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Anatomy > CNS 5 (Oct 8) > Flashcards

CNS 5 (Oct 8) Flashcards

1
Q

When you see someone crying, what response is activated?

A

-empathetic response is activated in the amygdala

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2
Q

What muscle opens the eyelid? What nerve is it innervated by?

A
  • levator palpebrae
  • CN III
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3
Q

What muscle is repsonsible for creating facial expressions by closing the eye?

A
  • orbicularis oculi
  • innervated by CN VII
  • blinking happens automatically through this sytem and voluntarily
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4
Q

What are saccadic eye movements?

A
  • necessary for fovea to be exposed to the entire scene
  • muscles need to be able to move around within the orbit to be able to scan
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5
Q

What purpose do the oblique muscles in the eye serve?

A

-correct movements of eye up and down (rectus muscles do not go straight back)

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6
Q

How is eye adducted and abducted?

A
  • contraction of lateral rectus (CN VI) and medial rectus (CNIII) muscles
  • abduct: lateral, adduct: medial
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7
Q

What two muscles work together to get movement of eye directly upwards?

A
  • superior rectus will pull eye up and outwards (laterally)
  • inferior oblique will pull eye up and inwards (medially)
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8
Q

What two muscles work together to get movement of eye downwards?

A
  • inferior rectus pulls eye outwards and downwards (CNIII)
  • superior oblique pulls eye downwards and inwards (CNIV)
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9
Q

What causes diplopia?

A
  • diplopia=double vision
  • overstretched extraocular muscles or probelm with innervation left and right eye can sometimes not work together properly (conjugate eye movements)
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10
Q

Where is the lesion?

A
  • eye is in abducted position indicating abducens nerve is working properly
  • CNIII is paralyzed so eye is pulled outwards
  • oculomotor palsy
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11
Q

Where is the lesion?

A
  • trochlear palsy
  • CNIV innervates the superior oblique muscle
  • patients with CNIV palsy complain of double vision especially when going downstairs, double vision looking downwards, and even though they have been corrected for optical error they still have difficulty reading
  • can test by tilting the head and see that one eye has abnormal movement
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12
Q

Where is the lesion?

A

-abducens palsy

  • CN VI
  • supplies lateral rectus muscle

-CNIII innervating medial rectus muscle is contracting and is unopposed and as a result the eye is pulled nasally

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13
Q

What is happening with patient A?

A
  • patient asked to follow the pencil
  • eye is in abducted position
  • lesion is in CNIII
  • abducens nerve is pulling the eye to the side and is also accompanied with a ptosis
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14
Q

What is happening with patient B?

A
  • ask patient to fixate on pencil
  • right eye is paralyzed
  • medial rectus muscle on the left eye is working properly but they can’t look to the side with the right eye
  • CNVI which is innervating the lateral rectus muscle
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15
Q

Which structure controls lateral gaze?

A
  • PPRF
  • paramedian pontine reticular formation
  • located in pons
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16
Q

Describe the process of lateral conjugate eye movements

A
  1. Frontal eye field will inform the PPRF that your eyes need to move to the side (ex: want to move eye to the left, info will go into left PPRF)
  2. This causes stimulation of abducens nucleus (L in this example)
  3. Left lateral muscle contracts (innervated by CNVI)
  4. At the same time, the PPRF sends message along the left medial longitudinal fasciculus
  5. This crosses over and innervates the CNIII on the right side (right oculomotor nucleus)
  6. Causes right medial rectus muscle to contract and move to the left as well
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17
Q

Describe the system to see things close up

A
  • convergence and accomodation system
    1. optic nerve sees object and projects to the superior colliculus
    2. Superior colliculus sends impulse into CNIII nucleus which causes both medial rectus muscles to contract and converges the eyes to focus on the object
    3. CNIII is closely associated with neurons that constrict the pupil and contraction of ciliary body muscle; parasympathetic innervation from the area around CNIII to the ciliary body and the pupil constricter
    4. When ciliary body is constricted, it allows lens to round up and magnify
    5. Pupillary constricter closes to increase the depth of field of the eye
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18
Q

How does ciliary body muscle contraction affect the lens?

A
  • normally lens is flat and stretched
  • ciliary muscle contracts and pulls everything to the front
  • smaller distance at front of the eye so as it’s pulled towards the front, the fibres that attach the lens to the sclera now become more relaxed
  • lens becomes relaxed and more round
  • goes up several diopters
19
Q

What makes the iris so opaque?

A
  • pigmented iris epithelium
  • very dense black pigment at back of eye
  • even in blue eyed person who has no pigment in the stroma of the eye, it is just as effective because everyone (except albino) has the pigment layer lining back of iris
20
Q

Describe pupillary sphincter and pupillary dilator muscles

A

sphincter: runs around the iris so when it is constricted the pupil gets smaller
dilator: runs along the length of the iris and it pulls the tip of the iris apart to widen the pupil

21
Q

Describe parasympathetic pupil control

A
  • preganglionic neuron starts in midbrain then follows along the CNIII until it reaches the ciliary ganglion
  • synapses at ganglion then postganglionic nerves causes pupillary constriction
22
Q

Describe sympathetic pupil control

A
  • begins in thorax
  • link between lateral horn of the spinal cord and the hypothalamus where sympathetic nerves are located
  • preganglionic nerves to the eye have to enter the sympathetic trunk
  • synapse in the ganglion at the top of the neck: superior cervical ganglion
  • postganglionic nerves get on carotid artery and follow it along the BVs that go into the eye and innervate pupillary dilator
23
Q

What is Horner’s syndrome?

