S2 L6.2: Congenital Heart Disease Part 2 Flashcards

(30 cards)

1
Q

One of the most common anomalies of the great vessels 8/10,000 births

A

Patent Ductus Arteriosus

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2
Q

From the bifurcation of PA to aorta just distal to the left subclavian artery

A

Ductus Arteriosus

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3
Q

Statement 1: Patent Ductus Arteriosus has a maternal history of rubella
Statement 2: If no CHF by age 10, most likely symptomatic in adulthood

a. TF
b. FT
c. TT
d. FF

A

a. TF
2: Asymptomatic

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4
Q

Membranous type is most common congenital cardiac defect
12/10,000 births
F:M = 1:1

A

Ventricular Septal Defect

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5
Q

In ventricular septal defect it may occur in isolation.
May occur in isolation
Small defects may undergo ___ (do not
need intervention)

A

Spontaneous Closure

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6
Q

Clinical Features of Ventricular Defect

A

Exertional dyspnea, chest pain, syncope, hemoptysis (coughing out of blood)

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7
Q

VS is divided into three major components:

A

Inlet, Trabecular, Outlet

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8
Q

○ Found in the muscular septum
○ Bordered entirely by myocardium
○ Can be trabecular, inlet, or outlet in location

A

Muscular VSDs

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9
Q

○ Often have inlet, outlet, or trabecular extension
○ Bordered in part by fibrous continuity between the leaflets of an AV valve and an arterial valve

A

Membranous VSDs

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10
Q

○ More common in Asian patients
○ Situated in the outlet septum
○ Bordered by fibrous continuity of the aortic and pulmonary valves

A

Doubly committed subarterial VSDs

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11
Q

● Most common cyanotic congenital heart disease
● Due to unequal division of the conus resulting from anterior displacement of the conotruncal system

A

Tetralogy of Fallot

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12
Q

Reversal of the shunting from usual L→R to R→L and develop a R sided heart failure

A

Eisenmenger Syndrome

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13
Q

Statement 1: Eisenmengerization could either be an equalization of pressure or flow reversal
Statement 2: Final consequence is flooding or inundating of the lungs d/t increased blood flow

a. TF
b. FT
c. TT
d. FF

A

c. TT

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14
Q

Development of pulmonary HTN in the presence of increased pulmonary blood flow

A

Eisenmenger Reaction

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15
Q

Association of VSD with pulmonary HTN and shunt
reversal

A

Eisenmenger Complex

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16
Q

D/t lack of O2, there will be an increase in production of vehicles that carry O2

A

Compensatory erythrocytosis

17
Q

Failure of the conotruncal septum to follow its normal spiral course and runs straight down

A

Transposition of the Great Arteries

18
Q

T/F: In the transposition of the Great Arteries displays cyanosis at birth which worsens shortly therafter when ductus closes

19
Q

Failure of conotruncal ridges to fuse and to descend toward the ventricles

A

Persistent Truncus Arteriosus

20
Q

In Persistent Truncus Arteriosus:
Statement 1: Pulmonary artery arises some distance above the origin of the divided truncus
Statement 2: Always with a VSD to redirect the blood flow

a. TF
b. FT
c. TT
d. FF

A

b. FT
Undivided Truncus

21
Q

Symptoms of heart failure and poor physical development usually appear in the __

A

First weeks or months of life

22
Q

Facial dysmorphism, a high incidence of extracardiac malformations, atrophy or absence of the thymus gland, Tlymphocyte deficiency, and a predilection to infection

A

DiGeorge syndrome

23
Q

Prevalence of truncal valve regurgitation increases with age, causing __ and increasing susceptibility to __

A
  1. biventricular heart failure
  2. endocarditis
24
Q

Involves obliteration of the right AV orifice
Absence or fusion of tricuspid valve
Severe cyanosis due to obligatory admixture of systemic and pulmonary venous blood in the left ventricle

A

Tricuspid Artresia

25
If there is no tricuspid valve, how will the blood in the right atrium reach the lungs?
Have to open the PFO and then blood will flow into the left atrium
26
If there is no tricuspid valve, how will blood in the left atrium reach the pulmonary bed?
There must be an opening, VSD, so that it can flow into the lungs via the pulmonary artery
27
Can pts with CHD exercise or do sports?
Yes and No, if pt is then corrected and the pt stamina or physical status/functional capacity is poor, then definitely you can’t allow them to do sports
28
Why do heart defects predispose an individual to infection?
● Bacteria loves to thrive in abnormal places ● Like in valvular heart diseases and mitral stenosis, pathogens love to live in these abnormal areas
29
6-STEP APPROACH TO EXERCISE IN CHD
1. Know your patient 2. Assess functional parameters 3. Decide statis component of exercise 4. Do CP stress testing 5. Execute exercise program 6. Followup
30
Pathway of Circulation in the Blood a. Right Ventricle b. Bicuspid Valve c. Lungs d. Tricuspid Valve e. Left Ventricle f. IVC & SVC g. Pulmonary Semilunar Valve h. Left Atrium i. Pulmonary arteries j. Aortic Semilunar Valve k. Right Atrium l. Pulmonary veins m. Aorta
F K D A G C L H B E J M