Brainscape's Knowledge GenomeTM

Browse over 1 million classes created by top students, professors, publishers, and experts.


See full index

ORAL PATH > Lesson 6 > Flashcards

Lesson 6 Flashcards

1
Q

ZONES OF THE PULP

A

-› mature dentin
-› odontoblastic zone
-> cell-free zone
cell-rich zone
-› central zone

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

reaction of tooth

A

stimuli

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

explains tooth sensitivity even when pulp tissue is not diseased

A

HYDRODYNAMIC THEORY

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

HYDRODYNAMIC THEORY BY?

A

BRANNSTROM

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

stimulation of A-delta fibers

A

HYDRODYNAMIC THEORY BY BRANNSTROM

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

an external stimuli causes movement of dentinal fluid found inside the tubules causing an outward flow

A

HYDRODYNAMIC THEORY BY BRANNSTROM

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

nerve fibers extend into the dentinal tubules (______) and respond to the stimuli or movement

A

A-delta fibers

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

_______are found mainly in the central zone of the pulp

A

C fibers

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

formation of sclerotic dentin and formation of reparative or tertiary dentin

A

MILD INJURY

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

no pulp inflammation

A

MILD INJURY

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

no formation of sclerotic or reparative dentin

A

SEVERE PROLONGED INJURY

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

stimulus continues to act on pulp tissue developing into inflammation and furthermore pulp disease

A

SEVERE PROLONGED INJURY

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

pulp cannot repair and odontoblasts are damaged

A

SEVERE PROLONGED INJURY

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

a complex sequence of vascular and cellular changes of tissues in response to injury that subsides only after the effects of the causative factors have been eliminated and the damaged tissue is repaired or returned to a healthy state

A

PAIN DUE TO INFLAMMATION

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

rubor

A

Redness

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

calor

A

Heat

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

Tumor

A

Swelling

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

dolor

A

Pain

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
19
Q

function laesa

A

Loss of function

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
20
Q

irritant /stimulus

transient vasoconstriction

vasodilation due to release of inflammatory mediators

increased vascular permeability

transudation/exudation

edema

increased pulpal tissue pressure

compression of blood circulation

increased blood viscosity

stasis of blood flow

localized pulp inflammation

A

VASCULAR RESPONSE

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
21
Q

The ________ occurs first as it is what brings the inflammatory cells to the area of injury.

A

vascular response

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
22
Q

margination

diapedesis
chemotaxis
phagocytosis

A

CELLULAR RESPONSE

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
23
Q

inflammatory cells move into side of blood vessels

A

Margination

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
24
Q

movement through endothelial lining of the blood vessel

A

Diapedesis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
25
Q

attraction to the irritant

A

Chemotaxis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
26
Q

removal of irritant or stimulus

A

Phagocytosis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
27
Q

INFLAMMATION ACCORDING TO ONSET

A

Acute

Chronic

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
28
Q

exudative or immediate
response of tissue to
neutralize injurious agent

A

Acute

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
29
Q

> increase of PMNL

-> rapid course

A

Acute

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
30
Q

-> sudden and intense

short onset

A

Acute

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
31
Q

-> proliferative phase

• > persistent or long-standing

A

Chronic

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
32
Q

-May follow an acute phase or may develop from onset
- (-) pain

A

Chronic

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
33
Q

FEATURES OF THE PULP THAT PLAY A ROLE IN PULP INFLAMMATION

A
  1. LOW COMPLIANT ENVIRONMENT
  2. RESILIENT CONNECTIVE TISSUE (GROUND SUBSTANCE)
  3. PRESENCE OF ARTERIO-VENOUS ANASTOMOSES (AVA’S)
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
34
Q

> the pulp is a soft tissue surrounded by hard tissue (dentin)

when there is presence of inflammation, vasodilation causes increased flow into the area, edema, and increase in intracellular fluid leading to an increase in blood volume

-> because of that, the pulp cannot expand and there is an increase in pressure causing some sort of pain to the patient

A

LOW COMPLIANT ENVIRONMENT

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
35
Q

the ground substance is a gel-like material that becomes watery when damaged

A

RESILIENT CONNECTIVE TISSUE (GROUND SUBSTANCE)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
36
Q

in its gel-like state, it is able to stop or control the immediate spread of inflammation but when the irritant is too strong, it damages the gel-like consistency leading to further spread of inflammation