A
  • sympathetic lesion
  • eye droop (ptosis)
  • eyelid has two muscles: skeletal muscle that voluntary opens the eye and smooth muscle that keeps the eye open in sympathetic response (drooping of eyelid when that muscle is lesioned)
  • miosis
  • anhydrosis
  • hyperemia (flushed skin)
24
Q

What is happening to patient A?

A
  • pupil is dilated
  • mydriasis
  • possibly lesion of CNIII parasympathetic fibres
  • also could be drug use, sex, stroke
25
Q

What is happening to patient B?

A
  • eye is drooping
  • miosis
  • lesion of sympathetic nervous system
  • opioid use
  • smooth muscle of eyelid is lesioned
  • eye is not in abducted position so CNIII is not involved
26
Q

What does tensor tympani muscle do?

A
  • dampens sounds when you are chewing
  • CNV
27
Q

What does stapedius muscle of the middle ear do?

A
  • innervated by CNVII
  • protects against loud sounds
28
Q

What does CNVIII do?

A

-special sensory nerve carrying hearing to the brain

29
Q

What provides sensation to the ear canal?

A
  • CNX
  • can feel fluttering in chest when you activate CNX with a q tip because of the relationship between the autonomic and sensory branches of the vagus nerve
30
Q

What nerve do vestibular and cochlear systems travel through to the brain?

A

-CNVIII vestibulo-cochlear nerve

31
Q
A
32
Q

What is the mastoid process?

A
  • series of channels connecting pharynx to the back of the ear
  • empty air space
  • bump of bone behind the ear; for the attachment of the sternocleidomastoid muscle
  • hollowed out with air cells to keep bone light
33
Q

What is external auditory canal made of?

A

-2/3 cartilage at the beginning then inner 1/3 of it is bone

34
Q

Infection in subcutaneous tissue in inner part of external auditory canal

A
  • thin skin is firmly attached to periosteum of the bone
  • hardly any connective tissue here
  • no place to relieve pressure created by the infection so these are extremely painful
35
Q

Describe layers of the tympanic membrane

A

-covered with skin on the outside, connective tissue in the middle, and mucosa on inside

36
Q

What can we see when the tympanic membrane is removed?

A
  • ossicles of the middle ear
  • small bones hanging from roof of middle ear
37
Q

What happens when tympanic membrane vibrates?

A
  • bones transduce the force from the tympanic membrane to the inner ear
  • malleus, incus, stapes
  • bones can attach to muscles
  • muscles can modulate/dampen the sound
38
Q

Where does tensor tympani muscle attach?

A
  • at malleus/tympanic membrane
  • innervated by CNV which innervates muscles of mastication
  • this muscle also contracts when you are chewing which puts tension on the tympanic membrane and malleus dampening the sounds
39
Q

Where does the stapedius attach? What happens with prolonged exposure to loud sounds?

A
  • attaches to the stapes
  • when it contracts, it does so in response to very loud sounds
  • CNVII causes it to contract and prevents stapes from doing damage to the inner ear by putting too much pressure on the oval window
  • skeletal muscles fatigue so when you are exposed to prolonged loud sounds the stapedius muscle fatigues and weakens so the loud sounds can damage to receptors in inner ear
40
Q

What is the auditory tube normally doing?

A
  • normally stays shut by contraction from pharyngeal muscles
  • comes from pharynx
  • swallow hard/chewing temporally opens end of auditory tube and allows for air pressure to be equilibrated
41
Q

How do mastoid air cells equilibrate pressure?

A
  • channels from the mastoid leading to the middle ear
  • they will be equilibrated at the same time as the middle ear when the auditory tube opens
42
Q

How does a middle ear infection occur?

A
  • happen especially in children who have short pharyngeal tube
  • when you sneeze, you are also swalling at the same time; blow bacteria and viruses into middle ear
  • auditory tube allows for tubes to track up the channel and get into middle ear
  • can cause infection of middle ear through infection of the mucous membrane
43
Q

What are complications of a middle ear infection?

A
  1. Rupture of the tympanic membrane
  2. Drainage of the fluid into the external ear
    - when you have a cold, the mucosal membrane of the pharyngeal tube can get blocked which causes pressure to build up in middle ear and the only way that can be relieved is by rupturing the tympanic membrane (it is connective tissue so can repair)
    - repeated rupture of the membrane can be a problem because you get scarring of tympanic membrane
    - roof of middle ear is thin so infection of mucosa in this area can erode bone away which can be associated with a problem in the meninges
    - opening in middle ear to mastoid air cells which can lead to infection in mastoid air cells; repeated infection of these may require installation of tubes to continuously drain the infection

Anatomy (27 decks)

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