LESSON 2 Flashcards

1
Q

Loss of tooth structure / degradative changes of tooth structure due to non-carious causes resulting in wear of tooth structure and loss of function

A

REGRESSIVE CHANGES

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2
Q

REGRESSIVE CHANGES IN ENAMEL

A

Attrition

Abrasion

Erosion

Abfraction

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3
Q

PHYSIOLOGIC wear of tooth due to tooth-to-tooth contact, mastication, or parafunction

A

ATTRITION

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4
Q

Appearance of small, polished facet on a cusp tip or ridge
or a slight flattening of an incisal edge

A

ATTRITION

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5
Q

People who like to constantly eat course or hard food that can cause the teeth to flatten out

Course foods: hard crunchy foods like chicharron

A

ATTRITION

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6
Q

ETIOLOGY OF ATTRITION

A

Diet

Age

Parafunctional occlusal forces

Contact with poorly polished ceramic restorations.

Accelerated by poor quality or absent enamel.

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7
Q

True or False

The older a person becomes; the more attrition is exhibited.

A

True

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8
Q

when the person is conscious, and they like to bite strongly on their teeth and they’re
not chewing. (Conscious: when they are stress)

A

Clenching will cause attrtion

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9
Q

when people grind. A condition wherein the person is not aware of (Unconscious:
sleeping)

A

Bruxism will cause attrition

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10
Q

TRUE or FALSE

Using hard bristles when toothbrushing for 2-3 mins can cause abrasion

A

True

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11
Q

V-shaped notches with sharply defined margins on the root side of the cementoenamel junction in teeth with some gingival recession

A

ABRASION

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12
Q

affects large areas and small polished facets at the cuspal tips

A

ATTRITION

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13
Q

Vigorous tooth brushing or flossing (and people who likes to use toothpicks)

Improper use of dental floss and toothpicks may produce
lesions on the proximal exposed root surface.

A

ABRASION

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14
Q

Habits of constant biting of hard objects

o Pen chewing/ biting.
o Habitual pipe smokers may develop notching of the teeth that conforms to the shape of the pipe stem.
o Bottle opening with teeth.
o Fingernail biting

A

ABRASION

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15
Q

loss of tooth structure due to mechanical reasons (root area)

A

ABRASION

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16
Q

Irreversible loss of tooth structure due to a chemical process (does not involve bacteria)

A

EROSION

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17
Q

Shallow, scooped-out (smooth) surfaces.

A

EROSION

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18
Q

Causes

Intrinsic:

Stomach acid reaching teeth
Obesity, Pregnancy
Excessive alcoholism
Eating disorder (bulimia nervosa, anorexia nervosa)

A

EROSION

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19
Q

Extrinsic:
o Occupational causes
People who work in factories.
Sometimes they are exposed to some chemicals. Chronically, can affect their teeth.

o Acidic foods and drinks (has low pH, high acidity)
 Citrus fruits
 Fruit juices

A

EROSION

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20
Q

a condition or erosion that affects the general lingual
surfaces of the teeth.

A

Perimolysis

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21
Q

loss of tooth structure both enamel and dentin or under the CEJ

A

Abfraction

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22
Q

True or false

PERIMOLYSIS’
- erosion at lingual surfaces due to intrinsic acid pH: 1-2)