A

RESILIENT CONNECTIVE TISSUE (GROUND SUBSTANCE)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
37
Q

direct connections between arterioles and venules; no capillary bed

A

PRESENCE OF ARTERIO-VENOUS ANASTOMOSES (AVA’S)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
38
Q

open up to bring blood to the area of inflammation in order to decrease pressure in the pulp

A

PRESENCE OF ARTERIO-VENOUS ANASTOMOSES (AVA’S)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
39
Q

FACTORS CAUSING INJURY TO THE PULP

A

BACTERIA

PHYSICAL, THERMAL, MECHANICAL,

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
40
Q

-direct pulp invasion via carious -lesion bacterial toxins

A

BACTERIA

41
Q

anachoresis

A

BACTERIA

42
Q

-trauma
-iatrogenic
**etched dentin: washed and overdried
**desiccation: collapsed collagen layer
**vibration or heat

A

PHYSICAL, THERMAL, MECHANICAL, CHEMICAL, ELECTRICAL CAUSES

43
Q

-> regressive changes
-> barodontalgia or aerodontalgia

A

PHYSICAL, THERMAL, MECHANICAL, CHEMICAL, ELECTRICAL CAUSES

44
Q

galvanism

A

PHYSICAL, THERMAL, MECHANICAL, CHEMICAL, ELECTRICAL CAUSES

45
Q

increase of pressure to pulp tissue because of high altitude levels

A

barodontalgia or aerodontalgia

46
Q

contact of metal restorations

A

galvanism

47
Q

radiograph:
clearly delineated canals
(-) periapical radiolucency, resorption, calcifications

A

NORMAL PULP (PULP DISEASE)

48
Q

-› asymptomatic
-> mild or moderate response to thermal test or stimuli
-> (-) percussion, palpation, mobility

A

NORMAL PULP (PULP DISEASE)

49
Q

mild transient localized inflammatory response of the pulp

A

REVERSIBLE PULPITIS

50
Q

non-spontaneous pain or pain that occurs only when there is stimuli

A

REVERSIBLE PULPITIS

51
Q

quick, sharp pain to cold, less to hot

A

REVERSIBLE PULPITIS

52
Q

(-) percussion, palpation, tooth mobility

A

REVERSIBLE PULPITIS

53
Q

> less current with the electrical >pulp (EP) test recent dental treatment

A

REVERSIBLE PULPITIS

54
Q

RADIOGRAPHIC FEATURES
-› normal PDL and lamina dura

A

REVERSIBLE PULPITIS

55
Q

-> slight disruption of odontoblastic layer and inflammatory cell infiltration adjacent to area of irritation

A

REVERSIBLE PULPITIS

56
Q

-> pulp hyperemia
-> inflammatory cell infiltrates

A

REVERSIBLE PULPITIS

57
Q

TREATMENT
-> removal of damaged dentin >placement of liner or base

A

REVERSIBLE PULPITIS

58
Q

IRREVERSIBLE PULPITIS are:

A

ACUTE IRREVERSIBLE PULPITIS

CHRONIC ULCERATIVE PULPITIS

CHRONIC HYPERPLASTIC PULPITIS

59
Q

irreversible condition characterized by intense inflammatory response

A

ACUTE IRREVERSIBLE PULPITIS

60
Q

-> pain

Moderate to severe
Spontaneous
Intermittent
Continuous

A

ACUTE IRREVERSIBLE PULPITIS

61
Q

sharp (A-delta fibers)

dull and throbbing (C filers)

localized or referred

A

ACUTE IRREVERSIBLE PULPITIS

62
Q

-> thermal response:

immediate response to hot stimuli but may be relieved with cold stimuli

immediate response to cold stimuli

A

ACUTE IRREVERSIBLE PULPITIS

63
Q

immediate response to hot and cold stimuli but cold causes more pain than hot

A

ACUTE IRREVERSIBLE PULPITIS

64
Q

reacts to low levels of current with the electric pulp test

A

ACUTE IRREVERSIBLE PULPITIS

65
Q

-> may be (+) percussion
-> (-) palpation and tooth mobility

A

ACUTE IRREVERSIBLE PULPITIS

66
Q

RADIOGRAPHIC FEATURES

-> deep caries

> extensive restorations thickened or widening apical portion of PDL

A

ACUTE IRREVERSIBLE PULPITIS

67
Q

HISTOLOGIC FEATURES of
ACUTE IRREVERSIBLE PULPITIS

A

Early Phase

Late Stage

68
Q

What phase?

