Small ruminants Flashcards

(555 cards)

1
Q

what are the three main ways of managing infectious/parasitic disease?

A

biosecurity
reduce disease challenge
improve animal resistance

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2
Q

what are the three periods of a ewes years?

A

dry (3 months)
pregnant (5 months)
lactating (4 months)

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3
Q

what are common issues associated with inefficiency in sheep?

A

ewe/lamb mortality
chronic wasting diseases
poor nutrition
parasites, lameness, mastitis…

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4
Q

what characteristics are need in the stratification system?

A

lamb vigor
good mothering
growth rates
disease resistance

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5
Q

what are some of the main estimated breeding values of sheep?

A

litter size
maternal ability
scan weight lamb
carcass composition/conformation
mature size
worm resistance

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6
Q

what is the deadweight for lamb?

A

£5-6 per kilo

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7
Q

what is the price of a live lamb?

A

£250

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8
Q

what is the cost of a replacement ewe?

A

£200

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9
Q

what is the cost of a cull ewe?

A

£90

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10
Q

what are the general KPIs of sheep?

A

scanning %
lambing %
ewe mortality
culling ewe
ewe:tup ratio
lamb mortality

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11
Q

are sheep short or long day breeders?

A

short

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12
Q

what is the main advantage of spring lambing?

A

easy management and good grass growth

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13
Q

what is the main advantage of Christmas lambing?

A

better lamb prices at easter market

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14
Q

what are store lambs?

A

lambs that haven’t been finished that summer so are minimally fed through winter (no growth) for compensatory growth the following spring

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15
Q

why do store lambs not grow over winter?

A

concentrates are too expensive

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16
Q

how does seasonality work? (brain)

A

light enters eye
travels in optic nerve through hypothalamus to the superior cervical ganglion and then pineal gland
pineal gland then does/doesnt produce melatonin

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17
Q

what effect does light have on melatonin production?

A

blocks production

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18
Q

in spring melatonin decreases, how does this effect the GnRH of sheep?

A

decreases the pulses and hence decreases FSH and LH so the sheep doesn’t cycle

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19
Q

what do follicles produce? and what does this do?

A

oestrogen that suppresses the FSH

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20
Q

how long is the oestrus cycle in sheep?

A

16-18 days

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21
Q

how many follicular waves do sheep have?

A

2-5 waves

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22
Q

what is the ideal ewe to ram ratio?

A

1 ram per 30 ewes

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23
Q

why isn’t artificial insemination used as commonly in sheep?

A

structure of the cervix is different (has to be done laparoscopically)
low number of straws per ejaculate compared to cattle

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24
Q

what determines the fecundity of sheep?

