Renal Disorders 2 Flashcards

(47 cards)

1
Q

UTO

A

interference with flow of urine
causes urine to accumulate behind obstruction leading to damage to organs

backs up into kidneys

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2
Q

where can UTO’s occur

A

anywhere in UT

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3
Q

kidney stones

A

masses of crystal and protein

crystals made of stone forming substances

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4
Q

what is the most common cause of UTO

A

kidney stones

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5
Q

kidneys stones in men

A

distal- see slower streak, maybe scarring
no significant damage
above prostate, bladder affected

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6
Q

patho of kidney stones

A

crystals made up of stone forming substances trapped within urinary tract and accumulate other crystals –> stones

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7
Q

contributing factors of kidney stones

A

pH of urine- affects solubility

high urinary conc. of stone forming substance

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8
Q

types of kidney stones

A

calcium oxalate or phosphate
struvite
uric acid

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9
Q

calcium oxalate is most common in

A

men

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10
Q

etiology of calcium oxalate

A

idiopathic
inc leves of Ca causes formation
people who are immobile- Ca buildup

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11
Q

struvite is most common in

A

women

precipitated by inf

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12
Q

etiology of uric acid

A

inc urine acid production in body by diet

high purine diet, high meat, fish, poultry, gout

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13
Q

CM for kidney stones

A

pain
N/V
hematuria
s&s of UTI

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14
Q

complication of kidney stones

A

can travel, cause irritation to binary tract. jagged crystals are painful

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15
Q

diagnostic tests for kidney stones

A

urine pH
xRay of abd., IVP, ultrasound
stone analysis- determines cause

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16
Q

treatment for kidney stones

A
pain meds
anx if inf
fluids
dec dietary intake of comp. in stone
ultrasonography or laser lithotripsy
surgery
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17
Q

neurogenic bladder

A

functional obstructive condition caused by dysfunction in nerve supply to bladder. problem can occur at CNS or at level of spinal cord.

not physical obstruction

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18
Q

patho of neurogenic bladder

A

paralysis of bladder interferes with normal flow of urine and emptying of bladder

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19
Q

CM of neurogenic bladder

A

urgency/frequency
incontinence
dribbling of urine
urine retention

20
Q

diagnostic tests for neurogenic bladder

A

ultrasound
cystoscopy
urodynamic studies

21
Q

treatment for neurogenic bladder

A
bladder training
kegel exercises
catheters
manual compressions
surgery
22
Q

acute renal failure

A

rapid decline in kidney “f” leading to azotemia
when azotemia symptomatic= uremia

dec UO < 400 cc/24hrs. (oliguria)

23
Q

Bun, Cr levels for acute renal failure

24
Q

etiology of acute renal failure

A

surgery

  1. prerenal
  2. intrarenal
  3. postrenal
25
prerenal
conditions outside kidneys that impair renal bld flow and cause dec glomerularr perfusion or dec GFR
26
causes of prerenal
dec VV dec CO intravascular pooling of bld/ peripheral vasodilation inc renal vas. resistance or an obstruction
27
intrarenal
actual damage to renal tissue
28
causes of intrarenal
``` prolonged prerenal problem infection nephrotoxins intravascular hemolysis myoglobinuria ```
29
postrenal
mechanical obstruction of urinary outflow | anuria < 200ml/ day
30
causes of postrenal
calculi formation prostatic hypertrophy trauma
31
patho of acute renal failure
1. renal vasoconstriction 2. cellular edema 3. dec glomerular capillary perm. 4. intratubular capillary permeability 5. leakage of glomerular filtrate
32
stages of CM of acute renal failure
oliguric phase diuretic phase recovery phase
33
oliguric phase
``` UO < 400 ml/24hrs urinary changes fluid vol excess metab. acidosis elyte imbalance ```
34
elyte levels in acute renal failure
dec Na inc K dec Ca inc phosphate
35
diuretic phase
gradual inc output to 1-3 l/day
36
recovery phase
GFR inc
37
diagnostic tests for acute renal failure
urinalysis | renal ultrasound
38
therapeutic management of acute renal failure
treat precipitating cause correct fluid/elyte disturbances control symptoms and prevent complications
39
chronic renal failure
progressive, irreversible destruction of kidneys | nephrons destroyed, replaced with scar tissue
40
phases of chronic renal failure
1. diminished renal reserve: often undiagnosed 2. renal insufficiency: GFR < 25% of normal 3. end stage renal disease (ESRD) or uremia: GFR < 5-10 % of normal
41
etiology of CRF
most common: HTN diabetes
42
patho of CRF
adaptive response: depends on location of kidney damage | intact nephron hypothesis: remaining nephrons compensate. leads to hypertrophy of nephrons
43
CM of CRF
``` 8 types; urinary system metabolic disturbances respiratory cardiovascular gastrointestinal neuromuscular integumentary reproductive ```
44
diagnostic tests for CRF
serum BUN, Cr- inc | ultrasound, xRay- shrinkage, nothing bu scar tissue
45
treatment for CRF
pharmacological: elyte management, HTN, anemia nutritional: protein restrictions, carb and fats, fluid restrictions, Na restictions, K restrictions DIALYSIS
46
indications for dialysis
``` fluid overload hyperkalemia severe acidosis progressive uremia altered CNS "f" pericarditis ```
47
goals of dialysis
remove waste products maintain safe conc of elytes correct acidosis remove excess fluid