CLPS 1700 - Chapter 8 Flashcards

(40 cards)

1
Q

What is hysteria?

A

Emotional condition: extreme excitability and bodily symptoms for which there is no medical explanation (331); not a DSM IV TR disorder

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2
Q

What does dissociation mean?

A

The separation of mental processes, such as perception, memory, self-awareness, that are normally integrated (331)

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3
Q

What’s the difference between mental processes in dissociative disorders as opposed to, say, schizophrenia?

A

In DD, the mental processes are intact, but not integrated properly. In schizophrenia, the mental processes themselves are corrupted (332)

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4
Q

What are the four main dissociative symptoms?

A

Amnesia, identity problems, derealization (real world perceived as strange/unreal), and depersonalization (viewing oneself as an observer) (332)

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5
Q

What are the four types of dissociative disorders?

A

Dissociative amnesia, dissociative fugue, depersonalization disorder, and dissociative identity disorder (334)

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6
Q

What’s the difference between localized and selective amnesia?

A

In selective, you can remember some parts; in localized, a whole chunk of time is missing, usually the time just prior to a stressful event (334)

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7
Q

How common is dissociative amnesia?

A

Not very. Little is known. (335)

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8
Q

What’s the issue with the theory that hippocampus damage is what causes dissociative amnesia?

A

If it were actually that damaged, it would be impossible to retrieve the memories there at all (335)

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9
Q

What role may hormones play in dissociative amnesia?

A

Adrenal glands producing cortisol for fight-or-flight: excessive cortisol reduces hippocampus size,; but no defining evidence for this yet (336)

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10
Q

What does the dissociation/arousal disruption theory posit?

A

That traumatic experiences narrow the focus of attention and disorganize cognitive processes, so they can’t integrate normally to preserve/recall the memories (336)

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11
Q

What does neodissociation theory posit?

A

An “executive monitoring system” doesn’t have access to a certain system’s workings, because some systems can operate outside of the EMS’s scope: memory as an “independent cognitive system” causing an “amnestic barrier” to arise between memory and executive system: cut off, in a sense (336)

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12
Q

What characterizes dissociative fugue?

A

Sudden, unplanned travel and difficulty remembering the past (337)

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13
Q

What brain area is associated with dissociative fugue?

A

Frontal lobes, especially right hemisphere: role in retrieval of autobiographical memories (339)

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14
Q

What is the difference between depersonalization disorder and psychotic disorders?

A

DD patients still recognize reality and may not react emotionally to events (340)

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15
Q

What is a frequent comorbid diagnosis for depersonalization disorder, and why?

A

Anxiety disorder, because some of the symptoms are the same (numbness, detachedness, faintness) and because panic attacks often feature derealization or depersonalization (340)

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16
Q

What brain function abnormalities are associated with depersonalization disorders?

A

Temporal lobe: lower activity for visual/auditory recognition Parietal lobe: higher activation for orientation, representing the body (341)

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17
Q

How do depersonalization disorder patients handle emotional regulation?

A

Better than average, very well: perhaps why they don’t show emotional reactions to the strange situations they perceive (341)

18
Q

What is another name for dissociative identity disorder?

A

Multiple personality disorder: two or more alter selves (344)

19
Q

What is the neurological consensus on the idea that certain alters in DID have no memory of knowledge obtained by other alters?

A

Not really: brain may still show activation (345)

20
Q

What brain area plays a role in DID, and how?

A

Orbital frontal cortex (behind eyes), which regulates many cognitive/emotional processes: if early trauma prevents this area from maturing, then it can’t integrate representations of the self normally (346)

21
Q

What is the primary psychological factor associated with DID?

A

Hypnosis, which can even alter brain activity (347)

22
Q

What are the two dominant models for DID?

A

Posttraumatic and sociocognitive (the therapists accidentally encourages the formation of alters) (347)

23
Q

How does one treat dissociative disorder patients?

A

By reinterpreting symptoms to dissuade patients from avoiding certain situations, by teaching stress-coping skills, and by addressing alters/dissociated memories/identities if needed (349)

24
Q

What are somatoform disorders?

A

Characterized by complaints about physical well-being that can’t be entirely explained by a medical condition, substance use, or psychological disorder (353)

25
What are the two common features among somatoform disorders?
Bodily preoccupation and symptom amplification (353)
26
Which is the only somatoform disorder that does NOT include an actual somatic symptom?
Body dysmorphic disorder (353)
27
What similarity is there between dissociative disorders and somatoform disorders?
They both feature dissociation (353)
28
What are the four somatoform disorders?
Somatization disorder, conversion disorder, hypochondriasis, and body dysmorphic disorder
29
What physical conditions must be met to merit a SD diagnosis?
Four pain symptoms, two gastrointestinal symptoms, one sexual symptom, and one pseudoneurological symptom (354)
30
What role may genetics play in SD?
Not necessarily that SD can be inherited, but it does suggest a link between SD and hypochondriasis (357)
31
What social factors may contribute to SD?
Social learning from ill parents, operant condition through more attention when ill, etc, or social stress (358)
32
What is conversion disorder?
Involves sensory/motor symptoms that don't correspond to symptoms that arise from known medical conditions (360)
33
Give some examples of motor and sensory symptoms of conversion disorder
Motor: tremors/tics that worsen when attention is paid to them, pseudoparalysis; Sensory: loss of vision, deafness, lack of feeling, etc. ; Seizures (360)
34
How does "glove anesthesia" represent conversion disorder?
Patients report lack of feeling in the hand, but if it were a true neurologically-based numbness, the whole arm would be affected (361)
35
Why do some critics believe that somatoform symptoms should be classified as dissociative?
Because dissociation can affect memory, sense of self, AND integration of sensory/motor functioning (363)
36
How have researchers determined that conversion disorder isn't just malingering/faking of symptoms?
Brain scans: pretending to have muscle weakness appears differently than conversion patients (363)
37
What role might self-hypnosis play in conversion disorder?
Patient may unknowingly hypnotize self into believing symptoms: supported by neuroimaging (hypnotized paralysis activates similar brain areas to conversion disorder paralysis) and is consistent with the neodissociation theory (sensory system disconnected from executive monitoring system) (365)
38
To what category have some researchers suggested moving hypochondriasis and body dysmorphic disorders? Why?
To anxiety disorders: similar symptoms (366)
39
What neurotransmitter, when malfunctioning, probably contributes to hypochondriasis?
Serotonin (368)
40
What is one of the most successful treatments for somatoform disorders?
CBT (375)