Epi Mix AG 6401-6600 Flashcards

1
Q

The incubation time of rabies is generally 2-8 weeks; however, exceptions can occur

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2
Q

Rabies virus can only be transmitted with bite

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3
Q

The host range of rabies is very narrow, mainly dogs and foxes are susceptible

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4
Q

Dogs have to be observed for 14 days if they have bitten humans

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5
Q

Herbivorous animals bitten by foxes can be emergency slaughtered

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6
Q

Rabies virus can be detected with immunofluorescence test

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7
Q

Rabies virus appears in the saliva 2-3 days after the onset of the clinical signs

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8
Q

Lethality of rabies in humans is high

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9
Q

The resistance of rabies virus is low; it cannot survive in the environment for a long time

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10
Q

Rabies can be diagnosed by detection of antibodies in paired sera

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11
Q

Antibodies against rabies detected with ELISA confirm the diagnosis of rabies

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12
Q

Rabies virus is spreading alone the nerves in the host

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13
Q

Urban rabies has been eradicated from the Earth

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14
Q

Urban rabies is maintained by dogs and cats

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15
Q

Rabies occurs only in tropical countrie

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16
Q

Herbivores animals are generally dead end hosts of rabies

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17
Q

Rabies virus replicates in the lymphocytes

A

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18
Q

Postmortem examination of rabid animals is forbidden

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19
Q

Haemorrhages of the serous membranes are typical postmortem lesions of rabies

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20
Q

Rabies can be diagnosed only by using PCR

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21
Q

Post exposition vaccine against rabies can be given to ruminants bitten by foxes

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22
Q

Rabies virus does not penetrate the blood vessels

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23
Q

Rabies: one of the important symptoms is the change of behaviour

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24
Q

Rabies: one important symptom is paralysis

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25
Q

In rabies we find purulent encephalitis in negri-bodies

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26
Q

With immunofluorescence test we can diagnose rabies

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27
Q

We can diagnose rabies by antibody detection test

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28
Q

When an animal which is infected with rabies virus attacks another dog, it should be vaccinated immediately

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29
Q

Rabies is a uniform virus

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30
Q

Rabies is a resistant virus

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31
Q

Warm blooded animals can be infected with rabies

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32
Q

Rabies can be transmitted only by saliva

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33
Q

Fox rabies: we can use live vaccines

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34
Q

Since 2002 rabies is eradicated from Hungary

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35
Q

Humans can be infected by Rabies bite

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36
Q

Humans can be infected by Rabies during organ transplantst

A

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37
Q

Rabies virus is spreading in the infected animals peri-neural

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38
Q

Rabies virus is shed in the saliva, before appearance of the clinical signs

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39
Q

The incubation of rabies is generally less than one week

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40
Q

The rabies virus is a uniform virus without serotypes, genotypes, subtypes etc

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41
Q

High amount of rabies virus is shed in the saliva

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42
Q

The causative agent of rabies is a uniform virus, without different types or groups

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43
Q

The resistance of the rabies virus is good, it retains infectivity for several months in the environment

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44
Q

All warm-blooded animals are susceptible to rabies virus

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45
Q

Rabies virus is shed in high titre in the saliva

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46
Q

Paralysis is a clinical sign of rabies

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47
Q

Foxes are vaccinated with attenuated bait vaccine

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48
Q

Cattle bitten by rabid animals may be vaccinated post exposition or slaughtered

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49
Q

Humans can be infected with rabies, only by being bitten by rabid animals

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50
Q

Rabies virus is spreading along the nerves to the central nervous system

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51
Q

Rabies virus is shed in the saliva only after the appearance of the clinical signs

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52
Q

Focal necrosis in the liver is a typical lesion of rabies

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53
Q

Immunofluorescence test is widely used diagnostic method in the case of rabies

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54
Q

Virus neutralization test is used to the laboratory diagnosis of rabies cases

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55
Q

Dogs have to be vaccinated for the prevention of rabies at the age of 3 months for the first time

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56
Q

Increased salivation is a clinical sign of rabies

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57
Q

Always the furious form of rabies can be seen in dogs

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58
Q

Colic can be a sign of rabies in horses

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59
Q

Changed behaviour is a clinical sign of rabies of wild living animals

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60
Q

Only the classical rabies virus can cause clinical signs, the other genotypes not

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61
Q

The sylvatic form of rabies is maintained by different wild living animals

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62
Q

Rabies virus causes viraemia soon after infection

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63
Q

Saliva can contain rabies virus, before the appearance of clinical signs

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64
Q

Humans are not susceptible to European bat lyssaviruses

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65
Q

There is no haematogenic spreading of the rabies virus

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66
Q

Increased sexual activity is seen in rabid cattle

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67
Q

Dogs have to be vaccinated against rabies in the first week of life

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68
Q

Cattle bitten by a rabid animal can be vaccinated after exposition

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69
Q

The rabid bats fly during the day

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70
Q

Immunofluorescence (IF) is reliable in the diagnosis of rabies

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71
Q

Only the classical rabies virus is present in Europe

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72
Q

Urban form of rabies is maintained by the fox in Europe

A

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73
Q

Rabies virus is highly resistant

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74
Q

Rabies virus cannot be cultured

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75
Q

Presence of antibodies to rabies virus confirms the diagnosis of rabies

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76
Q

Detection of Negri bodies is more sensitive than immunofluorescence test, in the case of rabies

