BLue 1-4 Flashcards

(35 cards)

1
Q

Difference between PACS, PAC and PACG

A

PACS
>= 2 quadrants of iridotrabecular contact,
Elevated IOP +/- peripheral anterior synechiae
No glaucomatous optic neuropathy

PAC
>= 2 quadrants of iridotrabecualr contact
Elevated IOP +- PAS
No gluacomatous optic neuropathy

PACG
>= 2 quadrants of iridotrabecular contact
Elevated IOP +/- PAS
Glaucomatous optic neuropathy

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2
Q

Fucntion of PGA in glaucoma

A

Increased uveoslceral outflos

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3
Q

Beta blocker MOA in gluacoma

A

Reduces aqueoud production

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4
Q

CAI MOA in gluacoma

A

Reduced aqueous production

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5
Q

Alpha agonist MOA in gluacoma

A

Increase uveosclearl outflow
REduced acquoeous production

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6
Q

What does pilocarpine do in gluacoma

A

Increased conventional outflow.

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7
Q

What hsould OHT be reviewed if no COAG, no control of IOP

A

1-4 months

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8
Q

OHT uncertain COAG, Control of IOP

A

6-12 months review

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9
Q

OHT No converstion

A

Control of IOP
12-18 month review

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10
Q

What happens in aqueous misdirection

A

Aqueous drainage into the vitreous cavity reuslting in anteiror displacement of the vitreous, ciliary body and lens with subsequent secondary angle closure

Myopic shift
Raised IOP
Shallow/flat AC in abscence of pupil block

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11
Q

Management of aqueous misdirection

A

Avoid miotics

Atropine can encourage posterior rotation of ciliary body

YAG anteiror hyaloidotomy though extisting PI

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12
Q

What is lens particle glaucoma?

A

Secondary open angle glaucoma caused by inflammation of lens particles after breach in lens capsule form surgery or traum

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13
Q

What is phacomorphic glaucoma

A

Secondary angle closure glaucoma caused by large cataractous lens with narrowing and blockage of AC angle

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14
Q

What is phacolytic glaucoma

A

Secondary open angle glaucoma caused by leakage of soluble lens proteins of hypermature cataract into AC causing trabecular obstruction (no trauma or surgery)

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15
Q

What is phacoanaphylactic glaucoma

A

Secondary open angle glaucoma caused by granulomatous inflammatory reaction to lens antigen after trauma or post op lens retention

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16
Q

What is Irvine Gass syndrome? Riskf factors

A

Post operative CMO

Diabetes
RVO
ERM
Macular hole
Uveitis

17
Q

How should power of lens be adjusted for sulcus placement

A

Reduce power by 0.5-1D

If optic capture, can use same power

18
Q

What are causes of myopic refractive surprise?

A

Previous hyperopic laser
Higher A constant
Retained visco behind IOL

Poor biometry, incorrect IOL calculation, poor wound construction
Incorrect IOL positioning

19
Q

What are causes of hypermetropic refractive surprise

A

Myopic laser
Lower A constant
Undiagnosed staphyloma

Poor biometry, incorrect IOL calculation, poor wound construction
Incorrect IOL positioning

20
Q

What lens changes occur in homocystinuria?

A

Autosomal recessive metabolic disorder of methionine

Abnormal accumulation of homocysteine

Defeiceincy of cysthionine b-synthase

Inferonasal lens subluxation wtih no trauma

Avoid GA due to increased risk of thrombosis

21
Q

What is lens change in MArfans

A

Ectopia lentis
Superotemporally

22
Q

Sign in WHO checklist

A

Identity
Site marked
Anaesthesia
Allergy
Airway
Postiioning/draping
Warfarin
Tamsolusin/alphablocker

Pre op VTE

23
Q

Time out WHO checklist

A

Team members introduced
NAme
Procedure
Which eye
Refractive outocme
Lens model and power
Correct lens implant present
Any special equipment
Any variation to standard procedure
Spare lens
Patient concerns anaesthetic
ASA grade
Special monitoring
Sterility confirmed
Equipment issues or concerns

24
Q

Sign out

A

Name and side of procedure recorded
Instruments, swabs, sharps counts complete and correct
Any equipment problems
Variations to standard recovery and discharge protocol

25
PXF angles
Narrower
26
When is CTR effective in managing zonular dehiscence
LEss than 3 clock hours
27
What can PXF cause during cataract surgery
Poor pupil dilation Zonular instability
28
What management for PXF before cataract surgery
Extensive discussion with patient pre-op to be aware of issues Pupil manipulation Adequate hydrodissection - minimise torsional forces on zonules during lens rotation Minimal phaco energy CTR if zonular dehiscence VR team if phacodonesis at slit lamp Gentle slow movement of instruments
29
Who is reverse pupil block more common in? How do you resolve this?
Myopes Second instrument to lift iris anteriorly relieving the pupil block
30
What reduces risk of PCO
Square edge design IOL optic Haptics with flexible arms and posterior flexion Complete overlapping of anterior capsulorrhexis and anterior surface of IOL optic Acryllic hydrophobic lens
31
Effect of silicone oil in pseudophakic eyes? Phakic? Aphakic?
Pseudophakic and phakic - Silicone oil fills vitreous cavity and concavity of the anteiror part at interface with posteiror lens render eye more HYPERMETROPIC (concave lens) IN aphakic Convexity of lens at anteiror part of silicone oil acts as convex lens making eye less hypermetropic
32
What is Toxic Anterior Segment Syndrome
Sterile post op inflammatory reaction caused by non-infectious substances that enter through anteiror segment resulting in ifnlammatoin and toxic damage to intraocualr tissue Starts within 24 hours (compared to 3-7 days fo POE) Severe ey epain SEvere AC ifnlammation Diffuse corneal oedema NEgative micro Improves with topical steroids
33
What is associated with Fabry's diseasae
X linked lysosomal disease Cortex keratopathy PSC Tortuous conj/retinal vessels
34
What is seen in Alports disease
Anteiror lenticonus (protrustion of anterior lens capusle) Nephritic haematuria Deafness
35
FLACS vs standard
Same in VA, AC/PC tears, Post op CMO, IOP, patient reported outcomes and cost effectiveness