Packet 10 - Fluid Balance (2) Flashcards

1
Q

One of the major divalent cations in the body. Influences membrane potential and permeability, and is necessary for contraction of all muscle types.

a. ) Sodium
b. ) Calcium
c. ) Potassium

A

Calcium

One of the major divalent cations in the body. Influences membrane potential and permeability, and is necessary for contraction of all muscle types.

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2
Q

Helps regulate intracellular osmolality. Helps maintain resting membrane potential and generate action potentials in nerve and muscle tissue. Involved in regulating acid/base balance.

a. ) Sodium
b. ) Calcium
c. ) Potassium

A

Potassium

Helps regulate intracellular osmolality.

Helps maintain resting membrane potential and generate action potentials in nerve and muscle tissue.

Involved in regulating acid/base balance.

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3
Q

Regulates extracellular and vascular volume (r/t effect on osmolality). Helps maintain resting membrane potential and generate action potentials in nerve and muscle tissue. (Mostly extracellular)

a. ) Sodium
b. ) Calcium
c. ) Potassium

A

Sodium

Regulates extracellular and vascular volume (r/t effect on osmolality)

Helps maintain resting membrane potential and generate action potentials in nerve and muscle tissue.

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4
Q

Transcompartmental shifts (release of intracellular K related to burns, crush injuries, extreme exercise, or other types of cell damage).

a. ) Hyponatremia b.) Hypernatremia
c. ) Hypokalemia d.) Hyperkalemia
e. ) Hypocalcemia f.) Hypercalcemia

A

Hyperkalemia

increase in serum levels

P/C Factors:

  • Decrease in elimination
    • Renal failure
    • Aldosterone deficiency
    • K-sparing diuretics
  • Excessive intake
    • Oral or IV
  • Transcompartmental shifts
    • release of intracellular K related to burns, crush injuries, extreme exercise, other types of cell damage
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5
Q

Results in decreased membrane excitability (harder to generate action potentials).

a. ) Hyponatremia b.) Hypernatremia
c. ) Hypokalemia d.) Hyperkalemia
e. ) Hypocalcemia f.) Hypercalcemia

A

Hypokalemia

decrease in serum levels

P/C Factors:

  • Inadequate intake
  • Excessive loss
    • Renal (i.e. diuretic therapy, excessive aldosterone or glucocorticoids)
    • GI (i.e. vomiting, diarrhea)
    • Skin (i.e. heavy sweating, burns)
  • Transcompartmental shifts
    • from extracellular to intracellular, r/t pH changes
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6
Q

Acute form causes decreased membrane excitability (muscle weakness, flaccid muscles, cardiac dysrhythmias, constipation, and stoopor. Chronic form causes kidney stones.

a. ) Hyponatremia b.) Hypernatremia
c. ) Hypokalemia d.) Hyperkalemia
e. ) Hypocalcemia f.) Hypercalcemia

A

Hypercalcemia

increase in serum levels

Acute form:

  • Decreased membrane excitability (too much of the membrane stabilizer
    • muscle weakness
    • flaccid muscles (no reflexes, no muscle tone, flabby muscles)
    • cardiac dysrhythmias
    • constipation (no peristalsis)
    • stoopor (extreme lethargy)

Chronic form:

  • kidney stones

P/C Factors:

  • increased bone resorption
    • excess parathyroid hormone
    • prolonged immobility
    • some cancers
  • increase in intestinal absorption
    • excess vitamin D3
    • excess dietary calcium
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7
Q

Excessive water intake in relation to output.

a. ) Hyponatremia b.) Hypernatremia
c. ) Hypokalemia d.) Hyperkalemia
e. ) Hypocalcemia f.) Hypercalcemia

A

Hyponatremia

Decrease in serum sodium

P/C factors:

  • increase in sodium/water loss, accompanied by replacement with sodium-free fluid.
    • Skin (i.e. excessive sweating, burns)
    • GI loss (i.e. vomiting, diarrhea, tap water enemas)
    • Renal (i.e. aggressive diuretic therapy)
  • Excessive water intake in relation to output (dilutional hyponatremia)
    • Psychogenic polydipsia
    • increased intake + impaired elimination
      • kidney disease and increase in ADH levels
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8
Q

S/S include alkalosis, muscle weakness, decreased peristalsis (constipation → paralytic ileus), cardiac disrhythmias, and polyuria.

a. ) Hyponatremia b.) Hypernatremia
c. ) Hypokalemia d.) Hyperkalemia
e. ) Hypocalcemia f.) Hypercalcemia

A

Hypokalemia

decrease in serum levels

P/C Factors:

  • Inadequate intake
  • Excessive loss
    • Renal (i.e. diuretic therapy, excessive aldosterone or glucocorticoids)
    • GI (i.e. vomiting, diarrhea)
    • Skin (i.e. heavy sweating, burns)
  • Transcompartmental shifts
    • from extracellular to intracellular, r/t pH changes
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9
Q

If severe, cells can only depolarize and cannot repolarize (cardiac arrest).

a. ) Hyponatremia b.) Hypernatremia
c. ) Hypokalemia d.) Hyperkalemia
e. ) Hypocalcemia f.) Hypercalcemia

A

Hyperkalemia

increase in serum levels

P/C Factors:

  • Decrease in elimination
    • Renal failure
    • Aldosterone deficiency
    • K-sparing diuretics
  • Excessive intake
    • Oral or IV
  • Transcompartmental shifts
    • release of intracellular K related to burns, crush injuries, extreme exercise, other types of cell damage
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10
Q

Transcompartmental shifts (from extracellular to intracellular related to pH changes).

a. ) Hyponatremia b.) Hypernatremia
c. ) Hypokalemia d.) Hyperkalemia
e. ) Hypocalcemia f.) Hypercalcemia

