L7: Neurobehavioural, Language, Comm Profiles of Aphasia P2 Flashcards

1
Q

Wernicke’s aphasia is a type of

A

fluent/posterior apahsia

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2
Q

anomia

A

word finding difficulties

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3
Q

wernicke’s aphasia presents as ____ except for periods of ______, normal to excessive ____ output, press of speech, and logorrhoea

A

fluent

anomia

verbal

logorrhoea = incoherent talking

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4
Q

wernicke’s speehc can be described as

A

semanticlaly empty, primarily functors, and info empty words (stuff, that, those)

morphosyntactic structures near normal, if abnormal, considered paragrammatic rather than agrammatic, may observe correct use of complex grammatical forms

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5
Q

paragrammatism vs agrammatism

A

para = the incorrect use of grammar

agram = omitting grammatical words

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6
Q

what are the features of paragrammatism?

A

errors may or may not occur in writing as well as in spoken output

generally well constructed syntax of sentences

substitution of grammatical morphemes

paragrammtic errors theorized to occur later in the sentence

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7
Q

in wernicke’s:

articulation and prosody are generally _____

serial spoken output is ____

_____ rate in conjunction with press of speech

_____ interuption

_______ of comm difficulties

A

unimpaired

strong

inc

resists

usually unaware

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8
Q

wernicke’s involves no verbal or oral apraxia BUT…. (2 things)

A

all types of paraphasias (generally not responsive to phonemic cues)

perseveration

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9
Q

paraphasias=

A

a substitution in speech

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10
Q

perseveration=

A

unintentional production of an info unit previously produced or heard or seen

substituted for correct response

gets stuck

often unaware

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11
Q

what are the 3 types of paraphasias and the 2 additional related types?

A

semantic

phonemic

neologistic (neologism)

extended jargon

circumlocution

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12
Q

semantic paraphasias are

A

disruption at the lexical level

semantically related or unrelated to the target word

ex. jelly fish for octopus (related), or chicken for octopus (unrelated), or chair for octopus (verbal)

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13
Q

phonemic paraphasias are

A

disruption at the phonological level

similar word or non word substitutions; 50% or more of the error overlaps phonologically w the target

octagon for octopus (real word) vs ocoput for octopus (non-word)

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14
Q

neologistic paraphasias are

A

not a word phonologically or semantically

often follows the phonological rules of the language

ex. ertig for octopus

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15
Q

extended jargon paraphasias are (not a type of paraphasic error by can be included)

A

running/connected utterances (phrases or sentences) in which includes senseless words or neologisms

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16
Q

circumlocution is

A

talking around the intended word - informative description

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17
Q

in wernicke’s reptition is

A

disturbed

may be related to severe auditory comp difficulties, contains paraphasias

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18
Q

in wernicke’s auditory comp is

A

severely impaired -understand v little

may comp simple words, phrases, sentences

system overloads easily

may have phoneme discrimination probs

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19
Q

in wernicke’s reading comp is

A

often reading parallels auditory comp deficit (connected!)

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20
Q

since reading is learned thru our auditory sys, if aud comp is impaired then…

A

reading comp impaired and/or angular gyrus sys involved; storage of symbols is disturbed and reading comp is disturbed

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21
Q

in wernicke’s reading aloud is

A

disturbed but not to the same extent as reading comp

filled w paraphasic errors (literal/phonemic and verbal/semantic)

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22
Q

in wernicke’s writing is

A

impaired on a linguistic level but not often at motoric level

paraphasic errors

lacks meaning - semantically empty

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23
Q

in Wernicke’s, they may exhibit initial paresis but…

and abulatory losses are ____, tactile losses _____….

and visual losses _____

A

transient and disappears shortly post onset

rare

can be present

may occur

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24
Q

in Wernicke’s the patients may become ____ as others focus on deficits client unable to _____

A

paranoid

see or appreciate (bc they are unaware)

