cardiac Flashcards

(63 cards)

1
Q

blood flow of heart

A

superior and inferior vena cava (deoxygenated)–R atrium–R ventricle–pulmonary artery–goes to lungs and becomes oxygenated–pulmonary veins (oxygenated now)– L atrium–L ventricle–aorta– body

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2
Q

preload

A
blood returning to the R side of the heart
muscle stretch (strength) that the volume causes
ANP released during stretch

increased preload = increased strength (workload)

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3
Q

afterload

A

pressure in aorta and PERIPHERAL arteries that the L ventricle has to pump against to get blood out

aka resistance

high afterload = low cardiac output and low forward flow

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4
Q

stroke volume

A

amount of blood pumped out of the ventricles with each beat

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5
Q

cardia output

A

HR x SV

tissue perfusion is dependent on adequate CO

CO changes according to body’s needs

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6
Q

factors that affect CO

A

HR
arrhythmia (brady and tachy)
blood volume (less volume = less CO) (more volume = more CO)
decreased contractility

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7
Q

meds that affect preload

A

vasodilator/diurese to decrease preload

diuretics (furosemide)
nitrates (nitroglycerin)

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8
Q

meds that affect afterload

A

vasodilator to decrease afterload

ACE inhibitors (enalapril) “pril”
ARBS (losartan) “sartan”
hydralazine
nitrates (nitroglycerin)

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9
Q

meds that improve contractility

A

inotropes (dopamine) “amine”

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10
Q

meds that control rate

A
beta blockers (propranolol) "olol"
calcium channel blockers (diltiazem, verapamil, amlodipine)
digoxin
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11
Q

meds that control rhythm

A

antiarrhythmics (amiodarone)

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12
Q

patho of low CO

A
will not perfuse properly 
LOC goes down
chest pain
crackles
SOB
cold and clammy
urine output goes down
weak peripheral pulses
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13
Q

arrhythmias that are always a big deal

A

pulseless vtach
vfib
asystole

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14
Q

coronary artery disease

A

broad term

chronic stable angina
acute coronary syndrome

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15
Q

chronic stable angina patho

A

intermittent decreased blood flow
ischemia so pain/pressure in chest
low O2 r/t exertion
rest or nitro to relieve pain/pressure

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16
Q

chronic stable angina treatment

A

nitroglycerin
beta blockers
calcium channel blockers
acetylsalicylic acid (aspirin)

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17
Q

nitroglycerin

A

causes venous and arterial dilation
decreased preload and afterload
dilation of coronary arteries which increases blood flow to the heart muscle (myocardium)

take 1 q 5min x 3doses

DONT SWALLOW
keep in dark, glass bottle, dry, cool
will get a headache
renew every 6 months
renew spray every 2 years

BP drops so make them sit when taking

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18
Q

beta blockers

A

prevention of angina

check BP and pulse before giving
“olol”

decrease BP, pulse, and myocardial contractility
decreases workload of heart
CO decreases

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19
Q

calcium channel blockers

A

prevention of angina

"pine"
nifedipine
verapamil
amlodipine
diltiazem

decrease BP
vasodilate arterial system
dilate coronary arteries
decrease afterload and increase O2 to the heart muscle

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20
Q

acetylsalicylic acid

A

aka aspirin

dose determined by provider

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21
Q

teaching for chronic stable angina

A
rest
avoid overeating
avoid excess caffeine
avoid drugs that increase HR
wait 2 hours after eating to exercise
dress warm in cold weather
nitro prophylactically 
smoking cessation
weight loss
isometric exercise
reduce stress
*****do everything you can to decrease the workload of the heart
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22
Q

cardiac catheterization

A

ask if they are allergic to iodine or shellfish

check kidney function because dye is excreted through the kidneys

hot shot (warm, flush, sweaty)
palpitations are normal
get baseline VS and 5Ps

after:
monitor VS
watch puncture site **for bleeding and hematoma formation
assess extremity distal to puncture site (5 Ps)
puleslessness
pallor
pain
paresthesia
paralysis
temp CRT
bedrest– flat, extremity straight x4-6hours
**
*major complication is bleeding/hemorrhage
report pain ASAP
hold metformin 48hours after

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23
Q

unstable chronic angina

A

impending MI

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24
Q

acute coronary syndrome patho

A

aka MI
aka unstable angina

decreased blood flow to myocardium– ischemia and necrosis
can happen at any time
rest and nitro do NOT relieve pain

