GI Disorders Flashcards
(43 cards)
What stops diarrhea
Immodium
Who’s at risk for c-diff
Those on chemo or abx
Goals for care and nursing interventions:
Replace fluid and electrolytes
. Pharmacological management – meds? Table 45-3
3. Limit/prevent peri-anal skin breakdown
4. Manage nutritional intake
5. Prevent transmission
What does not kill C-diff
Hand santitizer
Inflammatory Bowel Disease
Autoimmune disease
Tissue damage caused by overactive, inappropriate, & sustained inflammatory response
More prevalent in industrialized regions of world
Two types of IBD
Crohns
Ulcerative Colitis
Crohns disease occurs where
Any part of the GI tract may be affected (mostly small intestine)
Inflammation involves all layers of the bowel wall (transmural)
Inflammation is discontinuous – skip lesions
SS of Crohns
*diarrhea, *abdominal pain, malabsorption and nutritional deficiencies, weight loss (severe), fever (during acute episodes),
Prognosis of Crohn’s
No curative sx
Ulcerative Colitis occurs where
Only the colon is involved
Inflammation of inner lining
Continuous inflammation from rectum upward
May appear as a fulminating crisis (severe)or as a chronic disorder.
SS of Ulcerative colitis
Bloody diarreha*abdominal pain, tenesmus (Still need to go after you’ve gone), rectal bleeding
Prognosis for colitis
Removal of large bowel is generally curative
STUDY IBD complications
Crohn’s disease complications
Intestninal
- Scar tissue, strictures, obstructure
Fistulas
Perforation, abscesses, peritonitis
Fat malabsorption - low Vit K
Extra intestinal, ;iver dx, anemia
Intestinal complications of colitis
Intestinal
*Bleeding, perforation (most often associated with toxic megacolon), *toxic megacolon, colonic dilation, *fulminant colitis, pseudopolyps
Increased risk of colorectal cancer
Extraintestinal colitis complications
Directly related to colitis, or nonspecific mediated by disturbance in immune system – anemia
Arthritis, osteoporosis, erythema nodusum, Pyoderma gangrenosum, mouth ulcers.
Toxic Megacolon
inflammation and infection cause the colon to dilate(enlarge). Walls thin as colon enlarges - loses functionality
- Cannot remove gas or feces from the body
- Can cause rupture
- Life threatening if it ruptures
Labs for IBD diagnositics
Anemia
Increased CRP (Increases with inflammation), increased WBC
Electrolytes
Stool samples
Studies (scopes) of IBD
Varium enema, colonscopy, sigmoidoscopy endoscopy
When should scopes NOT Be done in IBD
In ulcerative colitis when recturm and colon are severely inflamed
Biopsy
Malabsorption in IBD
Varies bw crohns and UC - depends on area affected
- Low albumin levels (protein in blood that is not being absorbed with IBD)
Drug therapy for IBD
Sulphasalazine (Salofalk, Dipentum etc)- locally acting anti-inflammatory
Corticosteroids
Immunosuppressive drugs (cyclosporin)
Nutrition for IBD
NPO in acute state
High-calorie, high-protein, low-residue diet with vitamin & iron supplements
Special dietary restricitons usually not necessary
Enteral supplements and TPN
Avoid fiber, brown rice, whole wheat bread
- White rice, white bread etc.
Why sx for IBD
Blockages, ruptures, tissue changes indicating cancer, exacerbations that can not be controlled