Path 1.2 Necrosis Flashcards Preview

Question 1

1

types of necrosis

Answer

coagulative,
liquifactive,
caseous,
fat,
fibrinoid,
gangrenous

Question 2

2

coagulative

Answer

preservation of cell outlines of dead cells,
intense staining with eosin, more eosin binding sites bc the cell is dying

Question 3

3

liquifactive necrosis

Answer

tissue turns to liquid

Question 4

4

liquifactive necrosis etiology

Answer

occurs in absess (liquid-pus--a collection of neutrophils),
also occurs in the CNS (bc the brain when its injured does everything it can to maintain its 3D structure - surrounding brain tissue froms a gliosis to try and keep the 3D structure in the brain - by forming scars

Question 5

5

caseous necrosis

Answer

cheese-like (cream cheese)

Question 6

6

caseous necrosis etiology

Answer

majority is due to granulomas inflammation and TB.
Some fungi can also form this, this is the macrophages forming this by releasing cytokines that kill the tissue in the particular area

Question 7

7

fat necrosis

Answer

chalky white lesion - you get soaponification of fat liberating the fatty acid that react with calcium to form calcium salts in fatty acid.
Drop in calcium concentration is something you would see in fat necrosis

Question 8

8

fat necrosis example

Answer

injury to pancreas and it releases enzymes like lipases,
the lipases react with the fatty deposits in the omentum

Question 9

9

ex 2 of fat necrosis

Answer

fatty area of the body subjected to trauma
-->whole area of fat dies and just goes away

Question 10

10

Fibrinoid necrosis

Answer

walls of organs, bc of damage to the vessles. Caused by malignant hypertension and vasculitis due to increased permeability of vessel walls

Path 1.2 Necrosis Flashcards Preview

Pathology > Path 1.2 Necrosis > Flashcards

types of necrosis

coagulative,
liquifactive,
caseous,
fat,
fibrinoid,
gangrenous

coagulative

preservation of cell outlines of dead cells,
intense staining with eosin, more eosin binding sites bc the cell is dying

liquifactive necrosis

tissue turns to liquid

liquifactive necrosis etiology

occurs in absess (liquid-pus--a collection of neutrophils),
also occurs in the CNS (bc the brain when its injured does everything it can to maintain its 3D structure - surrounding brain tissue froms a gliosis to try and keep the 3D structure in the brain - by forming scars

caseous necrosis

cheese-like (cream cheese)

caseous necrosis etiology

majority is due to granulomas inflammation and TB.
Some fungi can also form this, this is the macrophages forming this by releasing cytokines that kill the tissue in the particular area

fat necrosis

chalky white lesion - you get soaponification of fat liberating the fatty acid that react with calcium to form calcium salts in fatty acid.
Drop in calcium concentration is something you would see in fat necrosis

fat necrosis example

injury to pancreas and it releases enzymes like lipases,
the lipases react with the fatty deposits in the omentum

ex 2 of fat necrosis

fatty area of the body subjected to trauma
-->whole area of fat dies and just goes away

Fibrinoid necrosis

walls of organs, bc of damage to the vessles. Caused by malignant hypertension and vasculitis due to increased permeability of vessel walls

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Path 1.2 Necrosis Flashcards Preview

Pathology > Path 1.2 Necrosis > Flashcards

types of necrosis

coagulative, liquifactive, caseous, fat, fibrinoid, gangrenous

coagulative

preservation of cell outlines of dead cells, intense staining with eosin, more eosin binding sites bc the cell is dying

liquifactive necrosis

tissue turns to liquid

liquifactive necrosis etiology

occurs in absess (liquid-pus--a collection of neutrophils), also occurs in the CNS (bc the brain when its injured does everything it can to maintain its 3D structure - surrounding brain tissue froms a gliosis to try and keep the 3D structure in the brain - by forming scars

caseous necrosis

cheese-like (cream cheese)

caseous necrosis etiology

majority is due to granulomas inflammation and TB. Some fungi can also form this, this is the macrophages forming this by releasing cytokines that kill the tissue in the particular area

fat necrosis

chalky white lesion - you get soaponification of fat liberating the fatty acid that react with calcium to form calcium salts in fatty acid. Drop in calcium concentration is something you would see in fat necrosis

fat necrosis example

injury to pancreas and it releases enzymes like lipases, the lipases react with the fatty deposits in the omentum

ex 2 of fat necrosis

fatty area of the body subjected to trauma -->whole area of fat dies and just goes away

Fibrinoid necrosis

walls of organs, bc of damage to the vessles. Caused by malignant hypertension and vasculitis due to increased permeability of vessel walls

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coagulative,
liquifactive,
caseous,
fat,
fibrinoid,
gangrenous

preservation of cell outlines of dead cells,
intense staining with eosin, more eosin binding sites bc the cell is dying

occurs in absess (liquid-pus--a collection of neutrophils),
also occurs in the CNS (bc the brain when its injured does everything it can to maintain its 3D structure - surrounding brain tissue froms a gliosis to try and keep the 3D structure in the brain - by forming scars

majority is due to granulomas inflammation and TB.
Some fungi can also form this, this is the macrophages forming this by releasing cytokines that kill the tissue in the particular area

chalky white lesion - you get soaponification of fat liberating the fatty acid that react with calcium to form calcium salts in fatty acid.
Drop in calcium concentration is something you would see in fat necrosis

injury to pancreas and it releases enzymes like lipases,
the lipases react with the fatty deposits in the omentum

fatty area of the body subjected to trauma
-->whole area of fat dies and just goes away