Skin Integrity & Wound Care. Ch 32 Flashcards
(37 cards)
Dermis
Second layer, Elastic connective tissue, contains nerves, hair follicles, glands, blood vessels
Epidermis
Top outermost layer, regenerates quickly
Subcutaneous tissue
The underlying layer that anchors the skin layers to the underlying tissues of the body
Functions of the skin
- Protection
- Temp regulation
- Psychosocial. Pg 919
- Sensation
- Vit D production
- Immunological
- Absorption. (Increase use in medicinal patches)
- Elimination
Age related skin changes
- Skin becomes thin, easily injured, less insulating, sensation of pain and pressure is reduced
- decrease in sweat gland activity, skin becomes dryer, pruritis (itching)
- healing time is delayed
- amount of melanocytes decline, hair becomes gray-white, uneven skin pigmentation
- collagen fiber is less organized, skin loses elasticity.
- infants skin is easily injures,d subject to infection
Wound
Break or disruption in the normal integrity of the skin and tissue
Wound classification
Intentional and unintentional
Open and closed
Acute and chronic
Partial thickness,full, complex
Examples pg 923
Phases of wound healing
- Hemostasis- occurs immediately, vessels constrict and clotting begins, exudate causes swelling, incr perfusion creates heat, platelets stimulate migration of cells to next phase
- Inflammatory- lasts 4-6 days, WBCs/macrophages move to wound, ingest debris, release growth factors for healing
- Proliferation- w/in 2-3 days, last up to 2-3 wks, regenerative phase, new tissue is built, capillaries grows cross wound, thin layer epi cells and granulation tissue forms.
- Maturation-final stage, begins about 3 wks-6 mos after injury, collagen is remodeled to make skin stronger, new collagen tissue deposited, scar becomes thin, flat, white
Exudate
Plasma and blood components to leak out into the area that is injured forming a liquid
(Homeostasis)
Granulation tissue
Foundation for scar tissue development. Made of thin layer epithelial cells formed across the wound reinstating blood flow
Desiccation
Dehydration
Maceration
Over hydration
Tissue had been soaked in water/fluid
Necrosis
Death of tissue
Two Factors affecting wound healing
Local and Systemic
Local Factors affecting wound healing
Pressure- interferes with blood flow, delays healing
Desiccation- cells dehydrate forming crust cover
Maceration- over hydration
Desiccation-
Trauma-
Edema- interferes with blood supply to the area,
inadequate supply of oxygen and nutrients
Infection-toxins produced by bacterial death
interfere with healing and cause cell death
Necrosis- dead tissue; must be removed for
healing to occur
Systemic factors affecting wound healing
- Age- older adults are more likely to have one or more chronic illnesses impeding healing. Infants and young children at risk due to loose skin binding.
- Circulation and oxygen- (smoking) adequate blood flow delivers nutrients and oxygen to remove local toxins, bacteria, and other debris.
- Nutritional status-body requires adequate proteins, carbs, fats, vitamins, minerals
- Wound Condition- size, presence of infection, foreign bodies.
- Medications- corticosteroids decrease the inflammatory process, radiation depresses bone marrow function, chronic illnesses, chemotherapy, prolonged antibiotics
- Immunosupression-diseases (aids,lupus), medication (chemo), age
- Health status - diabetes
Wound complications
Infection, hemorrhage, dehiscence and evisceration, fistula formation
Dehiscence
Partial or total separation of wound layers as a result do excessive stress on wounds that are not healed.
Picture of open surgical wound sutures have come apart
Evisceration
The wound completely separates with the protrusion of the viscera through the incisional area.
(Picture of intestine coming through incision)
Fistula
An abnormal passage from an internal organ to the outside of the body or from one internal organ to another
Pressure ulcer
Wound with localized area of tissue necrosis. Acute or chronic
Most occur in older adults as a result of aging, chronic illnesses, immobility, malnutrition, fecal and urinary incontinence, and/or altered consciousness
Factors in pressure ulcer development
External Pressure- ischemia, hypoxia, edema,
inflammation, then necrosis and ulcer formation
Friction and Shear- two surfaces rub against each
other. Skin breakdown, wear and tear of tissue
layers.
Ischemia
Deficiency of blood in a particular area
Difference between friction and shear
Friction- looks like an abrasion, superficial blood vessels damaged
Shear- one layer of tissue slides over another layer. Separates the skin from underlying tissue. Vessels can tear decreasing circulation