A

bone morphometric protein signalling from ovary

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25
what is the ram effect?
introduction of a male after 2 weeks of male absence (also works if new ram is introduced) that triggers cyclicality in ewes
26
what happens to the oestrus activity after male introduction? (ram effect)
1st ovulation (can be silent) 19 days - second ovulation (first for some) 25 days - second ovulation for ones that had silent first
27
what is the name of the ram often used for the ram effect?
teaser - vasectomies
28
how can seasonality be manipulated?
expose to artificial light (difficult with sheep) melatonin supplement genetic selection
29
how can the ram effect be used to alter seasonality?
alter the rams seasonality (melatonin...) then introduce to ewes and they will alter their seasonality due to the ram effect
30
how is melatonin given to a sheep?
ear implant (regulin)
31
what is done on day 1 of a melatonin plan to induce seasonality?
30 weeks prior to lambing move ewes out of sight/sound/smell of ram
32
what is done on day 7 of a melatonin plan to induce seasonality?
implant melatonin in ear base
33
what is done on day 42 of a melatonin plan to induce seasonality?
introduce ram (30-40 days post implant)
34
what is the short term effect of flushing?
effects the current follicular wave (3-7 days)
35
what is the long term effect of flushing?
allows sheep to come out of seasonal trough in fertility sooner (6-8 weeks)
36
what is the static effect of flushing?
ewes with higher BCS have higher ovulation rate
37
what is the dynamic effect of flushing?
ewes that are increasing in BCS have a higher ovulation rate
38
what is the only method to hormonally synchronise ewes?
chronogest - progesterone sponge
39
how long do progesterone sponges go in the vagina for?
12-14 days
40
what is the issue with progesterone sponges?
progesterone is an immunosuppressant so can get vaginitis
41
when combined with PMSG injection at sponge removal, when does oestrus occur?
36-72 hours after sponge removal
42
what hormone does PMSG have the same activity as?
FSH
43
how long should you wait after removal of the progesterone sponge (with PMSG injection) before introducing the ram? and why?
48 hours to ensure ram doesn't over serve first ewes coming into oestrus and deplete their semen reserves (then most ewes will be in heat)
44
how long is a CIDR left in a sheep?
12 days and eCG on removal
45
how can lambing be induced?
dexamethasone injection (dexafort) from day 140 after ram has been in - this isn't authorised
46
how long after inducing a ewe with dexamethasone will they lamb?
around 40 hours later
47
when are most replacement ewes introduced to the flock?
19 months
48
what is the optimal BCS of a ewe at mating?
3.5
49
how long does it take sperm to develop in ewes?
roughly 60 days (ensure you check rams well before mating)
50
what is the optimal BCS of a ram before breeding?
3.5
51
what is the rough gestation of a sheep?
145 days
52
how much should a ewe be fed from conception to 42 days?
just maintenance
53
how much should a ewe be fed from 42-90 days gestation?
maintenance plus 2 MJ/day
54
why is it important to feed well from day 42-90 of gestation?
considerable placental growth - underfeeding retards placenta and hence foetal growth
55
what type of scanning head is used to PD ewes?
sector - broader range of vision into the abdomen
56
roughly when are ewes scanned?
70 days (ensure ram has been out for 28 days)
57
why is nutrition important from 90 days to parturition in ewes?
rapid foetal growth colostrum quality/quantity (mammary development) prevent metabolic disease
58
how much food do ewes require at lambing time?
2x maintenance (concentrates)
59
how should a vasectomy be closed?
don't use nice opposing sutures - want to leave a scar to be able to identify the ram
60
what is checked on a ram pre-breeding examination?
feet brisket (sores for raddle...) caseous lymphadenitis (lymph nodes) scrotal measurement and palpation (30cm, firm, even...) exteriorise penis and vermiform appendage semen examination libido testing (mounting ewes)
61
where is the sperm stored?
epididymis
62
what are some bacteria that can cause epididymitis?
Actinobacillus seminis Haemophilus somnus Histophilus ovis
63
what is epididymitis associated with?
ascending urinary tract infection intensively reared trough fed rams (lie down more)
64
