EEE/WEE/VEE Exam 2 Flashcards

1
Q

How many types of equine encephalitis virus are there?

A

3 (EEE, WEE, and VEE)

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2
Q

How is EE transmitted?

A

between birds or rodents and mosquitoes

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3
Q

When does the clinical disease occur during the year?

A

during mosquito season: late summer and early fall in temperate zones
-year-round in tropical zones

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4
Q

What forms cannot be passed from horse to horse?

A

EEE and WEE

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5
Q

Where is EEE more prevalent?

A

southeastern states (north FL and Carolinas)

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6
Q

Can humans get EEE?

A

yes (not from horses or other humans though)

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7
Q

What is the mortality rate of EEE in humans?

A

~10% every year

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8
Q

What is the mortality rate of EEE in horses?

A

75-90% every year

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9
Q

Why is EEE not transmitted to others once a horse contracts it?

A

they are affected by the disease but don’t produce high enough viremia to serve as a reservoir (considered dead end hosts)

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10
Q

Can WEE affect people?

A

yes

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11
Q

When was WEE the most deadly?

A

in the 30’s for both people and horses

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12
Q

Is WEE common today?

A

no, cases in horses are sporadic

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13
Q

What is the mortality rate of WEE in horses?

A

<50%

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14
Q

What is one of the most important human and vet pathogens in the Western Hemisphere?

A

VEE

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15
Q

What area was severely affected by VEE (humans and horses) in the 60’s?

A

Colombia

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16
Q

What is the mortality rate of VEE in horses?

A

90%

17
Q

Where are there multiple outbreaks (most common) of VEE?

A

in Central and South America

18
Q

Does the United States often suffer from VEE?

A

no

19
Q

Who is the reservoir of VEE?

A

horses themselves spread the disease

20
Q

Mortality rate of VEE in humans?

A

1%

21
Q

What other animals also develop VEE?

A

-rabbits
-dogs
-small ruminants

22
Q

Has the human disease ever been demonstrated in the absence of the equine disease?

A

NEVER

23
Q

Why are vaccinations not recommended in the US for VEE?

A

because if the US brings it and gets the vaccination, the antibodies are now mixed up with the virus (cannot appropriately diagnose afterwards)

24
Q

Which forms are more neuroinvasive?

A

EEE and VEE

25
Q

Clinical signs

A

-obtunded mentation
-ataxia
-paralysis
-ultimately stupor
-irregular gait
-grinding of teeth
-incoordination
-circling
-head pressing
-hyperexcitability
-profound depression (sleeping sickness)

26
Q

Clinical signs are _______ in nature

A

progressive (mortality EEE and VEE > WEE)

27
Q

How is it diagnosed?

A

definitive diagnosis ante-mortem may not be possible, serology may be indicative

28
Q

What is possible to detect in horses that survive and are diagnosed?

A

rising antibody titers

29
Q

How can it be treated?

A

-support
-corticosteroids
-tranquilizers
-diuretics

30
Q

Prevention methods

A

-vaccination
-mosquito control
-aerial spraying of insecticide
-treating bodies of water
-spray horses with permethrin-based products