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77
Q

Virus isolation is the most widely used way of diagnosis of rabies

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78
Q

Inhalation of the virus is the main way of infection with rabies virus

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79
Q

Rabies virus is replicating in the lymphoid cells and causes viraemia before the appearance of the clinical signs

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80
Q

The incubation time of rabies is variable, generally between 2 and 8 weeks

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81
Q

Immunofluorescence test can be used to the detection of rabies virus in the brain

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82
Q

Rabid animals have to be vaccinated immediately

A

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83
Q

In cats furious form of rabies is typical

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84
Q

In dogs both furious and silent form of rabies can occur

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85
Q

Rabies has been eradicated in Europe

A

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86
Q

Rabies spread through venereal

A

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87
Q

The high-titre of virus neutralizing antibodies confirms the diagnosis of rabies

A

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88
Q

Live vaccines are used for the vaccination of foxes against rabies

A

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89
Q

Rabies symptoms appear only after CNS signs

A

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90
Q

Rabies virus can only be found in the nervous system

A

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91
Q

Borna disease occurs in Africa, Asia and South America, but Europe is free

A

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92
Q

Dyspnoea is the main clinical sign of borna disease

A

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93
Q

Only horses are susceptible to Borna disease virus

A

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94
Q

Borna disease virus is spreading from cell to cell

A

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95
Q

Mainly central nervous clinical signs can be seen in the case of Borna disease

A

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96
Q

The lethality of Borna disease is low

A

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97
Q

Borna disease is widespread all over the world

A

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98
Q

Pneumonia is the main clinical sign of Borna disease

A

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99
Q

Most animals showing clinical signs of Borna disease die due to it

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100
Q

Horse is the reservoir species of Borna disease virus

A

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101
Q

Clinical signs of Borna disease appear very slowly

A

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102
Q

Dyspnoea, nasal discharge and cough are the main signs of Borna disease

A

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103
Q

Borna disease causes dementia/CNS signs

A

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104
Q

Borna disease occurs mainly in the Far East

A

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105
Q

Borna disease replicate in the nucleus of cells and is called Joest Degen bodies

A

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106
Q

Borna disease can be seen all over the world.

A

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107
Q

Respiratory signs are the most typical ones in the case of Borna disease

A

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108
Q

Only ruminants are susceptible to Borna disease virus

A

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109
Q

The agent of proventricular dilation disease causes inflammation of the peripheral nerves

A

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110
Q

Proventricular disease is a zoonosis

A

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111
Q

Proventricular disease can occur in parrots

A

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112
Q

Retroviruses are frequently carried lifelong

A

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113
Q

Retroviruses carry an integrase enzyme

A

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114
Q

Malignant transformation of host cells is a typical effect of several retroviruses

A

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115
Q

Retroviruses are enveloped, their resistance is low

A

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116
Q

The reverse transcriptase transforms DNA of the retroviruses to mRNA

A

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117
Q

Retroviruses are stable viruses; genetic changes are rare

A

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118
Q

Retroviruses are euryxemic agents

A

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119
Q

Retroviruses are generally host specific viruses

A

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120
Q

Mutation of retroviruses is very rare

A

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121
Q

Immunosuppression is a typical effect of several retroviruses

A

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122
Q

Retroviruses can integrate into the genome of host cells

A

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123
Q

Reverse transcriptase is an important enzyme of retroviruses

A

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124
Q

Retroviruses results in lifelong infection

A

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125
Q

Retroviruses replicate mainly in the endothelial cells

A

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126
Q

Several retroviruses can cause malignant transformation in the hosts

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127
Q

Retroviruses are generally species specific

A

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128
Q

Retroviruses are generally resistant, they can survive in the environment for several weeks

A

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129
Q

Retroviruses frequently cause permanent infection

A

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130
Q

Retrovirus has weak resistance

A

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131
Q

Retrovirus has a wide host spectrum

A

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132
Q

Retrovirus has a good immunogenicity

A

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133
Q

Retrovirus infection is long-lasting

A

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134
Q

Retroviruses show high host specificity

A

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135
Q

Retroviruses are generally not carried for more than a month

A

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136
Q

Retroviruses generally cannot survive in the environment for a long time

A

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137
Q

Retroviruses are enveloped viruses

A

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138
Q

Retroviruses transcribe their nucleic acid to DNA

A

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139
Q

Frequent genetic changes of retroviruses are common

A

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140
Q

Retroviruses carry reverse transcriptase enzyme

A

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141
Q

Retroviruses generally cause long, frequently life-long infection

A

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142
Q

Retroviruses are generally genetically very stable

A

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143
Q

The resistance of retroviruses is generally good, they survive in the environment well