how can epididymitis spread be prevent between rams?
have small groups that graze and rotate between clean paddocks
65
what should be done at the time of treating inguinal hernias in rams?
castrate - stop possible genetic/congenital spread
66
what is the usual cause of testicular degeneration or hypoplasia?
specific local injury (can also be due to extreme fatness, stress, inflammation...)
67
how does scrotal mange appear in rams?
crusty scabs on lower 1/3 of scrotum sore weeping dermis
68
what causes scrotal mange?
Chorioptes bovis
69
how can scrotal mange be confirmed?
skin scrape in multiple places (low mite number)
70
what is an effective treatment of scrotal mange in rams?
doramection (ivermectin also used) diazinon sheep dip is effective
71
what is balanoposthitis also known as?
pizzle rot
72
how does pizzle rot present?
small ulcers and discharge at prepuce scabbing and fly strike
73
what bacteria is pizzle rot associated with?
Corynebacterium renale
74
what are the risk factors for pizzle rot?
high protein concentrates and lush grass
75
what metabolic diseases are seen around lambing time?
pregnancy toxaemia (twin lamb disease) hypocalcaemia hypomagnesaemia
76
when are lambs usually weaned?
early summer (summer period to recover before tupping)
77
what is the target BCS of a ewe at weaning?
2.5
78
what is the target BCS at tupping for a lowland ewe?
3.0-3.5
79
how much of an increase in BCS is aimed for when flushing sheep?
0.5
80
why is appropriate feeding important in mid pregnancy of ewes?
placental development (underfed means low birth weight) mammary development
81
what are the aims of feeding the ewe in late pregnancy?
support lamb growth support mammary tissue development colostrum production prevent pregnancy toxaemia (ketosis) ensure strong healthy lamb underfeeding related to mastitis and maternal behaviour
82
what is the most important aspect of ewe nutritional ration in late pregnancy?
good quality forage
83
what factors could be used to split ewes into feeding groups?
single, twins, triplets BCS lambing dates separate young ewes
84
why does care need to be taken with trough space in pregnant ewes?
ewes are fatter so require more room
85
what are the three main requirements of colostrum?
energy, protein, antibodies
86
what is used as a rough estimate of milk production in ewes?
8 week weight of lamb
87
what age will lambs start to eat grass?
6 weeks old
88
what age of lambs weaned?
10-16 weeks
89
what may indicate there is an issue with peri-parturient ewe nutrition?
twin lamb disease hypocalcaemia lamb mortality/morbidity (weak small lambs, watery mouth, joint ill/infection, hypothermia...) mastitis lamb growth
90
what is pregnancy toxaemia?
energy deficiency (hypoglycaemia and hyperketonaemia)
91
what are the risk factors for pregnancy toxaemia?
last 6 weeks of pregnancy thin/fat ewes stress broken mouth concurrent disease multiple foetuses inadequate/change in diet
92
what are the clinical signs of pregnancy toxaemia?
(neurological due to low glucose) separate from group inappetent blindness, tremors, hyperaesthesia, star gazing recumbency and death
93
what can be measured to confirm pregnancy toxaemia?
beta-hydroxybutyrate (greater than 3mmol and they will show clinical signs)
94
what is done to treat pregnancy toxaemia?
separate and give fresh food/water IV glucose (dextrose) propylene glycol calcium supplement NSAIDs (meloxicam...)
95
what can be considered if a pregnant ewe isn't improving from pregnancy toxaemia?
induce with dexamethasone (after 135 days gestation) caesarian or euthanasia
96
what mechanism mediates the mobilisation of calcium from bones? (hypocalcaemia)
parathyroid hormone and vitamin D
97
what is the difference between when hypocalcaemia occurs in sheep and cattle?
sheep usually occurs pre-lambing (6 weeks)
98
what are possible risk factors for hypocalcaemia in ewes?
stress (transport, snow, gathering...) acidosis older ewes rapid growing lush pasture
99
what are the clinical signs of hypocalcaemia?
weakness and excitability recumbency dilated pupils, constipation, bloat, coma death
100
what is done to treat hypocalcaemia?
slow IV calcium SC calcium
101
what is the classic history for hypomagnesaemia in ewes?
post lambing at peak lactation lush grass or bare pasture
102
what are the clinical signs of hypomagnesaemia?
(neurological) excitable, tremors, convulsions death
103
what are some key points about dealing with dystocia as a vet?
