neuro 4b Flashcards
(31 cards)
where do voluntary saccades originate
frontal eye field and superior colliculus contralateral to the direction fo gaze
other areas that contribute to saccadic control
PPRF, dorsolateral prefrontal cortex, supplementary eye field, and parietal lobe
vertical saccades
may also originate in frontal eye fields or superior colliculi and connect to the contralat riMLF
inability to produce saccades is called
oculomotor apraxia
hypermetric saccades
overshoot
hypometric saccades
undershoot
VOR (vestibulo-ochlear reflex)
coordinates eye movements with head movement; elicited by slow passive head movements
pathway for VOR
semicircular canals (rotation) and otoliths (linear acceleration) and travels to the vestibular nuc and from there to the abducens nuc and then to CNs 3 and 4 through the MLF
abnormalities of the VOR (vestibulo-ochlear reflex)
nystagmus
nystagmus
rhythmic to and fro movement of the eyes
jerk nystagmus
eye drifts away from fixation in a pursuit-like movmenet and returns with a fast saccadic movement
direction of nystagmus named how
by the direction of the fast component
comitant strabismus
misalignment is constant in all directions of gaze and each eye has full range of movement (usually an eye problem)
incomitant strabismus
the degree of misalignment varies with the direction of gaze (usually a neuro problem)
phoria (eso or exo)
misalignment of the eyes when binocular vision is absent
tropia (eso or exo)
misalignemnt fo the eyes when both eyes are open and binocular vision is possible
optokinetic nystagmus
normal response to a continuously moving object
vestibulo-ocular nystagmus
by rotations of the subject’s head. Also irrigation fo the ear (caloric tes)
endpoint nystagmus
few beats of nystagmus in eccentric gaze
congenital nystagmus
jerk or pendular, present after birth and remains throughout life
periodic alternating nystagmus
horizontal jerk nystagmus that changes direction every 2 or 3 mins
downbeat nystagmus
present in primary position
upbeating nysagmus
in primary position is associated with lesions of the
differentiating between peripheral (vestibular) and central (brainstem) nystagmus
peripheral is unidirectional w fast phase away from lesion, visual fixation inhibits nystagmus and vertigo, tinnitus or deafness often present, romberg toward the slow phase, severe vertigo, short duration