Lecture 1 Flashcards
(35 cards)
1 Gram of CHO/Fat/Protein/ Ethanol is equal to how much Kj/g & kcal/g?
CHO: 16.7 kj/g (4 kcal/g)
Protein: 16.7 kj/g (4kcal/g)
Fat: 38.1 kj/g (9 kcal/g)
Ethanol: 29.3 kj/g (7 kcal/g)
How might energy intake be measured?
Direct Calorimetry - Bomb Calorimeter
1 kcal = ?? Joules
1 kcal = 4.18 Joules
How much of protein is nitrogen and what happens to it?
15-19%, which is excreted and does not contribute to EI
What is the difference in soluble and insoluble fiber when ingest in terms of energy intake? How does this effect food labels?
Soluble can be partially fermented/broken down while insoluble cannot be broken down which means it does NOT contribute to EI while soluble fiber may contribute a minor amount. Each country interprets the contribution of soluble fiber differently so depending on the country your food label will read differently for energy contribution of soluble fiber.
What limitations are associated with Energy Intake (EI) estimates (3)?
- Energy availability (EA) may change depending on specific food compared to “Antwater” factors
- Mixed/cooked meals does not equal the sum of its individual parts
- EA may change depending on other factors such as Obesity, Gut Microbiota, disease etc
From an EI perspective does it matter if Cal are absorbed from Fat or CHO?
Long term may affect RQ but short term “calories are calories”
Will you ever deplete the body of CHO and glucose?
Likely not as CHO is the main fuel source for the brain and body. Regulation of CHO and glucose is primary concern.
What happens to insulin once food (CHO) is ingested, what happens to oxidation of Fat and CHO?
Once CHO is ingested, Insulin is secreted.
Body will begin digesting and utilizing the CHO.
CHO oxidation (RER 1.0) increases while Fat oxidation (AKA lipolysis) (0.7) decreases
Does increasing fat consumption increase fat oxidation?
No it does not.
What happens to excess CHO that does not get oxidized?
Stored as fat
Describe the Oxidative Hierarchy
This is the order of utilization/oxidization of the macronutrients +alcohol. Alcohol, CHO, Protein, Fat which means that Alcohol is oxidized before all others, CHO before protein/fat etc.
The one catch is that if the body is in need of CHO it will preserve the CHO it has and begin utilizing Protein/Fat first. Regulation of Macronutrients supersedes the hierarchy of oxidation.
How do we measure TDEE?
Double Labeled Water or estimate RMR + TEF + EEA
What is RMR and how do we measure it?
Resting Metabolic Rate - The basic energy required to survive
Can be measured directly (bomb) or indirectly by measuring inspired and expired gas (VCO2/VO2)
What is the RER of Lipid oxidation?
0.7
What is the RER of CHO oxidation?
1.0
What can influence RMR?
Body temp, Posture, Food consumed, level of alertness, time of day
What should be the test conditions for the subject when measuring RMR?
Rest them supine Room should be temperature neutral Fasted over night Be awake during test No exercise w/in 12 hours Female: Controlled phase of menstrual cycle
Who will have higher metabolic rate?
Male/Female
Young/Old
Fat/Normal
Male
Young
Fat
Explain the relationship between RMR and Body weight and how does this apply to the 3500 kcal/ 1 pound of fat rule in the Hall et al. reading?
RMR is the minimum amount of energy needed to survive, a larger body will require more fuel to perform activities (compared to a smaller person) because it takes more to move a larger mass. Inversely it will cost a smaller person less energy to do the same thing as a larger person. As a body loses weight, the amount of energy to perform certain activities will also drop. This means that a person may have to perform more activity or take in less energy to lose weight at the same rate throughout an intervention. This means the 3500kcal rule should not be applied throughout the WHOLE intervention and may only be useful at one phase.
What could influence the accuracy of RMR?
- Last bout of exercise (EPOC)
- Last meal (Diet Induced Thermogenesis)
- Rested state
- Low grade infections
- Tester accuracy
- Equipment accuracy
- Other factors (stress, sleep, etc)
What are the effects of ACUTE or CHRONIC physical activity on RMR?
Chronic: (Similar to the 3500kcal theory) Chronic PA changes FM and FFM which influences RMR. This means RMR is variable.
Acute: Single doses of exercise can raise EE due to variability in exercise (FITT) and sustained after effects due to (EPOC)
RMR is effected more by Skeletal muscle or visceral organs?
Visceral organs, which actually account for 60% of RMR
What can increase EPOC and what allows for more energy expenditure, the actual exercise or EPOC?
EPOC can be varied by time and intensity of exercise, but in the end the actual exercise itself is responsible for most of the EE.