A

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144
Q

Reverse transcriptase is produced by retroviruses

A

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145
Q

The nucleic acid of retroviruses can be integrated into the genome of the host cell

A

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146
Q

Retroviruses frequently cause immune suppression

A

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147
Q

The host range of retroviruses is generally narrow

A

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148
Q

Reverse transcriptase converts RNA of retroviruses into DNA

A

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149
Q

Retroviruses are generally shed in infected lymphoid cells

A

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150
Q

Certain retroviruses can cause proliferation of the lymphoid cells

A

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151
Q

Retroviruses spread with infected lymphocytes

A

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152
Q

Retroviruses have a tegument or rind

A

f

153
Q

You cannot multiply retrovirus artificially

A

f

154
Q

Retroviruses cannot spread from animal to animal

A

f

155
Q

Retroviruses are widely distributed in Hungary

A

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156
Q

Retroviruses replicate mainly in endothelium cell

A

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157
Q

Retrovirus can replicate without helper retroviruses

A

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158
Q

Retroviruses have own metabolic enzymes

A

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159
Q

Antibodies against enzootic bovine leukosis virus can be detected 1-4 months after infection

A

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160
Q

Antibodies against enzootic bovine leukosis virus can be detected only for 1-2 months after infection

A

f

161
Q

Maternal Antibodies against enzootic bovine leukosis virus can be detected only for 1-2 months

A

f

162
Q

Lymphosarcoma can be seen postmortem in the case of enzootic bovine leukosis

A

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163
Q

Generation shift is the only way of eradication of enzootic bovine leukosis

A

f

164
Q

Enzootic bovine leukosis virus does not spread from animal to animal

A

f

165
Q

Mild clinical signs can be seen in the incubation phase of enzootic bovine leukosis

A

f

166
Q

Enzootic bovine leukosis virus is not shed in the colostrum

A

f

167
Q

Enzootic bovine leukosis virus can be transmitted with blood

A

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168
Q

Enzootic bovine leukosis virus can spread from cattle to sheep, goats , and other ruminants

A

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169
Q

Enzootic bovine leukosis virus has uniform antigenic structure

A

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170
Q

In the case of Enzootic bovine leukosis the clinical signs appear at the age of 6-8 months

A

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171
Q

Enzootic bovine leukosis is carried lifelong

A

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172
Q

Enzootic bovine leukosis virus can be transmitted in tracheal discharge

A

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173
Q

Enzootic bovine leukosis occurs only in Holstein Friesian cattles

A

f

174
Q

Enzootic bovine leukosis virus can infect foetuses of pregnant animals

A

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175
Q

Enzootic bovine leukosis virus has several serotypes and subtypes

A

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176
Q

Enzootic bovine leukosis can spread by air within the herd

A

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177
Q

Enzootic bovine leukosis can spread by the veterinarian

A

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178
Q

Enzootic bovine leukosis virus cannot result tumour formation

A

f

179
Q

Serological examinations cannot be used to the diagnosis of enzootic bovine leukosis

A

f

180
Q

Immune tolerance can happen in the case of enzootic bovine leukosis

A

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181
Q

Selection cannot be used for eradication of enzootic bovine

A

f

182
Q

Bovine enzootic leukosis infect only bovine

A

f

183
Q

Bovine enzootic leukosis does not spread with excretion

A

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184
Q

Bovine enzootic leukosis spreads slow in the herd

A

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185
Q

Bovine leukosis virus causes seropositivity in latency period

A

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186
Q

Enzootic bovine leukosis the pre-tumour phase usually in 6-10 months old animals

A

f

187
Q

Enzootic bovine leukosis during pre-tumour phase causes lymphocytosis

A

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188
Q

Bovine enzootic leukosis virus can be transmitted with lymphoid cells

A

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189
Q

Iatrogenic infection is frequent in the epidemiology of bovine enzootic leukosis

A

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190
Q

The target cells of the bovine enzootic leukosis virus are the T-lymphocytes

A

f

191
Q

The typical signs of bovine enzootic leukosis can be seen in cattle under 1 year of age

A

f

192
Q

Antibodies against enzootic bovine leukosis virus can be detected in the ELISA test

A

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193
Q

Antibodies against enzootic bovine leukosis virus can be detected in the milk

A

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194
Q

Selection (test and slaughter) method cannot be used to eradicate enzootic bovine leukosis virus

A

f

195
Q

Generation shift method cannot be used to eradicate enzootic bovine leukosis virus

A

f

196
Q

Enzootic bovine leukosis virus is spreading horizontally in a cattle herd

A

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197
Q

Enzootic bovine leukosis virus cannot infect foetuses

A

f

198
Q

Enzootic bovine leukosis virus is passed to newborn calves mainly with colostrum in endemically infected herds

A

f

199
Q

By the end of the incubation phase the animals become seropositive leukosis virus

A

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200
Q

Tumours can be seen in about 90% of the animals infected with enzootic bovine leukosis virus.

A

f