be too nice
    -grow on replacers
    -labor intensive
  2. Nurse mare
    -2-4k
    -rebreeding cost
    -require experience
    -disease risk if another farm
    -medically induced lactation