Spinal Cord Injury Flashcards

(35 cards)

1
Q

What is the most common cause of traumatic SCI?

A

MVA (40.4%)

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2
Q

How are they classified?

A

Traumatic or non-traumatic

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3
Q

Mechanisms of injury

A

Flexion (most common in lumbar injury)
Flexion-rotation (most common in cervical injury)
Axial compression
Hyperextension
Penetrating injuries

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4
Q

Spinal shock

A

A transient period of areflexia immediately following SCI
Approximately 24 hours
Hypotension, loss of control of sweating
Goosebumps
Will eventually lead to hyper reflexia (UMN S&S)

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5
Q

Tetraolegia

A

All four extremities
Lesions of Cx SC
56%

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6
Q

Paraplegia

A

Tx Lx L2 (caudal equina)
46%

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7
Q

How is SCI standardized

A

international standards for neurological classification of SCI *ISNCSCI

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8
Q

Neurological level of injury

A

Most caudal level of SCI level with INTACT motor and sensory fxn

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9
Q

Motor and sensory level

A

Most caudal level Intact

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10
Q

Complete and incomplete SCI

A

Complete is every paralysis below neurological level
Incomplete are those with some persevered function (zones of partial preservation)

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11
Q

ASIA impairment scale

A

A- complete
B- incomplete: has sensory
C- incomplete: has sensory and motor but muscle grade is less than 3
D- incomplete: has sensory and motor but muscle grade is more than 3
E- normal

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12
Q

Clinical syndromes

A

Incomplete
- brown-sequard
- anterior cord
- central cord
- posterior cord

Other
- conus medullaris
-caudal equine

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13
Q

Brown- Sequard syndrome

A

Damage to one half of the spinal cord (usually penetrating injury)
Ipsilateral loss of:
- all sensory modalities at the level of lesion
- motor function (descending: lateral corticospinal tract)
- proprioception, discriminative touch, and vibratory sense (ascending- dorsal column)

Contrateral loss:
- pain and temperature (spinalthalamic tract)

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14
Q

Anterior cord syndrome

A

Commonly due to flexion injuries
Loss of motor fxn, pain and temp below level of lesion

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15
Q

Central cord syndrome

A

Hyperextension in Cx- compressive forces cause edema
Loss of motor > sensory
motor loss UL>LL

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16
Q

Posterior cord syndrome

A

Loss of proprioceptions, pressure, and vibratory sense
NO motor loss

17
Q

Caudal Equina

A

Damages to the nerve roots below L1
Flaccid paralysis
LMN injury, areflexive bowel/ bladder, and sacral anesthesia

18
Q

Autonomic Dysreflexia

A

Sympathetic over activity in the body
Typically in lesions above T6
EMERGENCY SITUATION

19
Q

Pathophysiology of autonomic dysreflexia

A

Noxious stimulus, increased sympathetic outflow, wide spread vascoconriction (Increase HR an BP), basorecetirs stimulate increase in fatal output causing decreased HR, but insufficient to counteract Increased BP

20
Q

Most common triggers of AD

A

Bladder and bowel distenson/irritation
Lots more

21
Q

AD S&S

A

Hypertension
Initial tachycardia but then bradycardia
Severe headache
Profuse sweating
Increased spasticity/ hypertonia
THERES MORE

22
Q

AD interventions

A

Sit patient up to decrease BP
Notify nearby nurse or doc
Check catheter for kink, block or fullness
Loosen tight clothing
Look for other potential noxious stimulus below NLI
Document

23
Q

Functional outcomes: NLI C1-4

A

Most severe
Paralysis of arms, hands, trunk and legs
Require assistance with breathing and secretion clearance
Dependent in all ADLs
Dependent in transfers
Power wheelchair

24
Q

Functional outcomes: NLI C5

A

Can breathe but labored because lack of abdominal tone so no diaphragm counterpressure
Dependent in transfers
Manual wheelchair with propulsion aids for short distances
Can drive a van using adaptive hand controls
Power wheelchair with adapted joystick for communities

25
Functional outcomes: NLI C6
Tenodesis grasp allows for limited self-care activities Independent to min assist with sliding board Independent with manual cough Wheelchair propulsion possible with the use of hand rim projections for short distances Power wheelchair for community Independent with pressure relief maneuvrss in wheelchair Can drive a car or van using adaptive hand controls Capable of living independently
26
Functional outcomes: NLI C7
Easier for sliding board transfers Most ALDs are possible Manual wheelchair with friction surface hand rims
27
Functional outcomes: NLI C8
Independent more No wheelchair adaptions needed
28
Functional outcomes: NLI T1-T12
The Lower the lesions level the better the trunk control HKAFO and KAFO for short distances Wheelchair for comminutit
29
Functional outcomes: NLI L1-3
Same as previous
30
Functional outcomes: NLI L4-S1
AFO with assistive device. NLI L4 may choose to use wheelchair for long distances
31
Respiratory management
- IPPV - deep breathing exercises - Glossophryngeal breathing (frog breathing) - Respiratory muscle strengthening - Assisted cough - Abdominal binder
32
Skin care
Positioning - every two hours Pressure relief - maneuvers every 15 ins Skin inspection Education Wound care
33
Early strengthening and ROM
Perform daily (except for areas that are contraindicated) Pelvis should be let in neutral LSP injury- SLR >60 deg and hip flex ion > 90 degs Tetraplegic- mvmt of the head/neck, and shoulder flex ion/abduction >90 deg is contraindicated until given orthopedic clearance Selective stretching - tightness in certain muscles can enchanted function - adequate length in certain muscles can enhance function Splinting (intrincsic plus splint- hamburger hands)
34
Early mobility
May experience postural hypotension Focus on transfers and functional mobility
35
Active rehab
Continue with resp care, skin and ROM Strengthin Cardiovascular endurance training Wheelchair skills Bed mobility skills Balance Transfers Gait training