Exam 2 Flashcards

(51 cards)

1
Q

Pressure equation

A

Pressure = flow X resistance

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2
Q

quantity of delta P

A

size of the pressure gradient

represent the driving force that pushes the liquid through the vessel

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3
Q

quantity R (resistance)

A

a measure of the various factors that hinder the flow of liquid through a vessel

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4
Q

what factors change resistance

A

vessel radius
vessel length
blood viscosity

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5
Q

vessel radius

A

As radius decreases, resistance increases

vasoconstriction or vasodilation

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6
Q

vasoconstriction

A

a decrease in blood vessel radius

-increases network resistance

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7
Q

vasodilation

A

An increase in vessel radius

-decreases network resistance

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8
Q

Vessel length

A

longer vessels have greater resistance than shorter ones

Vessels do not change length except as a person grows

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9
Q

Blood viscosity

A

Vascular resistance increases as viscosity increases

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10
Q

what determines blood viscosity

A

concentration of cells and proteins in the blood

-As either concentration increases, blood viscosity increases.

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11
Q

total peripheral resistance

TPR

A

In the systemic circuit, the combined resistances of all the blood vessels within the circuit
-resistance in the systemic circuit

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12
Q

TPR and pressure gradient relationship

A

The total flow increases in proportion to the pressure gradient along the network
-as total flow decreases, the resistance of the network increases

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13
Q

what drives the flow of blood

A

the pressure gradient represented by the difference between MAP and CVP
-MAP in the systemic circuit

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14
Q

capillaries

A
  • smallest of all blood vessels

- consists of a layer of endothelial cells

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15
Q

P wave

A

Atrial depolarization

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16
Q

QRS complex

A

Ventricular depolarization & atrial depolarization

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17
Q

T wave

A

Ventricular repolarization

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18
Q

P-Q or P-R interval

A

AV nodal delay

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19
Q

Q-T interval

A

Ventricular systole

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20
Q

T-Q segment

A

Ventricular diastole

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21
Q

R-R interval

A

Time between heart beats

22
Q

First degree heartblock

A

Slowed/diminished conduction through the AV NODE occurs in varying degrees

  • increases duration PR or PQ segment
  • increases delay between atrial and ventricular contractions
23
Q

Second degree heart block

A

Lose 1 to 1 relationship between p wave and QRS complex

-and also atrial and ventricular contraction

24
Q

Third degree heartblock

A

Loss of conduction through AV node

  • P wave becomes independent of QRS
  • atrial and ventricular contractions are independent
25
Extra systole
An abnormal depolarization spread throughout the heart, causing extra contraction
26
Fibrillation
Occur when the heart muscle no longer demonstrates synchronized depolarization
27
Cardiac output
Rate at which ventricles pumps blood - determine by hr and sv - CO=HR*SV
28
Factors affecting CO | Changes hr
Age Health Level of activity Emotional state
29
Hormonal control heart rate (extrinsic)
Epinephrine in response to increased sympathetic activity | -increases action potential at SA node and increases heart rate
30
Ventricular contractility
A measure of ventricles capacity for generating force
31
After load
The pressure that the ventricles have to work against as they pump blood out of the heart
32
What changes stroke volume
Ventricular contractility End-diastolic volume Afterload
33
I band
Only actin
34
H band
Only myosin
35
A band
Actin and myosin
36
M line
Center of filament
37
Thin filaments
Contain actin
38
Thick filaments
Contain myosin
39
significance of Starlings Law
By adjusting stroke volume so that cardiac output matches venous return, the heart regulates its size. If the ventricles get too big, they become unable to generate enough pressure to maintain adequate cardiac output; such an inability is called heart failure
40
Factors affecting end diastolic volume
Pre load | central venous pressure (the pressure of blood contained in the large veins that lead into the heart)
41
What is preload
(1) filling time, which depends on heart rate (2) atrial pressure, which is determined by venous return and the force of atrial contraction. As heart rate decreases, filling time increases because diastole increases in duration
42
where does the heart get its nourishment
coronary arteries
43
three heart layers
1) epicardium: CT 2) myocardium : cardiac 3) endocardium: epithelium
44
which ventricle is the thickest
the left, because it pumps blood to the whole body
45
mitral valve/bicuspid
on the left consisting of two flaps or cusps of connective tissue
46
SA node
located in the wall of the upper right atrium near where it joins with the superior vena cava -Faster inherent rate of spontaneous depolarization
47
AV node
located near the tricuspid valve in the interatrial septum | Depolarization driven by the SA node
48
pace maker cell
initiate action potentials and establish the heart rhythm | Spontaneously generate action potentials
49
Intercalated disks
gap junctions which permit electrical current to pass in the form of ions from one cell to another help to rapidly transmit action potentials from pacemakers to conduction fibers
50
Desmosomes
form a physical bond between the disks that resist mechanical stress, enabling the myocardium to withstand the tension that is generated every time the muscle cells contract
51
MAP equation
MAP = HR x SV x TPR