ECG Flashcards

(52 cards)

1
Q

Shockable rhythms

A

Pulseless VT
Pulseless VF

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2
Q

Non shockable rhythms

A

Pulseless asystolie
Pulseless electrical activity

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3
Q

Management of sinus tachycardia if no pulse

A

ACLS

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4
Q

Management Sinus tachycardia if pulse

A

ABC
Monitoring
Correct electrolytes
If still not stable ->. Synchronized DC shock
If stable -> vagal maneuvers or give adenosine

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5
Q
A

Sinus bradycardia

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6
Q

Causes of sinus bradycardia

A

Normal in athletic patient
MI
Hypothyroidism
Hypothermia
Hyperkalemia
Sick sinus syndrome
Drugs (BBlocker, digoxin, anticholinesterase erase, halothane, sux)

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7
Q
A

Sinus bradycardia

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8
Q

causes of sinus bradycardiardia

A

oNormal athletes
Drugs - bblocker, digoxin
MI
sick sinus syndorme
Raised ICP
Hypovolemiathyroisidm y

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9
Q

Management of sinus bradycardia

A

Correct underlying cause
No correction if person healthy and HR>40bpm
Incremental dose of atropine up to 20mcg

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10
Q
A
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11
Q

Causes of sinus tachycardia

A

Inadequate depth of anaesthesia
Pain
Surgical stimulation
Fever sepsis
Anemia
Shock
Thyrotoxicosis
Drugs - atropine ktamine

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12
Q
A

Atrial fibrillation

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13
Q

ECG features of atrial fibrillation

A

Irregularly irregular rate - variable ventricular rate
No P waves
QRS <120ms

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14
Q

AF management

A

Determine if presence of shock, syncope, MI , ,heart failure

If stable - correct underlying cause , rate control (BBlocker esmolol 0.5mg/kg IV bolus over 1min or amiodaroneif hfref) or rhythm control (DC cardio version or amiodarone 300mg IV over 1h and 900mg over 23h) management possible

If unstable - urgent synchronized DC cardio version 120-150j and increase
Anticoagulants

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15
Q
A
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16
Q
A

Atrial flutter

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17
Q

Atrial flutter features on ecg

A

Narrow complex tachycardia
Loss of isooelectric baseline
saw tooth pattern
Inverted flutter in leadsII, III, avF

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18
Q

Management atrial flutter

A

Hemodynamic unstable -> synchronized DCCV (GAor sedation)

Stable -> vagal manœuver , carotid sinus massage, vasalva,, adenosine 6mgIV followed by 0.9% nacl

Rate and rythm control - bblockk or amiodarone

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19
Q

Ventricular ectopics features on ecg

A

Broad QRS complex (more than 0.12s or 3 boxes)
Premature
Discord a tST and T wave changes

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20
Q

Management ventricular ectopics

A

Correct reversible causes
If sinus rythm slow less than 50bpm -> increase IV atropine or glycopyrronium

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21
Q
A

Ventricular tachycardia

22
Q

VT features on ECG

A

Regular broad complex tachycardia
Uniform QRS

23
Q

Triggers of VT

A

MI
Hypoxia
Hypotension
Fluid overload
Electrolyte imbalance
Adrenaline and catecholamines
Drug which prolong QT interval

24
Q

Management of VT

A

Pulseless VT -> CPR and ACLS
VT with pulse -> if adverse features do synchronized DCCV , if no adverse features amiodarone 300mg IV over 20-1h then 900 mg over 23h

25
Torsades de pointes
26
Management torsades de pointes
If compromise -> Immediate defibrillation even if pulse If stable -> give Mg2+2g IV 10-20mins, infusion at 1-4g/h Monitoring plasma levels
27
Ventricular fibrillation
28
Management ventricular fibrillation
Immediate DCCV
29
MI
30
Features MI
St elevation St depression T wave inversion New onset bbb Old MIwith pathological Q waves
31
Management MI
ICU MONA- O2, morphine IV, aspirin PO, SL glyceryl trinitrine Definitive management Treat Heart failure Investigations - 12 lead ECG, cardiac enzymes, echo
32
Do you need to investigate or treat a first-degree heart block?
No
33
Per management of a patient with a third of second-degree heart block
ABC If hypertension isoprenaline Transcutaneous pacing
34
Indication for preoperative pacing in heart block
Symptomatic first-degree heart block Second-degree or thirddegree heart block Sinus node, disease, symptomatic
35
First degree heart block
36
Features first degree heart block
Increased PR interval more than 200ms
37
Causes of first degreee heart block
Increased vagal tone Athletic training Inferior MI Mitral valve surgery Myocarditis Electrolyte disturbances Dugs like bb, ccb, digoxin, amiodarone r
38
Morbitz type i
39
ECG features mobitz type 1
Progressive prolongation of PR interval
40
ECG features mobbitz type 2
Intermittent non conducting p waves with no progressive prolongation of pr interval
41
Mobitz type 2
42
Causes of mobitz type 2
Anterior mi Idiopathic fibrosis Cardiac surgery’Inflammatory conditions Autoimmune conditions Infiltrative myocardial dx Hyperkalemia Drugs bb , ccb, digoxin, amiodarone
43
Hypokalemiaalemia
44
Causes of complete heart block
Inferior MI Drugs Idiopathic
45
Complete heart block
46
Hypokalemia
47
Feature hypokalemia
T wave inversion St depression Prominent U wave
48
Management of hypokalemia
Admit Monitoring with ECG Central line -> KCL Urethral catheter to monitor urine output
49
Hyperkalemia features on ecg
Tall T waves P wave flattening PR prolongation Wide QRS
50
Hyperkalemia
51
52
Hyperkalemia management
if K+ more than 6.5mmol/LL or ECG changes ABC Monitoring with ECG IV access - insulin 10U+ 50ml 50%dextrose over 30mins to 1h Calcium gluconate 5-10mls of 10% or calcium chloride 3-5mls of 10% If severe consider dialysis B2 agonists