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25
site of lesion for Wernicke's
wernicke's area, posterior 1/3 of STG plus inferior parietal BA 22
26
conduction aphasia is a type of
fluent/posterior aphasia
27
conduction aphasia is considered a fluent aphasia but...
verbal output limited to brief bursts of utterances
28
in conduction aphasia, conversation output is impaired due to
word finding pauses and attempts to self correct
29
in conduction aphasia, verbal output in # words/min approaches _____ vs wernicke's ____ output
normal copious
30
in conduction aphasia there are _____ paraphasic errors, _____ of phonemes is impaired
literal/phonemic selection
31
in conduction aphasia, ____ are used in the setting of impaired word retrieval
circumlocutions
32
in conduction aphasia, verbal output is facilitated by
singing and rhythmic patterning serial talk is robust if you give a starting phonemic cue
33
in conduction aphasia they have _____ intonation, _____ syntax, and _____ of errors
normal near normal aware
34
the most classic sign of conduction aphasia is... why?
repetition problems they understand what they hear but cannot transfer to Broca's area for rep purposes
35
in conduction aphasia aud comp and reading comp is ____
good (contrast aud comp w wernicke's)
36
reading aloud in conduction aphasia is ...
impaired bc info cannot be transmitted to Broca's area for verbal output
37
describe how writing is effected in conduction aphasia
impaired on a linguistic level but not often at motoric level spelling errors bc of storage disturbances to angular gyrus words omitted and interchanged in sentences
38
in conduction aphasia, there are often ___ neurological deficits, but...
no other can exhibit some
39
in conduction aphasia, motor hemiparesis and sensory disturbances are ____, and involves...
variable depends on location of lesion usually only arm or R side of face sensory loss may only involve distal portions of upper limb (fingers)
40
in conduction aphasia, the visual field is ...
involved - usually apparent either R-homonymous hemianopia or inferior or superior quadrantanopia
41
in conduction aphasia, ideomotor apraxia is inconsistent for ___ but consistent for ______ structures.... sometimes diff to separate comp probs from ___ component
limb face/oral/resp structures apraxic
42
site of lesion of conduction aphasia
interruption along the arcuate fasciculus; white matter pathways under supramarginal gyrus
43
anomic aphasia is a type of
fluent/posterior aphasia
44
wernicke's and conduction may evolve into an
anomic aphasia
45
in anomic aphasia, there is ______ naming relative to other difficulties
disproportionately greater difficulty
46
in anomic aphasia, there is ______ hesitations, grammatically ______ spoken output
intermittent well-formed
47
in anomic aphasia, ____ of substantive words, _____ of content aka "____"
absence vagueness emptiness semantically empty
48
in anomic aphasia, _____ can be vague and bizarre
circumlocutions
49
in anomic aphasia, they may say ______ phrases, ex. little things
non-specific
50
in anomic aphasia, _____ are infrequent, but semantic when present
paraphasias
51
in anomic aphasia, repetition skills are ______
good
52
in anomic aphasia, auditory comp is
relatively or entirely intact
53
in anomic aphasia, writing/reading skills..
vary along a broad spectrum spelling also varies considerably
54
in anomic aphasia, you do not usually see hard or soft...
neurological signs
55
site of lesion for anomic aphasia
least reliably localized of all aphasia syndromes; usually in L temporo parietal area and may extend into angular gyrus resulting in severe alexia and agraphia
56
alexia =
impairment in reading
57
agraphia=
impairment in ability to write
58
transcortical sensory aphasia is a type of
fluent/posterior aphasia
59
transcortical sensory aphasia is quite
rare, but w a similar profile to Wernicke's
60
in transcortical sensory aphasia, you'll see: _______ speech _____ neologisms, paraphasias, circumlocutions _____ severly impaired will not initiate _____, but evoked are ________
well articulated frequent naming utterances, fluent and empty
61
in transcortical sensory aphasia, repetition is (!!!!!!!)
excellent! (words>sentences) may be considered echolalia (involuntary rep of everything heard)
62
in transcortical sensory aphasia, auditory comp is
impaired w moderate to severe levels
63
in transcortical sensory aphasia, reading comp is
poor
64
in transcortical sensory aphasia, reading aloud has
a wide range of performance from preserved to defective
65
in transcortical sensory aphasia, writing is
poor (similar to W)
66
in transcortical sensory aphasia, there are sensory
impairments
67
transcortical sensory aphasia may involve gerstman's syndrome which requires the following 4 characs (in dispute)
R/L disorientation finger agnosia agraphia severe acalculia
68
finger agnosia =
close eyes, examiner touches fingers, client cannot tell which finger touched
69
acalculia =
acquired arithmetic deficits
70
in transcortical sensory aphasia, there is ____ awareness of the extent of impairment
poor
71
site of transcortical sensory aphasia
posterior parieto-temporal, sparing wernicke's area; parietal and temporal border zones disruption in blood supply from the PCA affecting the inferior temporal lobe and anterior occipital lobe
72
subcortical lesions may cause aphasia in 3 ways:
thalamus and BG subcortical vascular lesions (striate arteries) combination of 1 and 2
73
why would thalamus/BG damage cause aphasia?
play direct regulatory or indirect gating roles in cortical language functions via projection fibres/tracts (ex. internal capsule and cornona radiata)
74
subcortical vascular lesions (striate arteries) could cause aphasia bc
they may have widespread effect, creating hypofusion in LH perisylvian cortical regions
75
anterior capsular/putamen lesions cause a combination of
TCM and Broca's aphasia characs
76
for all subcortical variants of aphasia we will see...
borderline fluent vs non fluent
77
anterior capsule/putamen lesions cause the following spontaneous language symps
borderline fluent (6-8 words) hypophonia w poor articulatory agility variable syntax phonemic and semantic paraphasias
78
repetition in anterior capsule/putamen lesions is
relatively good
79
auditory comp in anterior capsule/putamen lesions is
relatively good
80
posterior casular/putamen lesions show a combination of
Broca's and Wernicke's aphasias
81
posterior casular/putamen lesions show the following spontaneous language symps
borderline fluent (6-8 words) hypophonia w good artic agility variable syntax phonemic, semantic, neologistic paraphasias
82
posterior casular/putamen lesions show ___ repetition
poor
83
posterior casular/putamen lesions show ___ auditory comp
poor
84
thalamic lesions = thalamic aphasia, effects thalamic projections to
both frontal and posterior cortical language zones and to motor cortex
85
thalamic lesions = thalamic aphasia may result in ____ and _____ aphasia like linguistic characteristics
TCM and TCS
86
thalamic lesions = thalamic aphasia have the following spontaneous language symps
borderline fluent (6-8 words) somewhat perseverative anomia w semantic (verbal) paraphasias intact syntax
87
thalamic lesions = thalamic aphasia show ______ repetition
relatively spared (like transcortical)
88
thalamic lesions = thalamic aphasia show ___ auditory comp
variable
89
during the acute stage, there is variability in aphasic syndromes bc
diaschisis is more extended and complex unusual aphasia manifestations due to 1) focal brain pathology and 2) extended brain dysfunction secondary to diaschisis
90
there is also variability in aphasic syndroms because the brain is a dynamic system...
with areas of interconnectivity simple brain functions localized into single brain areas diverse and simultaneous activation of complex brain functions
91
there is also variability in aphasic syndromes bc of individuality...
indv diffs in brain organization and connectivity indv life and linguistic experiences influence the organization of cog and language
92
3 reasons for variability in aphasic syndromes:
acute stage dynamic and interconnected individuality