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Flashcards in Neurology_TN_UW_0823 Deck (74)
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1

Greatest risk factor for prematurity

Cerebral Palsy

2

EEG of complex partial seizures

Normal or brief discharges

3

What is pseudomotor cerebri

idiopathic intracranial hypertension

4

Homocystinuria?

A/R disorder due to cystathionine synthase deficiency results in error in methionine metabolism

5

What are some of the pathognomoic feature/events in homocystinuria?

Intellectual disability and cerebrovascular accidents/thromobosis

6

Tx for homocytinuria

vit supplementation, antiplatelet or anticoagulation to prevent thromboembolic events

7

Loss of consciousness is seen with what kidn of partial seizures

Complex partial and partial seizure with secondary generalization

8

Infant botulism is caused by? Where can it come from?

Clostridium botulinum spores. Honey and also spores from environmental dust/soil.

9

What is the presentation of infant botulisnism?

Descending flaccid paralysis, autonomic symptoms like constipation, drooling, bulbar palsies (ptosis, sluggish pupillary responses, poor suck/gag reflex)

10

What is the treatment for infant botulism

Human derived botulism immune globin

11

Epidural hematoma clinical ppt

Lucid interval and then rapid deterioriation

12

CT appearance of epidural hematoma

Biconvex mass on CT scan

13

tx for epidural hematoma

If patients show deteriorating neurological status or increasing ICP = emergent craniotomy

14

Ascending polyneuropathy after recent GI infection?

Guillan barre

15

Underlying pathology of guillan barree involves what ?

Peripheral motor nerves

16

Final stage of guillan barre?

Flaccid paralysis, absent deep tendon reflexes and nerve conduction abnormalities

17

What does CSF in guillan barre show?

Elevated protein levels and normal cell count

18

Illnesses that involve anterior horn cells?

spinal muscular atrophy (flaccid paralysis in infancy), poliomyelitis, and ALS

19

What is the best means of monitoring respiratory function in patients with Guillan barre

Serial measurements of forced vital capacity

20

What are important predisposing risk factors for brain abscesses

otitis media, mastoiditis, frontal or ethmoid sinusitis, dental infection, bacteremia from other sites of infection, congenital heart disease

21

Classic triad of brain abscesses

Fever, headaches (nocturnal or morning), focal neurological changes. Seizures present in 25% of cases.

22

What is polycythemia

Increased concentration of hemoglobin

23

Infantile spasms can develop into

West syndrome (infantile spasms, psychomotor developmental arrest, hyperarrythmia) or lennox gastuaut

24

Typical EEG for infantile spasms

Hypsarrythmia (high voltage slow waves, spikes and polyspikes, background disorganization)

25

Infantile spasm onset

4-8 months

26

Tx for infantile spasms

Vigabatrin, ACTH, benzodiazepines

27

Lennox-Gastaut onset?

3-5 years

28

Lennox-Gastaut triad?

1) multipe seizure types 2) diffuse cognitive dysfunction 3) slow generalized spike and slow wave EEG

29

Tx for lennox-gaustaut

Valproic acid, benzoes, ketogenic diet; however, response often poor

30

Juvenile myoclonic epilepsy aka

Janz