wear gloves/be clean lots of lubricant NSAIDs epidural antibiotics (5 days) colostrum to lamb
104
what is the method of a simple embryotomy?
remove fore limbs - cut through skin above carpus then up to elbow, undermine attachment of shoulder and remove leg repeat for other leg
105
what are some possible issues to look at when getting dystocia related deaths?
ewe age ram breed/size/characteristics ewe BCS hygiene lambing/pens staff numbers/training
106
what are the indications for caesareans?
oversized lamb ringwomb vaginal prolapse foetal monster malpresentation
107
what are the contraindications for caesarians?
rotten smelly lambs (contamination into abdomen and wound breakdown)
108
how should a ewe be placed for caesarian?
right lateral recumbency (left side up)
109
what are the options for anaesthetic for caesarians?
line block (local infiltration) - procaine inverted L block paravertebral - T13, L1-3 sacra-coccygeal epidural
110
what is the site for making an incision for caesarians in sheep?
15cm incision between last rib and wing of ileum 10cm below transverse process
111
what type of suture is used for the uterus in caesarians?
continuous inverting pattern (catgut often used)
112
what is metritis?
infection of all the layers of the uterus (endothelium to muscle)
113
what usually causes metritis?
opportunistic environmental bacteria
114
what are the risk factors for metritis?
dystocia poor hygiene dead lambs or abortion
115
what are the clinical signs of metritis?
dull, depressed, recumbent (systemic illness) toxaemic with congested MM red/brown or purulent vaginal discharge
116
what is done to treat metritis?
IV antibiotics - oxytetracycline NSAIDs
117
what are some possible risk factors for vaginal prolapses?
hill breeds litter size age history of prolapse nutritional - obesity, hypocalcaemia, bulky poor quality forage high stocking rates slopes (anything increasing intra-abdominal pressure and reduced pelvic floor strength)
118
what are possible treatments for mild prolapse cases?
retention device harnesses NSAIDs LA antibiotics
119
how are prolapses treated surgically?
epidural (1.75ml procaine and 0.25ml xylazine) NSAIDs antibiotics clean, replace and suture shut (purse string) mark to remove sutures
120
when does mastitis occur in sheep?
after lambing
121
what are the three main presentations of mastitis?
peracute/gangrenous/toxic acute chronic
122
why is mastitis often more serious in sheep?
can't monitor them as closely because they don't go through a milking parlour everyday
123
when does gangrenous mastitis occur?
4-8 weeks post lambing (peak lactation)
124
how does the udder/milk of a ewe with gangrenous mastitis present?
swollen, cold, blue boundary between diseased and normal tissue diseased tissue often sloughs off milk severely discoloured
125
how will a ewe with gangrenous mastitis present?
sick ewe toxaemia, rumen stasis, inappetent, lame, recumbent (lambs will be hungry - making noise)
126
when does acute mastitis occur?
immediately post-lambing or 4-8 weeks later at peak lactation
127
how does the udder and milk present in acute mastitis cases?
swollen, red, painful clots, discoloured, watery
128
when is chronic mastitis often detected in ewes?
at drying off/weaning or possibly culling
129
how does the udder and milk present in chronic mastitis cases?
abscesses, swollen light purulent discharge
130
what are the most common bacteria causing mastitis in UK ewes?
Staph aureus Mannheimia haemolytica
131
what is the source of Staph aureus that leads to mastitis?
teat skin
132
what is the source of Mannheimia haemolytica that leads to mastitis?
lambs mouths
133
what are the risk factors for mastitis?
under nutrition in late pregnancy and lactation (metabolic stress) over suckling lambs and teat lesions concurrent disease multiple lambs older ewes abnormal udder confirmation
134
how is gangrenous mastitis treated?
euthanasia or cull
135
how is acute mastitis treated?
systemic antibiotics (culture and sensitivity) - amoxicillin as first line strip udder IV fluids NSAIDs
136
how is chronic mastitis treated?
cull
137
how can mastitis be prevented?
good ewe and lamb nutrition (creep feed) wean abruptly control disease (orf, worms...) cull older and diseased ewes good bedding hygiene
138
what is a neonatal lamb?
period straight after birth (<2 weeks old)
139
what does perinatal mean?
the time around parturition
140
