PERFUSION (EXAM 2) Flashcards

(84 cards)

1
Q

what are the contributing factors in perfusion

A

Heart
veins
arteries

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2
Q

what is the normal range for MAP

A

60 mmHg or greater

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3
Q

what is the main consequence of hypertension

A

there will be alot of perfusion issues

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4
Q

what are negative outcomes of bad perfusion

A

necrosis
loss of tactile function
fainting
confusion
DVT
decrease in organ function
ischemic pain

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5
Q

Normal BP

A

systolic <120
diastolic <80

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6
Q

Elevated BP

A

systolic 120-129
diastolic <80

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7
Q

Stage 1 high BP

A

systolic= 130-139
diastolic=80-89

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8
Q

stage 2 high BP

A

systolic= 140 or higher
diastolic= 90 or higher

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9
Q

Hypertensive crisis

A

systolic=higher than 190
diastolic=higher than 120

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10
Q

what is hypotension the most important risk factor for?

A

stroke
HF

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11
Q

Essential hypertension

A

the specific cause is unknown

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11
Q

Additive factors to contribute to essential hypertension

A

Genetics
behavioral
environmental factors
obesity
hyperlipidemia
African American

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12
Q

Secondary hypertension

A

it is the result of another disease
related to other issues

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13
Q

Example of secondary hypertension

A

renal artery disease
endocrine
cns disorders
sleep apnea

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14
Q

what happens when the cause of the hypertension is eliminated

A

hypertension goes away
BP return to normal

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15
Q

what can happen when hypertension is left untreated

A

cause end stage organ damage

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16
Q

GOAL of hypertension

A

reduce cardiovascular and renal morbidity and mortality

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17
Q

goal of hypertension

A

reduce cardiovascular and renal morbidity and mortality

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18
Q

Autoregulation

A

-the body tissues regulate their own blood flow to preserve 02
-think automatic

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19
Q

Parasympathetic N.S

A

little to no effect on vessels
dilates to blood vessels

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20
Q

Sympathetic N.S

A

Contraction of blood vessels to maintain BP
Maintain renin-angiotensin-aldosterone system (RAAS) in the kidneys

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21
Q

what factors affect blood flow

A

atherosclerosis
thrombus
embolus

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22
Q

what is the first action with a patient who has hypertension

A

non pharmological interventions

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23
Q

what is the BP goal

A

systolic less than 120
diastolic less than 80

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24
name some lifestyle modifications
weight Na intake regular physical activity moderate alcohol no smoking
25
lifestyle management
exercise 30 minutes/day at least 4 times a week
26
what lab test should you take
liver BUN creatinine
27
Name some manifestations of hypertension
headache blurry vision nausea vomiting
28
symptoms reflect target organ damage
angina MI HF stroke renal disease
29
categories of medications with hypertension do what
decrease BP decrease peripheral vascular resistance
30
what is peripheral vascular resistance
resistance that the blood encounters as it circulates through the body
31
where do these drugs work
Heart veins
32
what do alpha blockers do
they block vasoconstriction they open the veins and decrease vasoconstriction vasodilation of veins
33
what is clonidine and what does it do
it is an alpha blocker lowers BP
34
how does clonidine decrease BP
relaxing the arteries (vasodilation) and increasing the blood supply to the heart
35
what kind of alpha blocker is clonidine
alpha 2 agonist
36
what do clonidine do
-decrease sympathetic response -stimulate the alpha 2 receptor -reduce peripheral vascular resistance and increase vasodilation
37
what the main action of clonidine
reduce the peripheral vascular resistance which increases vasodilation
38
what happens when peripheral vascular resistance is decreased
there is vasodilation
39
what are some side effects of alpha 2 agonist
Remember it vasodilates -drowsiness -drymouth dizziness -bradycardia
40
why does alpha 2 agonist cause drowsiness
when the blood pressure decrease from vasodilation the brain will not get enough blood causing that drowsiness
41
alpha 2 agonist interventions
-monitor serum liver enzyme -do not stop abruptly -take as prescribed -may cause sodium and water retention
42
why must alpha 1 agonist do not stop abruptly
they might cause hypertensive crisis
43
what should be ordered with alpha 2 agonist
-A diuretic -there is a chance of sodium and water retention which causes that peripheral edema
44
who should avoid clonidine
those who are pregnant
45
how do you administer clonidine
oral transdermal patch
46
how do you take a patient off clonidine
gradually worst case scenario hold the medication and do assessment
47
What must be administered with clonidine
a diuretic clonidine is known to cause a lot of peripheral edema from the sodium and water retention
48
side effects/adverse effects of clonidine
Rebound hypertension if stopped abruptly drowsiness dizziness fatigue orthostatic hypertension
49
administration for clonidine with patients with hypertension
oral transdermal patch
50
when should clonidine be given and why
at bedtime because they cause drowsiness
51
how often should the patch be changed
every 7 days
52
before administering new patch what should you do
remove the old patch don't place the new one at the same spot
53
what is the most important pt teaching with clonidine
taper off DO NOT STOP ABRUPTLY REBOUND HYPERTENSION!!!!
54
What is the function of an ACE
it is a vasoconstrictor
55
what does an ace inhibitor do?
decreases vasoconstriction
56
Captopril is an example of a what
ace inhibitor
57
MOA of captopril
decrease effects of renin angiotensin crease vasoconstriction
58
captopril is medication of choice for who
patients with HTN and HF
59
what else does captopril do
decrease retention of sodium and water
60
Captopril is a first line drug for who why?
patients with diabetes slow the progression of renal impairment
61
primary effect of ACE inhibitor
cardiovascular effects decrease morbidity and mortality in those with HF
62
SE/AE of ACE inhibitors
dry non productive cough angioedema hypokalemia
63
what is the most common effect of ACE inhibitors
hyperkalemia
64
Interventions with ACE inhibitors
GO LOW AND SLOW!!!!! do not give with potassium sparing diuretics or salt subsitutes that contain potassium monitor dry cough, angiedema,BP,potassium
65
most important thing to note with ACE inhibitors
START LOW AND GO SLOW!!!!!
66
what should you not give ACE inhibitor with
-potassium sparing diuretics=spironolactone -salt substitutes that contain potassium TOO MUCH POTASSIUM
67
what should you do before administering ACE inhibitors
take BP
68
what are some S/S of hyperkalemia that you should educate your patient to look out for
muscle twitching palpitations weakness paresthesia
69
what else should you teach your patient
-report dry hacking cough -assess bp, hr, respiratory status -report swelling around the face, mouth and throat -refrain from taking potassium supplements
70
how do you know if a patient has angioedema
swelling in face mouth throat
71
can you give ACE inhibitors to a preg lady
NOOOO!!!!
72
what does ARBs stand for
angiotensin 2 receptor blockers
73
why is it better to give ARBs than ACE inhibitors
ARBs is less likely to cause hyperkalemia and dry cough
74
most ACE inhibitors end with?
PRIL
75
most ARBs end with
SARTAN
76
MOA of valsartan
they block the effects of angiotensin 2
77
SE/AE of ARBs
erectile dysfunction orthostatic hypotension NEUTROPENIA
78
what is neutropenia what should you do to monitor
low WBC count CBC
79
Interventions teaching with ARBs
treat angioedema monitor and report CNS effects Monitor BP and apical pulse hypotension fluid volume
80
nursing intervention with potassium high or low
monitor cardiac EKG
81
ARBs administration
oral with or without food
82
what should you do if ARBs is being taken with a diuretic or pt have liver failure
reduce the dose
83
what can ARBs be used with
diuretics if treating HTN or HF