> odontoblasts near the cause are destroyed

A

Early phase

69
Q

What phase?

increased inflammatory cell infiltration

A

Early Phase

70
Q

What phase?

vascular dilation and edema

A

Early Phase

71
Q

What phase?

-> destruction of pulp zones

A

Late Stage

72
Q

What phase?

inflammation involves entire pulp

A

Late stage

73
Q

What phase?

inflammatory cells fill pulp

A

Late Stage

74
Q

TREATMENTof ACUTE IRREVERSIBLE PULPITIS

A

RCT or pulpectomy

75
Q

FORMATION OF PULP ABSCESS

A

ischemia

necrosis

degeneration of leukocytes

release of lysosomal enzymes

abscess formation

76
Q

persistent inflammatory reaction of the pulp characterized by the presence of granulation tissue over the exposed pulp surface

A

CHRONIC ULCERATIVE PULPITIS

77
Q

CLINICAL FEATURES
-> presence of carious lesion on either as a closed carious lesion or a cavity

A

CHRONIC ULCERATIVE PULPITIS

78
Q

-> in an open carious lesion, pain is present when lesion is packed with food

A

CHRONIC ULCERATIVE PULPITIS

79
Q

› possible (+) percussion

-> varied thermal response

A

CHRONIC ULCERATIVE PULPITIS

80
Q

RADIOGRAPHIC FEATURES
-› near pulp exposure or presence of pulp exposure

A

CHRONIC ULCERATIVE PULPITIS

81
Q

HISTOLOGIC FEATURES
-> localized excavation of the pulp surface
-> (+) of ulcer on the pulp surface

A

CHRONIC ULCERATIVE PULPITIS

82
Q

-> plasma cell infiltration

> drainage of the inflammatory exudate through necrotic dentin

-> inflammation remains localized because drainage prevents build-up of pressure

A

CHRONIC ULCERATIVE PULPITIS

83
Q

TREATMENT

-> RCT or extraction depending on the severity of crown involvement or restorability of the tooth

A

CHRONIC ULCERATIVE PULPITIS

84
Q

-> hyper means “increase”; plastic means “tissue”

A

CHRONIC HYPERPLASTIC PULPITIS

85
Q

excessive and exuberant overgrowth of a chronically inflamed dental pulp outside the pulp chamber

A

CHRONIC HYPERPLASTIC PULPITIS

86
Q

complete exposure of pulp tissue which responds by growing or increase in terms of number and size

A

CHRONIC HYPERPLASTIC PULPITIS

87
Q

-> the overgrowth of tissue is called a pulp polyp

A

CHRONIC HYPERPLASTIC PULPITS

88
Q

common on young immature teeth

A

CHRONIC HYPERPLASTIC PULPITS

89
Q

TREATMENT
> depends on restorability of the tooth
-> RCT or extraction

A

CHRONIC HYPERPLASTIC PULPITS

90
Q

epithelial cells from surrounding oral tissue can fall into this growing granulation tissue to form lining epithelium of polyp

A

PULP POLYP WITH STRATIFIED SQUAMOUS EPITHELIUM

91
Q
  • plasma cell infiltration
  • granulation tissue with delicate CT, filers and blood vessels
A

PULP POLYP WITH STRATIFIED SQUAMOUS EPITHELIUM

92
Q

True or false

early polyps without stratified squamous lining will bleed while mature polyps with the lining will not

A

True

93
Q

end stage of pulp inflammation

A

PULP NECROSIS

94
Q

with or without crown discoloration
&raquo_space;as the pulp breaks down, products of the cells release and cause discoloration

A

PULP NECROSIS

95
Q

-› can be partial or total necrosis

-> no presence of blood vessels in pulp

-> root canal will appear radiolucent

A

PULP NECROSIS

96
Q

TREATMENT
-> RCT or extraction
-depends on restorability of the tooth

A

PULP NECROSIS

97
Q

Bacteria from another part of the body

A

Anachoresis

98
Q

No capillary bed

A

Arterio-venous anastomoses

ORAL PATH (15 decks)

Brainscape helps you reach your goals faster, through stronger study habits.
© 2024 Bold Learning Solutions. Terms and Conditions