readings Flashcards
(123 cards)
heart failure
effects of beta blockers on HR and BP with rest and exercise
HR and BP both decrease with rest and exercise
heart failure
effects of nitrates of HR and BP with rest and exercise
rest: increase HR; decrease BP
exercise: increase/no change HR; decrease/no change BP
(Dilates vessels)
heart failure
effects of calcium channel blockers of HR and BP with rest and exercise
CC blockers: end with “dipine”
both decrease with rest and exercises
heart failure
effects of digitalis of HR and BP with rest and exercise
HR: decrease in pts with aFib and possibly HR
BP: no change with rest and exercise
heart failure
effects of diuretics of HR and BP with rest and exercise
HR: no change with rest and exercise
BP: no change or decrease with rest and exercise
heart failure
effects of vasodilators of HR and BP with rest and exercise
HR: increase/stay the same with R and E
BP: decrease with R and E
heart failure
effects of ACE inhibitors, Angiotensin II Blockers, and Alpha Adrenergic Blockers of HR and BP with rest and exercise
HR: no change with R and E
BP: decrease with R and E
heart failure
effects of nicotine of HR and BP with rest and exercise
HR: increase/stay the same with R and E
BP: increase with R and E
heart failure
left sided heart failure
- pathology of the LV reduces cardiac outpu leading to a backup of fluid into the LA and lungs.
- the increased fluid in the lungs produces 2 hallmark pulmonary signs of left sided HF:SOB and cough
right sided heart failure
- occurs from direct insult to the RV caused by conditions that increase PA pressure.
- increased oressure within the PA subsequently increases afterload, thereby placing greater demands on the RV and causing it to go into failure
- blood is not effectively ejected from the RV and backs up into the RA and venous vasculature, producing 2 hallmark peripheral signs: jugular venous distention and peripheral edema
ejection fraction norm range
EF: 55-75%
terminology difference of compensated vs decompensated HF
compensated: the pt’s congestive symptos can be relieved by medical intervention
decompensated: shows s/s of congestion and requires medical and pharmacological readjustment
common s/s of CHF
fatigue
dyspnea
edema (pulmonary and peripheral)
weight gain
presence of S3 heart sound
renal dysfunction
other symptoms:
- paroxysmal nocturnal dyspnea
- orthopnea
- bendopnea
describe S3 heart sound
low frequency heart sound heart in early diastoleand occrs due to poor ventricular compliance and sibsequent turbulence of blood within the ventricle
- correlated with increase left ventricular end diastolic pressures and pulmonary capillary wedge pressure
paroxysmal nocturnal dyspnea vs orthopnea vs bendopnea
- paroxysmal nocturnal dyspnea: sudden episodes of SOB occuring in the night
- orthopnea: increased SOB in the recument position
- bendopnea: presence of SOB when the pt bends fwd
NYHA heart failure classifications
- class 1: pts with cardiac disease but without resulting limitations of physical activity. ordinary physical activity does not cause undue fatigue, palpitation, dyspnea or anginal pain
- class 2: pts with cardiac disease resulting in slight limitation of physical activty. they are comfortable at rest. ordinary physical activity results in fatigue, palpitations, dyspnea, and aginal pain
- class 3: pts with cardiac disease resulting in marked limitations of physicalactivity. they are comfortable at rest. less than ordinary physical activity causes fatigue, palpitations, dyspnea or anginal pain
- class 4: pts with cardiac disease resulting in inablity to carry on any physical activity without discomfort. symptoms of cardian insuffiencey or of the anginal syndrome may be present even at rest. if any physical activity is undertaken, discomfort is increased
dyspnea scale
0- no dyspnea
1- mild, noticeable
2 - mild, some difficulty
3 - mod difficulty but can continue
4 - severe difficulty, cannot continue
angina scale
0 - no angina
1 - light, barely noticeable
2 - moderate, bothersome
3 - severe, very uncomfortable: preinfarction pain
4 - most pain ever experienced; infarction pain
hypoxemia vs hypercapnea
hypoxemia- decreased amount of oxygen in the aterial blood to the tissue
hypercapnea - increased amount of co2 within the arterial blood will develop
cor pulmonale
increased pulmonary vascular resistance 2/2 capillary wall damage and reflex vasoconstriction in the presence of hypoxemia results in pulmonary HTN and RV hypertrophy
most common type of restrictive lung disease
pulmonary fibrosis
AKA
usual interstitial pneumonia
classic signs and symptoms of interstitial lung disease
symptoms: dyspnea w activity and persistant non productive vough
signs: rapid shallow breathing, limited chest expansion, inspiratory crackles, especially over the lower lung fields, digital clubbing and cyanosis
clincal significane for a 6MWT
25-35 meters
clinical significance for gait speed
0.05m/sec