A

Hypokalemia

decrease in serum levels

P/C Factors:

  • Inadequate intake
  • Excessive loss
    • Renal (i.e. diuretic therapy, excessive aldosterone or glucocorticoids)
    • GI (i.e. vomiting, diarrhea)
    • Skin (i.e. heavy sweating, burns)
  • Transcompartmental shifts
    • from extracellular to intracellular, r/t pH changes
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11
Q

Results in increased membrane excitability (too easy to generate action potentials).

a. ) Hyponatremia b.) Hypernatremia
c. ) Hypokalemia d.) Hyperkalemia
e. ) Hypocalcemia f.) Hypercalcemia

A

Hyperkalemia

increase in serum levels

P/C Factors:

  • Decrease in elimination
    • Renal failure
    • Aldosterone deficiency
    • K-sparing diuretics
  • Excessive intake
    • Oral or IV
  • Transcompartmental shifts
    • release of intracellular K related to burns, crush injuries, extreme exercise, other types of cell damage
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12
Q

S/S include intercellular dehydration (dry skin, dry mucous membranes, and no tears).

a. ) Hyponatremia b.) Hypernatremia
c. ) Hypokalemia d.) Hyperkalemia
e. ) Hypocalcemia f.) Hypercalcemia

A

Hypernatremia

increase in serum sodium

P/C Factors:

  • Excess water loss
    • Renal (i.e. polyuria)
    • GI (i.e. watery diarrhea)
    • Skin (i.e. increase in sweating)
  • Decreased water intake
    • Thirst defect or inability to drink
  • Excessive sodium intake
    • Oral or IV administration, near-drowning in salt water
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13
Q

Acute form causes increased membrane excitability (paresthesia, seizures, cardiac dysrhythmias, tetany [muscle spasms/twitching]), and Laryngospasms (airway obstruction). Chronic form results in osteoporosis.

a. ) Hyponatremia b.) Hypernatremia
c. ) Hypokalemia d.) Hyperkalemia
e. ) Hypocalcemia f.) Hypercalcemia

A

Hypocalcemia

decrease in serum levels

Acute form:

  • increased membrane excitability (too little of the membrane stabilizer).
    • paresthesia
    • seizures
    • cardiac dysrhythmias
    • tetany (muscle spasms/twitching)
    • Laryngospasms (airway obstruction).

Chronic form:

  • osteoporosis.

P/C Factors:

  • Decrease in intestinal absorption
    • decrease in vitamin D3 malabsorption
  • Decrease in ability to mobilize from bone
    • related to altered parathyroid function
  • Abnormal renal loss
    • renal failure
  • increase in protein binding
    • alkaline pH
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14
Q

If mild, the person experiences tingling (paresthesia) and increased peristalsis (diarrhea).

a. ) Hyponatremia b.) Hypernatremia
c. ) Hypokalemia d.) Hyperkalemia
e. ) Hypocalcemia f.) Hypercalcemia

A

Hyperkalemia

increase in serum levels

P/C Factors:

  • Decrease in elimination
    • Renal failure
    • Aldosterone deficiency
    • K-sparing diuretics
  • Excessive intake
    • Oral or IV
  • Transcompartmental shifts
    • release of intracellular K related to burns, crush injuries, extreme exercise, other types of cell damage
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15
Q

ECF is hypotonic causing fluid to move into the cells (swelling of body cells). Notice first when brain cells start to swell (neurological symptoms: lethargy, headache, nausea and vomiting, motor weakness, confusion, seizures, or coma).

a. ) Hyponatremia b.) Hypernatremia
c. ) Hypokalemia d.) Hyperkalemia
e. ) Hypocalcemia f.) Hypercalcemia

A

Hyponatremia

Decrease in serum sodium

P/C factors:

  • increase in sodium/water loss, accompanied by replacement with sodium-free fluid.
    • Skin (i.e. excessive sweating, burns)
    • GI loss (i.e. vomiting, diarrhea, tap water enemas)
    • Renal (i.e. aggressive diuretic therapy)
  • Excessive water intake in relation to output (dilutional hyponatremia)
    • Psychogenic polydipsia
    • increased intake + impaired elimination
      • kidney disease and increase in ADH levels
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16
Q

ECF is hypertonic causing fluid to move out of the cells (intracellular dehydration). S/S include dry skin, dry mucous membranes, no tears, little saliva. Begin to notice when the brain cells shrink or swell.

a. ) Hyponatremia b.) Hypernatremia
c. ) Hypokalemia d.) Hyperkalemia
e. ) Hypocalcemia f.) Hypercalcemia

A

Hypernatremia

increase in serum sodium

P/C Factors:

  • Excess water loss
    • Renal (i.e. polyuria)
    • GI (i.e. watery diarrhea)
    • Skin (i.e. increase in sweating)
  • Decreased water intake
    • Thirst defect or inability to drink
  • Excessive sodium intake
    • Oral or IV administration, near-drowning in salt water
17
Q

Increase in sodium/water loss, accompanied by replacement with sodium-free fluid.

a. ) Hyponatremia b.) Hypernatremia
c. ) Hypokalemia d.) Hyperkalemia
e. ) Hypocalcemia f.) Hypercalcemia

A

Hyponatremia

Decrease in serum sodium

P/C factors:

  • increase in sodium/water loss, accompanied by replacement with sodium-free fluid.
    • Skin (i.e. excessive sweating, burns)
    • GI loss (i.e. vomiting, diarrhea, tap water enemas)
    • Renal (i.e. aggressive diuretic therapy)
  • Excessive water intake in relation to output (dilutional hyponatremia)
    • Psychogenic polydipsia
    • increased intake + impaired elimination
      • kidney disease and increase in ADH levels