w4 Flashcards

(36 cards)

1
Q

what is the what stream

A

temporal/ ventral

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2
Q

what is the where stream

A

parietal/ dorsal

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3
Q

lesions in the what stream lead to

A

specific impairments in object recognition

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4
Q

lesions in the where stream lead to

A

deficits in spatial attention

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5
Q

what is simultanagnosia

A

unable to focus attention on more than one object at a time

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6
Q

what is Balint’s syndrome

A

damage to parietal cortex leading to simultanagnosia and problems combining features of a stimulus

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7
Q

what has been found about the parietal cortex and feature binding

A

during conjunction search, posterior temporal cortex and parietal cortex show increased activation over baseline control conditions

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8
Q

what does TMS to parietal lobe disrupt

A

conjunction search, but not feature search

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9
Q

what is hemispatial neglect

A

lack of awareness of stimuli presented to the side of space on the opposite side to the brain damage

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10
Q

what is extinction

A

Patients detect a single stimulus presented to one visual field, but fail to detect the same stimulus when another stimulus is simultaneously presented to the other field

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11
Q

how much processing happens with objects in hemispatial neglect

A

a lot of basic perceptual processing happens to things in the neglected field

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12
Q

there is a priming effect with objects in the neglected field- what does this mean

A

An object presented to the neglected field can change the patient’s behaviour, so the object has been semantically processed

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13
Q

where would a lesion underly neglect

A

Right inferior parietal lobe

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14
Q

what are neglect and extinction deficits of

A

deficits of attention not
perception

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15
Q

what is neglect due to impairment of

A

the stimulus-driven system

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16
Q

what system is intact for neglect patients

A

endogenous orienting system

17
Q

when are neglect patients most impaired

A

when trying to disengage attention from intact side

18
Q

neglect/extinction has most prevalence after

A

right brain damage

19
Q

which type of attention has evidence suggested has disproportionately more problems

A

exogenous attention

20
Q

what is ADHD characterised by

A

hyperactivity, impulsivity, and inattention beginning in childhood

21
Q

ADHD is 3x more common in

22
Q

prevalence of ADHD in adulthood

23
Q

symptoms of predominantly inattentive ADHD

A

Difficulty in finishing a task, following instructions; easily distracted

24
Q

symptoms of predominantly hyperactive ADHD

A

Difficulty in sitting still for long periods; fidgeting; speaks or acts at inappropriate times

25
most common ADHD subtype
Combined inattentive and hyperactive
26
what iOS the heritability of ADHD
parents/siblings of a child with ADHD 5x more likely to be diagnosed with ADHD
27
which factors make ADHD more likely
- Pre-natal exposure to alcohol and nicotine * Premature birth and low birth weight * Perinatal brain injury * Environmental toxins e.g. lead, pesticides
28
there is a 3% reduction in volume of which area in ADHD patients
overall cortical and especially prefrontal
29
where is there reduced grey matter in ADHD patients
fronto-parietal attention network
30
where do ADHD patients have reduced cortical connectivity
between hemispheres and within fronto-parietal attention networks
31
where do ADHD patients have hyperactivity
prefrontal cortex, especially dACC
32
molecular differences in ADHD patients
Imbalance in dopamine and noradrenaline circuits
33
what does reduced volume, activity, and connectivity in brain regions in ADHD patients lead to
the endogenous attention system and executive functions
34
which drugs are commonly prescribed for ADHD
Methylphenidate (e.g. Ritalin) and Dextroamphetamine (e.g. Attentin)
35
how do medications work to treat ADHD
by blocking the reuptake of norepinephrine (NOR) and dopamine (DOP) and facilitating their release, enhancing availability in prefrontal cortex and basal ganglia
36