what can causes death in the first 0-24 hours of a lamb being born?
birth stress (results in failure to suck, hypothermia...) anoxia/hypoxia ruptured liver fractured ribs brain haemorrhage
141
what are some possible infections that lambs can get postpartum?
watery mouth salmonella cryptosporidia septicaemia meningitis joint ill clostridial disease pasteurellosis
142
what are the three overall factors which result in lamb loss?
lamb factors (weight, vigour, vaccination...) ewe factors (genetics, stress, colostrum...) environment (hygiene, equipment, shepherds...)
143
how much colostrum does a lamb require in the first feed?
50 ml/kg
144
how much colostrum does a lamb require in the first 24 hours?
200 ml/kg
145
how can failure of passive transfer be measured?
serum total protein (should be greater than 5.5g/dL
146
what are some risk factors for poor colostrum intake?
concurrent disease ewe undernutrition breeding - mothering... ewe age multiple births lambing environment - stress, weather, shepherd...
147
what can be used to help strip out ewes?
oxytocin
148
what are the risk factors for primary hypothermia of lambs?
outdoor lambing younger ewes wet weather low birth weight feed failures
149
what is done to treat lambs with hypothermia?
give glucose and place in warming box (intraperitoneal glucose)
150
why is it important to give hypothermic lambs glucose before warming?
as they warm and metabolism starts they will have a hypoglycaemic fit
151
how is an intraperitoneal injection done?
just to one side of the naval at a 45 degree angle
152
what age do lambs get watery mouth?
12-72 hours
153
what are the clinical signs of watery mouth?
dull unwilling to suck normothermic saliva drool on muzzle (watery mouth) profuse lacrimation abomasal tympany constipation or retained meconium
154
what is the main finding on post mortem of a lamb with watery mouth?
distended abomasum with gas, saliva and milk clots (bacteraemia signs)
155
what causes the clinical signs of watery mouth?
endotaemia (large numbers of endotoxin in blood)
156
what is there proposed hypothesis of why watery mouth occurs?
bacteria enter the abomasum before milk and is absorbed using the same transporters as milk these bacteria multiply in blood and then mass lysis leads to endotoxin release
157
what are the main risk factors for watery mouth?
dirty environment large litters poor colostrum intake
158
what can be used to treat watery mouth?
enteral/parenteral antibiotics (streptomycin orally, amoxicillin injection...) glucose/electrolytes stomach tube withhold milk until lamb can suck
159
does metaphylactic use of antibiotics work for watery mouth?
yes - but just as well as good colostrum intake (this is a management failure - colostrum and hygiene)
160
what causes lamb dysentery?
Clostridium perfringens type B (beta and epsilon toxins)
161
what age lambs is lamb dysentery seen in?
1-3 days old
162
what are the clinical signs of lamb dysentery?
sudden death and haemorrhagic diarrhoea (occasionally)
163
what is found on post mortem of lambs with lamb dysentery?
dark red intestines blood stained peritoneal fluid pale friable liver
164
how can lamb dysentery be prevented?
vaccinate ewes with multivalent clostridial vaccines
165
how often is enterotoxigenic E. coli seen?
uncommon
166
what age is enterotoxigenic E. coli seen in lambs?
24-48 hours
167
what is the main clinical sign of enterotoxigenic E. coli?
watery brown diarrhoea
168
what are the clinical signs of neonatal salmonellosis?
weakness profuse watery diarrhoea (blood stained) dehydration death
169
why does care need to be taken when treating lambs with neonatal salmonellosis?
it is zoonotic
170
what is the pathogenesis of cryptosporidium?
villous atrophy in distal small intestine causing malabsorption and diarrhoea
171
what is drunken lamb syndrome also known as?
lamb nephrosis
172
what age is drunken lamb syndrome seen?
7-10 days 1-2 months
173
what causes the clinical signs associated with drunken lamb syndrome?
increased D-lactate
174
what produces D lactate?
bacterial fermentation
175
what can be used to treat drunken lamb syndrome?
sodium bicarbonate solution
176
what age is joint ill seen?
2-3 weeks old
177
what is the main clinical sign of joint ill?
sudden onset lameness (single/multiple joints)
178
what can be done to treat joint ill?
antibiotics and NSAIDs (often poor response)
179
what is the main bacteria causing joint ill in lambs?