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25
acute coronary syndrome symptoms
pain (crushing) pressure radiating to the L arm and L jaw N/V pain between shoulder blades ``` women: GI s/s epigastric discomfort pain between shoulders aching jaw or choking sensation unusual fatigue inability to catch a breath ``` ``` ****SOB is the number one sign for elderly cold, clammy BP drops CO down ECG changes vomiting ```
26
labs for troponin isomers
troponin T <0.10 | troponin I <0.03
27
stemi vs nstemi
*****worry about stemi client stemi: ST segment elevation myocardial infarction having a heart attack get them to cath lab for PCI (percutaneous coronary intervention) in less than 90min nstemi: non-st segment elevation myocardial infarction less worrisome
28
labs for acute coronary syndrome
CPK-MB cardiac specific isoenzyme high w/ damage to cardiac cells elevates within 3-6hours and peaks in 12-24 hours troponin: cardiac biomarker w/ high specificity to myocardial damage elevates within 3-4 hours and remains elevated for up to 3 weeks myoglobin: increases with 1 hour and peaks in 12 hours negative results are a good thing troponin is the most sensitive for an MI or delays seeking care
29
untreated arrhythmia that can kill
pulseless vtach vfib asystole
30
vfib
treatment: defibrillate epinephrine or vasopressor amiodarone or lidocaine
31
lidocaine toxicity
neuro changes
32
amiodarone
anti-arrhythmic of choice | SE: hypotension
33
meds used for chest pain in ED
O2 if less than 90 aspirin (chewable) nitro morphine Head up to decrease workload and increase CO
34
thromboyltics
used to dissolve a clot that is blocking blood flow to the heart muscle and decrease the size of the infarction alteplase tenecteplase reteplase streptokinase should be given within 6-8 hours after onset of pain ****bleeding is a major complication obtain bleeding hx bleeding precautions during and after administration draw blood when starting IVs to decrease pokes do not do ABGs r/t high risk for bleeding
35
common meds that require bleeding precautions
``` abciximab acetaminophen acetylsalicyclic acid apixaban clopidogrel dabigatran enoxaparin sodium eptifibatide heparin rivaroxaban warfarin ```
36
PCI (percutaneous coronary intervention)
percutaneous transluminal coronary angioplasty stents angioplasty complication: MI if any problems-- surgery chest pain after procedure-- call provider r/t reoccluding
37
antiplatelet meds
``` acetylsalicylic acid clopidogrel prasugrel abciximab eptifibatide ```
38
coronary artery bypass graft
aka open heart surgery used for multiple vessel disease or coronary artery occlusion L main coronary artery supplies the entire L ventricle ****L main coronary artery occlusion-- think sudden death or widow maker
39
cardiac rehab
``` smoking cessation stepped care plan (increase activity gradually) low fat low salt low cholesterol no isometric exercise no valsalva no straining no suppository sex resumed 7-10days safest time for sex is 8-9am walk teach s/s of HF (weight gain, ankle edema, SOB, confusion) ```
40
heart failure causes
``` complication from cardiomyopathy valvular heart disease endocarditis acute MI *****hypertension ```
41
left sided heart failure s/s
blood not moving forward into aorta and go back into lungs ``` pulmonary congestion dyspnea cough blood tinged frothy sputum restlessness s-3 orthopnea nocturnal dyspnea ```
42
R sided heart failure s/s
not moving into lungs back into venous system causes: normally a disease in lungs (PE, COPD) ``` distended neck veins edema enlarged organs weight gain ascites ```
43
systolic heart failure
heart can't contract and eject
44
diastolic heart failure
ventricles can't relax and fill
45
heart failure diagnosis
BNP: sensitive indicator if on Nesiritide turn off for 2hr prior to drawing secreted by ventricular tissues in the heart when ventricular volumes and pressures are increased CXR: enlarged heart pulmonary infiltrates (fluid/edema) echocardiogram: looks at pumping action or ejection fraction of heart ``` New York Heart association functional classification of persons with HF: class 4 is the worse "can they complete activities of daily living test" ```
46
meds for HF
``` ace inhibitors: drug of choice suppress RAS prevent conversion of angiotensin 1 to 2 results in arterial dilation and high SV SE: dry, naggy cough ``` ARBs: block angiotensin 2 receptors cause low arterial resistance and low BP ``` beta blockers: first line therapy relax vessels decrease BP decrease afterload decrease workload of heart ``` ACE and ARBs both block aldosterone so we lose Na and H2O and retain K most likely sent home on ace inhibitor and/or beta blocker because they decrease workload on the heart and prevent vasoconstriction to increase CO and keep blood going forward digoxin monitor for toxicity sinus rhythm or afib and has accompanying chronic HF makes contractions stronger slows HR (gibes ventricles more time to fill with blood) high CO high kidney perfusion good to diurese r/t HF pts cannot handle extra fluid ****HF pts should not get whole blood-- only give what they need (RBCs, platelets) ``` diuretics: furosemide hydrochlorothiazide bumetanide spironolaction decreases preload give in AM ``` severe HF: IV inotropes (milrinone, dobutamine) vasodilators (nitroprusside, nesiritide, nitroglycerin)
