Respiratory syndromes I Flashcards

(44 cards)

1
Q

Which side of the heart’s cardiac output affect perfusion?

A

Right side of the heart

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2
Q

Emphysema

A

Abnormal permanent enlargement of air spaces distal to terminal bronchioles and destruction of alveolar septal walls.

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3
Q

Why is emphysema a problem?

A

Because smaller alveoli have more contact surface with capillaries per volume than larger alveoli

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4
Q

Why emphysema happens?

A

There is obstruction in the airways, expiration does not get air out and there is too much air inside the alveoli and they rupture.

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5
Q

Emphysema types

A

Alveolar

Interstitial (air in the connective tissue)

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6
Q

Emphysema causes

A
Trauma
Parasites
Tumor/mass
Chronic obstructive lung disease
Congenital bronchial hypoplasia
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7
Q

Emphysema physiopathology

A

Decreased alveolar surface -> diffusion rf
Decreased pulmonary compliance -> restrictive rf
Pulmonary hypertension -> right-sided cardiac failure

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8
Q

Emphysema clinical signs

A
Respiratory dyspnea
Dry cough
Tachycardia
Tympanic sound
Cyanosis in advanced cases
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9
Q

Pleural effusion

A

Excess fluid accumulates inside the pleural cavity

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10
Q

Pleural effusion aetiology

A
Increased hydrostatic pressure
Reduced plasma oncotic pressure
Increased vascular permeability
Lymphatic drainage obstruction
Bleeding
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11
Q

Increased hydrostatic pressure and reduced plasma oncotic pressure cause

A

transudate (clear) pleural effusion

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12
Q

Inflammation and alteration in protein metabolism cause

A

exsudate (opaque with clots) pleural effusion

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13
Q

lymphatic drainage obstruction and lymphoma produce

A

chylothorax (lymph in pleura)

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14
Q

Pleural effusion signs

A
mixed dyspnea
superficial and rapid breathing movement
overinflated chest
dull sound on percussion
reduced lung sounds on auscultation
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15
Q

What type of rf does pleural effusion produce`?

A

restrictive rf

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16
Q

Fibrosis

A

Replacement of lungs’ parenchyma by fibrous tissue/progressive scarring of the lungs.

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17
Q

Aetiology of fibrosis

A

Inhalation of toxin (asbestos, saw dust, gas)
Drugs
Autoimmune(sle)
Idiopathic

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18
Q

Physiopathology of fibrosis

A

Thickened alveolo-capillary barrier -> diffusion rf
Decreased pulmonary distensibility -> restrictive rf
Pulmonary hypertension -> right-sided cardiac failure

19
Q

Signs of fibrosis

A

Dry cough
crackle sounds
tachypnea
shallow respiratory movements

20
Q

How to tell apart fibrosis and emphysema by clinical signs?

A

Emphysema -> tympanic sound

Fibrosis -> crackles

21
Q

Mediastinal syndrome

A

Compression of mediastinal structures by any mass causing signs.

22
Q

Mediastinal syndrome aetiology

A

Mass:
tumour
hematoma
abscess

23
Q

Mass in the mediastinum can lead to

A

Cranial v cava compression
airway obstruction
sympathetic trunk, vagal or laryngeal nerve compression
oesophagus compression

24
Q

cranial v cava compression leads to

A

cr v cava syndrome

25
airway obstruction leads to
obstructive rf
26
oesophagus compression leads to
regurgitation
27
nerve compressions lead to
neurological signs
28
signs of cr v cava syndrome
edema in neck head and forelimbs
29
SYMPATHETIC TRUNK COMPRESSION
HORNER´S SYNDROME – Ptosis (upper eyelid drop) – Miosis (pupil constriction) – Enophtalmos (3rd eyelid)
30
Vagal nerve compression
cardiac activity disturbances
31
Laryngeal nerve
laryngeal paralysis
32
Pneumonic syndrome
lung parenchyma consolidates with fluid or solid
33
pneumonic syndrome etiology
pneumonia, tumor, hemorrhage
34
pneumonic syndrome physiopathology
increased acb thickness leads to diffussion rf decreased pulmonary distensibility leads to restrictive rf mismatch in ventilation/perfusion ratio (v/q) leads to increase in death space
35
clinical signs of pneumonic syndrome
signs linked to pulmonary consolidation - cough - decreased thorax motility - dull percussion - decreased vesicular murmur signs linked to infection - fever - lethargy
36
atelectasis
a decrease in pulmonary volume due to complete or partial lung collapse
37
atelectasis types
``` contraction atelectasis (lack of surfactant) resorption (obstruction) atelectasis (airway obstruction) compressive atelectasis (tumors, pleural effussion compress lung tissue) ```
38
atelectasis clinical signs
``` inspiratory dyspnea tachypnea reduced size and movement of the affected hemithorax percussion (dull sound) auscultation (no sound) ```
39
pneumothorax
abnormal accumulation of air pleural cavity
40
pneumothorax types
traumatic, spontaneous or iatrogenic
41
traumatic pneumothorax aetiology
open: - bite - gunshot closed: -due to internal breakage of trachea, bronchi or lung parenchyma
42
spontaneous pneumothorax aetiology
primary: -idipathic secondary: -due to previous pulmonary disease
43
iatrogenic pneumothorax aetiology
surgical procedures or in felines trachea breakage due to et tube
44
clinical signs of pneumothorax
- inspiratory dyspnea - tachypnea with superficial breathing - cyanosis - increased volume of affected hemothorax - decreased movement of affected hemothorax - tympanic percussion - increased auscultation sounds