Streptococcus dysgalatiae
180
how can joint ill be prevented?
colostrum, hygiene, naval dipping... (multiple factors)
181
what antibiotic is Streptococcus dysgalatiae joint ill resistant to?
tetracyclines
182
what is a much less common cause of joint ill, but is seen in lambs from 6 weeks old to 6 months old?
Erysipelothrix rhusiopathie
183
what is naval ill?
ascending infection of the naval leading to abscessation and omphalophlebitis
184
what are some possible sequelae of naval ill?
peritonitis, liver abscessation, systemic infection, polyarthritis...
185
what can Fusobacterium necrophorum cause in naval ill cases?
hepatic necrobacillosis
186
what is the characteristic appearance of hepatic necrobacilosis?
white spots on liver (areas of necrosis)
187
what can be done to prevent/control naval ill?
iodine naval within 15 minutes then repeat a few hours later goof hygiene colostrum treat - penicillin, potentiated amoxicillin
188
what substance can predispose to iodine deficiency in sheep?
thiocyanate (contained in some plants - root crops...)
189
how does iodine deficiency present relating to the neonatal lamb?
goitre of newborn late abortions minimal fleece on lambs
190
how much should a thyroid gland weigh at birth?
>0.4 g/kg live weight
191
how can iodine deficiency be prevented?
ewe supplementation (inject/bolus/drench) treat lamb with oral potassium iodide
192
what are some antagonists found in soil that can lead to copper deficiency?
iron, molybdenum, sulphur
193
what does copper deficiency cause in lambs?
swayback head tremor osteoporosis tendon abnormalities depigmented and poor quality wool (steely wool) anaemia
194
what is used to diagnose copper deficiency in lambs?
clinical signs histopathology of brain/spinal cord liver copper concentration
195
how is copper deficiency treated in lambs?
euthanasia (no effective treatment)
196
how can copper deficiency of lambs be prevented?
supplement ewe during pregnancy (care with toxicity of certain breeds...)
197
what can be used to supplement copper in ewes?
chelated copper injection mineral drench/supplements capsules/bolus (only use one source... toxicity!!)
198
what causes white muscle disease?
vitamin E and selenium deficiency
199
what is white muscle disease also known as?
nutritional muscular atrophy stiff lamb disease
200
what are the clinical and pathological signs of white muscle disease in lambs?
weak lambs that don't suck histopathology of cardiac musculature
201
what is used to treat white muscle disease?
vitamin E potassium selenate
202
how can white muscle disease be prevented?
supplementation at least 6 weeks before lambing
203
what is atresia ani?
failure of anus and sometimes part of colon/rectum to form properly
204
what is entropion?
in turned eyelids (usually lower)
205
what is the most successful way to treat entropion?
surgery
206
how are prolapsed intestines through the umbilicus treated?
general anaesthetic and surgically prepare/clean intestines enlarge the hole, replace and suture closed
207
before opening a lamb up for post mortem, what should be done?
weigh (excessive suggested dystocia) look at feet - hardened means they've walked meconium staining, trauma, swelling, scour...
208
when opening a lamb carcass, what should be inspected?
brown fat around kidneys (energy supply) milk/colostrum in abomasum lung aeration haemorrhage and trauma evidence of infection
209
what are signs on lamb post mortem of dystocia?
meconium staining swollen head/tongue head/shoulder oedema fractures haemorrhage
210
what are signs of lamb post mortem of hypothermia/starvation?
no brown fat minimal body fat no milk in abomasum
211
what are the main differential diagnoses for diarrhoea in lambs?
Nematodirus battus PGE coccidiosis Cryptosporidium acidosis Clostridium perfringens type B (lamb dysentery) and D (pulpy kidney) E. coli Salmonella spp.
212
what does Clostridium perfringens type B cause in lambs?
lamb dysentery
213
what does Clostridium perfringens type D cause in lambs?
pulpy kidney
214
what are the clinical signs of cryptosporidiosis in lambs?
profuse diarrhoea (blood) dehydration death
215
what age lambs does cryptosporidiosis effect?
3-7 days old
216
how can cryptosporidiosis in lambs be diagnosed?
faecal sample 6-10 from infected group and stain smear post mortem histopathology
217
how is cryptosporidiosis treated?
supportive oral fluids multiple times a day (no licensed drugs)