47
digoxin nursing considerations
``` good to diuresis working when CO goes up s/s of toxicity: early-- anorexia, N/V late-- arrhythmias and vision changes (yellow halos) ``` check apical pulse (5th ICS mid clavicular line) before giving monitor electrolytes (hypokalemia + dig = toxicity)
48
normal dig levels
0.5-2 toxicity if over 2
49
other treatment for heart failure
low Na diet: decreases fluid retention and helps decrease preload watch salt substitutes (contain excess K) elevate HOB daily weight (report 2-3lbs) s/s of recurring failure pacemaker
50
pacemaker
"natural" = SA node or sinus node sends impulses to make heart contract used to increase HR with symptomatic bradycardia ****always worry if HR drops below set rate demand pacemaker sets in only when they need it fixed rate fire at a fixed rate constantly rate should never decrease--okay to increase post procedure monitor incision common complication: electrode displacement immobilize arm assisted passive ROM to prevent frozen shoulder don't raise arm higher than shoulder ``` s/s of malfunction capture: no contraction follows stimulus sense: fires at inappropriate times may not be programmed right electrodes can dislodge battery may be dead watch for s/s of low CO or low HR ``` ``` teaching: check pulse daily ID card/bracelet avoid electromagnetic fields avoid MRIs ```
51
pulmonary edema patho
risk factors: IV fluid fast young/old hx of heart or kidney disease fluid backing into the lungs heart cannot move the volume forward usually occurs at night during bed
52
pulmonary edema s/s
``` sudden onset breathless restless/anxious hypoxia productive cough ```
53
pulmonary edema treatment
O2-- keep above 90 upright position legs down improves CO promotes pooling in lower extremities prevention: lung sounds avoid FVE
54
meds for pulmonary edema
diuretics: furosemide diuresis & vasodilate and traps more blood out in the arms and legs and reduces preload nitroglycerin: vasodilation decrease afterload = increase CO because heart is pumping again less pressure and more blood moved forward morphine decrease preload and afterload nesiritide IV infusion short term-- no more than 48hours vasodilates veins and arteries and has a diuretic effect turn off infusion 2 hours before drawing BNP
55
cardiac tamponade patho
blood, fluid or exudates leak into pericardial sac results in compression of the heart
56
cardiac tamponade s/s
``` low CO (ventricle is being squeezed) *****high CVP *****low BP muffled or distant heart sounds distended neck veins pressure in all 4 chambers shock narrowed pulse pressure (difference between systolic and diastolic reading) ```
57
narrow vs wide pulse pressure
narrow: cardiac tamponade wide: increased ICP
58
cardiac tamponade treatment
pericardioentesis to remove blood from the heart | surgery
59
arterial disorders patho
if you have atherosclerosis in one place you have it everywhere ``` medical emergency if occluded numbness and pain cold extremity no palpable pulse more symptomatic in lower extremities ****intermittent claudication (arteries only) arterial blood isn't getting to the tissue--> coldness, numbness, ***Decreased peripheral pulses, atrophy, bruit, skin/nail changes, and ulcerations pain at rest means severe obstruction ``` ***artery issues = something is not getting O2
60
arterial disorder treatment
pain will increase if you elevate legs angioplasty endarterectomy
61
venous disorders
****arteries carry oxygenated blood ****veins carry deoxygenated blood inflammation and chronic ulcers can occur could develop a DVT V: elevate veins A: dangle arteries
62
chronic arteria insufficiency s/s
intermittent claudication pain (pain progresses at rest) decrease or absent pulses pale when elevated red with lowering of leg cool mild or absent edema thin, shiny, loss of hair over foot/toes, nail thickening ulceration if present will involve toes or areas of trauma on feet (painful) gangrene may develop don't use compressions
63
chronic venous insufficiency s/s
``` none to aching pain normal pulses (may be hard to palpate r/t edema) normal color (may see petechiae or brown pigmentation with chronic) normal temp edema brown pigmentation around ankles possible thickening of skin scarring ulceration if present on sides of ankles no gangrene compression ```