SI3 Flashcards

1
Q

Anemia is

A

Not a disease, it’s a symptom

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

Decreased Hgb

A

Often r/t to iron def or chornic blood loss

C: Very young, very old, poor diet, women in repro years

SS: Pallor, glossitis, cheilitis

Tx Tx underlying cause, iron supplements, blood trsnfusion, nutriton

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

Glossitis

A

Swollen tonguw

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

Cheilitis

A

Swollen lips

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

Defective DNA synth of RBCs

A

Patho: Often r/t folic acid and Vit B12 Def

Tx Supplemental Vit

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

Dimished availability of erythrocyte precursors

A

Often lead to aplastic anemia

C: Renal dx, autimmune malignancy

SS: high ferritin and iron stores

Rarely w/ blood transfusion, often with EPO supplement

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

Acute blood loss causing anemia

A

May lead to hypobolemic shock, internal hemorrhage or tetroperitoneal bleed

C: Caused by trauma

SS: Pain that cant be explained otherwise
- Very bad back pain in LQ

Tx: Stop bleeding, tx pt, monitor lavs

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

Hemolytic anemia

A

Destruction of RBCs at a rate that exceeds production

Extrinisc (More common)
Damaged spleen
Intrisnic: SIckle cell

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

Sickle Cell Crisis SS

A

Pain+++, swelling, pallor of mucous membranes, fever, jaundice , vasooccluisve crisis - tachypnea, HTN, NV

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

Complications of sickle cell crisis

A

Gradual involvment of all body systems - spleen, lungs kidney and brain

Prone to infection, acute chest syndrome

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

Diagnostics of sickle cell anemia

A

Peripheral blood smear, sickl8ing test, electrophroesis of hgb, MRI
Genetic

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

Nursing mgmt of sikcle cell anemia

A

Aoid hifh altritiurdes, maintain fluid intake , tret infections, help with pain control, supplemental O2

Acute ches tsyndrome: Abx, O2, Fluid, transfusion

CURE: Stem cell transplant

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

Immune Thrombocytopenic purpura

A

Abn destruction of circulating platletsSS: Gums and nose bleed, purpura (Non blanching)

Tx: Corticosteriods splenectemy

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

HIT

A

Immune system cause splts to clot in presence of heparin

SS: Heparin causes pts plts count to fall 50% + From baseline

Tx: d/c heparin

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

Disseminatied intravascular coagulation

A

Abnormally initiated and accelerated clotting

SS: Bleeding out of everywhere

Tx: Underlying cause - must diagnose quickly, often too late

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

Leukemia BE ABLE TO INTERPRET what labs would look like for leukemia

A

Common in adults

Abrupt - dramatic onset, serious infection or abn bleeding

Sky high WBC - 80s to 100s (Blasts- immature)

Hyperplasia of bone marrow and spleen

Often responds well to chemo

Low RBC and Plt count bc there’s not room in the bone marrow for that

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

ALL Leukemia

A

Common in kids

SS: Fever, bleeding, fatigue, CNS manifsestations

More difficult to tx

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

CML Leukemia

A

Genetic marker - philadelphia chromosome

Can be well controlled w/ tx

Chronic onset - swelling of spleen, liver, lymph nodes - bleeding bruising

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
19
Q

CLL

A
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
20
Q

Lymphoma vs leukemia

A

Leukemia: Originates in bone marrow/blood

SS: Blood marrow crowed by increased WBC

DIfference in location causes difference in ss

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
21
Q

Risk factors of sickling

A

Significant blood loss
Illness
Climbing/flying at high altitudes
Keeping continued stress (mental/physicaal)
Low fluid intake
Elevated temp (fever)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
22
Q

Manifestation of cirrhosis

A

Jaundice
Skine lesions - Spiders and red palms (low levels of estrogen)
Hematologoical problems
Periph neuropathy - vitamin def and malnoutrish
Esophaogeal varices causing anemia

23
Q

Know the two types of encephalopathy

24
Q

Portal vein hypertension

A

Increase in BP in protl venous sytem r’t liver blockage

SS May lead to large swollen veins that can rupture and bleed

Tx: Propranolol

25
Esophageal Varices
Collateral circulation in esophagus that is less strong SS: Red blood in vomit black stool, high HR, low BP Tx: Diagnosed with endoscopy, tx with vitamin K, pantoloc, octreotide, octaplex if acute leeting, ligation sx
26
Ascities
Accumulation of flood in peritoeal cavity SS:Abd distention, wt gain, abd striae, ss of degydration hypoK Tx : Na restriction, diurtetics, fluid removal, paracentesis, IV albumin
27
Edema is a hige risk for
Skin breakdown
28
Where can crohns be found?
Technically anywhere in GI, ususally lower SI
29
Which IBD results in non bloody stools
Crohns
30
Anemia caused by UC
Acute blood loss from ulcers (drop in Hgb) - low iron
31
Crohns caueses anemia
Reduced absorption (malabosorption)
32
Which IBD goes ALL the way through the tissue layers
Crohns
33
Which IBD has an increased risk to colorectal cancer
Colitis
34
Cobblestone appearance, inflamed, edematos mucosa with deep longitudinal lacerations is found in which IBD?
Crohns
35
Colitis colon tissue looks like
Hypermic + Edematous in affected area Abscesses turning into ulceration that bleed
36
Tx for UC
Rest bowel Control inflam Mnaage fluids and nutrition Manage pt stress Provide education ab dx and tx Provide SS relief
37
Why aren't UC pts prescribed NSAIDS
bc they increase risk for GI Bleed
38
Drug therapy for UC
Sulphasalizine Corticosteriods Immunosuppressive drugs
39
Sx for UC
Removal of protions of the bowel
40
Crohn's dx tx durg
Sulphasalazine Corticosteriods Flagyl (DIFFERENT FROM UC) - Fistulas can cause secondary infections, so treated Bio Drug
41
Which IBD spreads in continuous pattern and has presence of pseudopolyps
UC
42
Whic hematopoietic supp is used for anemia in Crohns
Cobalamin (B12) Supplements
43
With Crohn's pts are on immunosuppressant therefore infections are often only shown by
Fever
44
MS manifestation
Weaknress or paralysis of body parts Scanning speach Sight and hearing loss Bowel and bladder - Constipation, spastic bladder (Contracts) Flaccid (No desire to void)
45
HD
Lots of abnormal movements and jerking Self care deficit Cognitive decline progresses, pt eventually becomes demented and incontinent Severe mood swings Sexual outbursts Tx is palliative Haloperidol is given to reduce ss of excessibe movment
46
PD
Tremor Rigidy Akinesia Postural instability SS worse with stress
47
Tx of PD
Dopaminergic Loveodp-Cardipoa
48
To reduce fall risk in PD pts
Consciously lift feet while walking
49
PID SS
Resulting for untreated cervicitis often Low abdom pain Spotting after intercourse
50
Endometriosis
Secondary dysmenorrhea Infertility Pelic pain Painful intercourse Irreguar bleeding Painful bm Dysuria
51
Why is semi-fowlers used during PID
To drain out infection
52
Which action is taken if a pap test reveals minor cell changes
Schedule another pap test in 4 motnhs
53