Quiz 3 Flashcards

(38 cards)

1
Q

ketosis

A

disease of lactating cows characterized by decreased milk production, anorexia, dry feces, nervous signs (neuromuscular/ twitching), decreased body weight and ketone bodies in milk and urine (take blood or urine test)

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2
Q

ketosis characteristics

A

negative energy balance
TPR normal
pregnancy toxemia of sheep
hypoglycemia
increased circulating ketone bodies most prevalent within 6 weeks post partum in high producing cows due to lactation curve
increased salivation, abnormal behavior, abnormal chewing habits, exaggerated licking, trembling, shaking

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3
Q

secondary ketosis

A

something else there that led to ketosis (uterine infection, etc)
easier to treat

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4
Q

pregnancy toxemia of sheep

A

toxin in blood of sheep, similar presentation as cows but different timing
-occurs in older sheep, multiple lambs, as they grow negative energy balance occurs
-can’t get up, happens prior to lambing

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5
Q

hypoglycemia

A

low glucose in blood, low energy

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6
Q

peak intake (feed)

A

7 weeks

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7
Q

peak output (milk)

A

3 weeks

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8
Q

what causes negative energy balance

A

milk is higher than how much cow is eating due to different peak timing
all cows have this, not all have ketosis
post calving disease, can’t keep up with energy

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9
Q

ketosis diagnosis

A

ketostix- turn purple when positive for ketones
-chemical compounds that suppress appetite , can smell on breath (sweet)

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10
Q

ketosis treatment

A

glucose IV, glucocorticoids +/-, propylene glycol (oral), insulin (control in blood to be consistent)

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11
Q

ketosis prevention

A

avoid diet changes
introduce lactation ration 2 week prepartum

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12
Q

BHBA

A

blood test for acetones

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13
Q

hardware disease commonly known as

A

reticulitis
reticuloperitonitis
reticulopericarditis

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14
Q

hardware disease etioloy

A

small, sharp foreign objects frequently swallowed by ruminants during eating
commonly lodge in reticulum where strong contractions cause penetration through wall into the abdomen, through the diaphragm into throax into the heart

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15
Q

hardware disease clinical signs

A

anorexia
acute abdominal pain
drop in milk production
low grade pyrexia (not much, 102.5-104)
rumen stasis
grunt test positive
elevate front end
abducts elbows
peritonitis
increased WBC
reluctant to move

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16
Q

abducts elbows

A

away from body to expand chest

17
Q

grunt test

A

pain when push on chest, run away

18
Q

hardware disease diagnosis

A

history, clinical signs, pe, grunt test
-compass, metal detector, radiographs $, ultrasound
-chronic cases- washing machine heart sound

19
Q

hardware disease differential diagnosis

A

ketosis
LDA
metritis
MF
trauma

20
Q

hardware disease treatment

A

varies due to severity and chronicity
surgery-> rumenotomy (do to get to reticulum)
supportive care, rumen magnets, antibiotics, decrease physical activity, elevate front quarters, isolate cows from herd, TLC

21
Q

rumen magnent

A

give heifers @ 6 months
used to prevent, works until passes but don’t usually pass

22
Q

hardware disease prevention

A

installation of magnets in feed handling equipment
preventive oral administration of rumen magnets
twine bales instead of wire bales
stop bailing/ chopping too close to fence rows

23
Q

reticulum

A

at front part of cow

24
Q

milk fever

A

metabolic disorder caused by decrease blood calcium (hypocalcemia) usually associated with onset of lactation
usually in cows >5 years, not good at milking until about 5 years
failure of response or subclinical response of parathyroid glands to produce PTH which mobilizes calcium from bone stores

25
milk fever clinical signs
unsteady on feet, weak, progressing to downer cow head turned to one side, kink in neck, characteristic S shape curve to neck dilatated pupils, blank stare, cool/cold extremities temperature normal or below, increased pulse and respiration decreases anal reflex, absence of defecation, frequent straining and protrusion of the tongue
26
decreases anal reflex
wont constrict when thermometer entered, air gets in
27
milk fever differential diagnosis
injuries, toxemias, paralysis, septicemias, downer cow syndrome
28
milk fever treatment
IV calcium, give slowly (get up in 10-15 minutes), causes slow heart rate, have to watch how fast you give SQ or oral gels
29
milk fever prevention
minimize degree of hypocalcemia that occurs in susceptible cows in parturition dry cow ration is key, calcium: phosphorus ratio avoid thin/ fat cows at parturition minimize stress at calving time treating all cows at freshening time $, increased labor, not 100% effective
30
lateral recumbency
bad, can't get up from side and bloat and suffocate -prop up
31
left displaced abomasum
abomasum normally lies to right of ventral midline assumes an abnormal position on left side of abdomen between rumen and left body wall disease limited almost exclusively dairy cattle increased incidence in 4-6 year old cows occur within 45 days of lactation increased incidence of herd outbreaks when producer changes feed occurs often secondary to primary diseases (ketosis, metritis, mf)
32
left displaced abomasum clinical signs
anorexia, weight loss, decreased milk production characteristic high-pitched resonant tympanic (ping)
33
left displaced abomasum treatment
surgery is best roll and tack-tranquilize reef and stitch roll bumpy trailer ride
34
right displaced abomasum
can develop into abomasal torsion or "volvulus" (pain, shock, dehydration, colic, kick at stomach) complication: atonic abomasum
35
right displaced abomasum treatment
surgery plus FLUIDS
36
downer cow syndrome
inability to rise symptom not a diagnosis many diseases can ultimately cause a cow to go down phosphorus deficiency ?
37
4 M's of downer cows
rule outs 1. Metritis 2. Mastitis 3. Milk fever 4. Musculo-skeletal disorders
38
downer cow treatment
TLC IV supportive therapy MuSe injectable hip lifts or rubber inflatables fluids meet caloric intake needs