A

True

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23
Q

• Deep, narrow, wedge-/ V-shaped only at cervical areas
• Subgingival

A

ABFRACTION

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24
Q

Abnormal biochemical (occlusal) stresses

o Swallowing, clenching, or cyclic
o Cervical lesion

A

ABFRACTION

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25
MANAGEMENT OF ENAMEL REGRESSIVE CHANGES
- Know the cause of loss of tooth structure - Immediate therapy for resolution of sensitivity and pain (desensitizers) - mouth guards - resto/prostho treatment - relax
26
the dentin formed when the tooth is still developing.
Primary dentin
27
REGRESSIVE CHANGES IN DENTIN
Primary dentin Secondary dentin Tertiary dentin
28
formed when the root is completely formed.
Secondary dentin
29
also called reparative dentin. There is a cause. (Dead tracts, Sclerotic dentin)
Tertiary dentin
30
occurs when fluid found inside dentinal tubules dry up
Dead tracts ( black)
31
occurs when dentin becomes hypermineralized
Sclerotic dentin ( white )
32
Where does dead tracts can be found?
Found in incisal and cuspal tip areas.
33
TRANSPARENT DENTIN
SCLEROTIC DENTIN
34
A regressive alteration in tooth substance that is characterized by calcification of the dentinal tubules.
SCLEROTIC DENTIN
35
Will appear as black because the dentinal tubules which is normally filled with dentinal fluid, is now empty of that dentinal fluid.
DEAD TRACTS
36
T/F In SCLEROTIC DENTIN, Dentinal tubules is filled with increased amount of mineral structures or even around your odontoblastic processes
True
37
Result of injury or manifestation of the normal aging process
SCLEROTIC DENTIN
38
True or false The presence of dead tracts and sclerotic dentin is one reason why teeth that has attrition or abrasion will not have sensitivity because the areas are protected by these two areas especially the sclerotic dentin
True
39
MECHANISMS FOR SCLEROTIC DENTIN
1. Increased deposition of intratubular dentin 2. Tubule occlusion by precipitated mineral (whitlockite) crystals
40
Fat droplet depositionk
Fatty degeneration
41
Earliest histology change that occurs int e pulp tissue
Fatty degeneration
42
Just like in vacuolization but you have an increase in intercellular fluid but this time, because of _____________ the fibers in your pulp will look like as if they’re knitted.
Reticular atrophy
43
thickening or increase in number
Pulpal Fibrosis
44
REGRESSIVE CHANGES IN THE PULP
1. Fatty degeneration 2. Vacuolization of Odontoblastic layer 3. Reticular atrophy 4. Pulpal Fibrosis 5. Denticles/ Pulp Stone
45
Hardened or mineralized structures found within your pulp area
PULP STONE / DENTICLES
46
Why are they called denticles?
Their structure on a cross section can resemble your dentinal structure.
47
o In cross section resembles dentin o Since these are mineralized structures, it appears radiopaque or white in radiographs.
True denticle
48
Doesn’t resemble dentin.
False denticle
49
Found free located in the pulp tissue.
Free
50
Part of it is in the pulp tissue, part of it is in the dentin surface
Attached
51
Completely found in dentin
Embedded
52
o Round in structure or circular o Can be found anywhere.
Nodular
53
o Irregular in shape o Can be found anywhere.
Amorphous
54
o Elongated or fibrous in form o Usually found in root canal area
Diffuse or fibrillar
55
The increase or thickening of cementum
HYPERCEMENTOSIS
56
Conditions ex. Paget’s disease
HYPERCEMENTOSIS
57
ETIOLOGY: • Accelerated tooth elongation. • Periapical inflammation • Tooth repair in trauma • Conditions ex. Paget’s disease
HYPERCEMENTOSIS
58
True or false CEMENTICLES • Can be free, embedded or attached.
True
59
Breakdown or destruction and loss of root structure of a tooth
ROOT RESORPTION
60
Is one condition that can be seen clinically because the tooth will appear pink.
INTERNAL RESORPTION
61
Sometimes called pink tooth of Mummery.
INTERNAL RESORPTION
62
Pink hue because of increase granulation of tissue within the chamber or the root
INTERNAL RESORPTION
63
True or false INTERNAL RESORPTION Needs to undergo root canal treatment
True
64
Etiology • Pulpal inflammation • Periodontal infection • Pressure from orthodontic movement, impacted tooth, or tumors. • Traumatic injuries • Presence of cementicles on root surface
EXTERNAL RESORPTION
65
increase of intracellular liquid
Vacuolization of Odontoblastic Layer
66
- occurs with time due to aging
Pulp Fibrosis
67
- difficulty in root canal treatment but not a cause for alarm
Denticles/Pulp Stones
68
calcific structures forming inside PDL space ( no clinical sognificance unless exposed to oral environment w/c can cause accumulation of dental plaque leading to cavities/periodontal disease)
CEMENTICLES
69
loss of root structure starts inside of tooth (dentin)
INTERNAL RESORPTION
70
loss of root structure starts outside of tooth (cementum)
EXTERNAL RESORPTION
71
-anything with excessive outside force may cause this - needs to undego tooth extraction
EXTERNAL RESORPTION
72
Severe jarring of a tooth with a contusion of the periodontal ligament
CONCUSSION
73
• Simplest form of traumatic injuries • The structures are still in place, but they are jilted from their normal position. • In the apical part you have a radiolucency
CONCUSSION
74
A TRAUMATIC INJURIES with no change in tooth position and no gingival bleeding
CONCUSSION
75
- dislocation of tooth from sockets - presence of sensitivity to percussion, increased mobility but no displacement
LUXATION
76
“Loosen”
LUXATION
77
- break in the continuity of dental hard tissues - will appear greyish or radioluscent
FRACTURE
78
(root has severe abrupt curves along the root) - deformity of tooth characterized by a sharp bend at the neck or root part of the tooth - happens when developing tooth undergo some form of traume during developing stage
Dilaceration
79
- forceful displacement of a tooth from the socket
Avulsion
80
True or false Avulsed teeth depending on the time can not be returned to the socket
False Avulsed teeth depending on the time can still be returned to the socket
81
A SUPERNUMERARY TOOTH extra tooth between maxillary central incisors
Mesiodens
82
SUPERNUMERARY TOOTH extra tooth found elsewhere than in the midline of the arch
Peridens
83
SUPERNUMERARY TOOTH extra teeth distal to 3rd molars
Distomolar
84
extra teeth on the buccal or lingual side of the maxillary molar
Paramolar
85
all teeth are missing, may involve both deciduous and permanent dentition
Total Anodontia
86
occurs as a result of extraction of all teeth
False Anodontia
87
sometimes applied to multiple unerupted teeth
Pseudoanodontia
88
common in central or lateral incisors; usually central incisors with a notch on the incisal edge
Hutchinson’s Teeth
89
usually found on the upper central incisors on the lingual side; these are over-developed cingulum
Talon’s Cusps
90
crossing the line of occlusion
Supraversion