Goats Flashcards

1
Q

what type of breeders are goats?

A

seasonally polyoestrous (sept-feb)

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2
Q

what is a bill rag

A

to ID if female on heat

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3
Q

who can disbud goat kids

A

vets only - cant tolerate local an, so GA! (v thin skull!)

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4
Q

what poisonous plants cause v+ in goats

A

rhododendron

pieris

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5
Q

what is the most common cause of enterotoxaemia

A

c. perfringens type d (pulpy kidney in sheep)

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6
Q

what are the signs of c. perfringens enterotoxaema

A

d++ w blood
rapid death
acute or peracute
PME - liver dark and haemorrhagic

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7
Q

what can trigger a c perfringens enterotoxaema

A
stress
illness
kidding
acidosis (gorging)
changes in housing/diet
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8
Q

how can you tx and prev enterotoxaemia from c perfringens

A

IVFT and oral FT
carprofen
vac - q6mths. lambivac

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9
Q

what are the signs of Listeriosis infection

A
encephalitis
septicaemia
pytalism (cant swallow)
droopy eyelid
droopy ear
lateral recumbancy
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10
Q

how do you tx listeria

A

abx - pot sulphonamides
NSAIDs
IVFT - often acidotic due to loss of buffer saliva
nursing care

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11
Q

what is CAE and what does it stand for?

A

caprine arthritis encephalitis,

lentivirus

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12
Q

what is the significance of the blood Ab test

A

if its +ve, its +ve for life

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13
Q

desc the signs of CAE

A

slow
not always sign - BIOS vital
arthritis - +- carpal pain
mastitis

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14
Q

how does CAE spread

A

colostrum

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15
Q

how do you tx CAE

A

you don’t - you cull

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16
Q

what are the notable signs of johnes

A
nothing outstanding, unlike cows:
poor yield
wasting
anaemia
d+ (end stage)
17
Q

is there a tx for johnes

A

gudair vaccine

18
Q

what does johnes appear like at PME in goats and sheep (not cattle!)

A

velvety mucosa

big mesenteric LN (calcified likely)

19
Q

what faecal stain IDs johens

20
Q

what is the path that causes CLA

A

corynebacterium pseudo-tb

CLA = caseous lymphadenitits

21
Q

is CLA anything to worry about

A

YEs - severe disease

rapid spread

22
Q

do milking goats get teat dipped?

A

not usually - clinical mastitis not an issue! sub clin is common however

23
Q

why isn’t the SCC rule applied to goat milk?

A

goats are apocrine milkers, cows are merocrine. apocrine means that cells MUST break away for milk to be ‘let downn’

24
Q

gooats get hypocal and ketosis just like ocws, but why is it uncommon to get mypoMg

A

they don’t JUST eat grass - other sources of Mg

25
name the 4 main causes of goat respiratory disease
- pasteurellosis (mannheimia haemolytica) - mycoplasma - lung worm (dictylocaulus filiaria & meulleria capillaria) - TB
26
how do you protect against pasteurellosis
vaccinate | use sheep vacc, or just use the 4-in-1 clostridium vacc too
27
because of so few licensed drugs, what is the cascade rule for milk withhold
7d
28
what is pygmy goat syndrome
heritable 1ry or 2ry keratinisation defect ears, eyes, nose
29
orf is....
zoonotic
30
why is PGE always a ddx for anything GI
goats never develop immunity to parasites
31
how do you tx round worm infections
double dose | get accurate kg (fat is abdominal..)
32
why are coccidia so bad
50% of goat spp are path | so its worth getting them 'speciated' soo you know
33
whats the name for false pregnancy
hydrometra
34
how do you treat hydrometra
2 doses of PGs, q12hr