218
what are ways of preventing cryptosporidiosis?
hygiene, reduced stocking density, clean pasture lamb nutrition/colostrum
219
what type of organism is cryptosporidium?
protozoa
220
what type of organism is coccidia?
protozoa
221
what is the source of coccidia infection?
ewes or older lambs
222
what are the risk factors for coccidiosis?
high stocking rates poor colostrum stress, concurrent infection and mixed ages
223
is finding coccidia species in faeces a definitive diagnosis?
no - many of the strains are non-pathogenic
224
what age lambs are often infected with coccidiosis?
4-8 weeks old
225
what are the clinical signs of coccidiosis?
diarrhoea, tenesmus, fever, weight loss, death (sub-clinical infections lead to poor growth)
226
how can coccidiosis be diagnosed?
faecal sample 6-10 from group then do a coccidial count (will need speciation as well to confirm pathogenic strains - usually treat whilst awaiting these results)
227
how is coccidiosis treated?
supportive oral fluids multiple times a day drugs - toltrazuril or diclurazil (can use metaphylactically)
228
how can coccidiosis be prevented?
hygiene, stocking rates, colostrum, nutrition batch rearing prophylactic/metaphylactic treatment
229
what causes acidosis in lambs?
consumption of rapidly fermentable carbohydrates (grain...)
230
what events occur leading to acidosis when a lamb consumes high volumes of concentrates?
fall in rumen pH due to lactic acid production which causes inflammation (rumenitis) leading to the blood being acidotic
231
what are possible sequelae of acidosis in lambs?
liver abscessation fungal (overgrowth) rumenitis death
232
what are the clinical signs of acidosis in lambs?
death dull, depressed, teeth grinding, colic, bloat, ataxic, recumbent dehydration, diarrhoea, no rumen sounds
233
how can acidosis be diagnosed?
clinical signs rumenocentesis (pH <5.5) post mortem
234
how can acidosis of lambs be treated?
IV fluids (plus bicarbonate) oral fluids and multivitamins good quality hay penicillin
235
what are the possible causes of rumen bloat in sheep?
grain overload, oesophageal obstruction, legumes (frothy)
236
what side will be distended if a sheep has rumen bloat?
left
237
what can be done to treat rumen bloat of sheep?
stomach tube, dimeticone, trochar (red devil)
238
what age is lamb nephrosis syndrome seen?
2-12 weeks old
239
how can lamb nephrosis be diagnosed?
raised urea, creatine and hyperkalaemia swollen kidneys and necrosis on post mortem
240
what is cobalt deficiency of lambs known as?
ill thrift
241
what vitamin is cobalt a constituent of?
B12
242
where is vitamin B12 synthesised in lambs?
rumen
243
what is cobalt important for?
red blood cell development amino acid synthesis energy metabolism
244
what are the clinical signs of cobalt deficiency?
weight loss anaemia slow growth debility watery eye discharge (just generally don't do very well)
245
how is cobalt deficiency diagnosed?
cobalt and vitamin B12 levels in blood/liver
246
what can be used to treat cobalt deficiency?
cobalt bolus/drench vitamin B12 injection cobalt in creep pasture treatment
247
what are the functions of selenium?
anti-oxidant immune function
248
what effects will a selenium deficiency have on lambs/ewes?
white muscles disease poor reproductive performance
249
what are the consequences of iodine deficiency?
death and weakness late abortions thyroid goitre (neonates are more susceptible to disease)
250
what are the clinical signs of copper poisoning?
ataxic, head pressing, jaundice, haemoglobinuria, recumbency, death
251
what can be used to treat copper poisoning?
(no specific treatment) sodium calcium EDTA supportive remove copper source add molybdenum and sulphur to water (binds copper)
252
what are bacterial causes of pneumonia in lambs?
Mycoplasma spp. Mannheimia haemolytica Pasturella multocida
253
what respiratory viruses can cause pneumonia in lambs?
parainfluenza virus respiratory syncytial virus
254
what lung worm effects lambs?
Dictyocaulus filaria
255
when is Mannheimia haemolytica found as a commensal in sheep?
upper respiratory tract - nasopharynx (causes disease when it enters the lungs)
256
what is usually the driving factor for Mannheimia haemolytica to move into the lungs and cause pneumonic pasturellosis?
stress/disease - diet change, worms, ticks, housing, transport, bad weather...
257
what are the clinical signs of Mannheimia haemolytica respiratory disease?
pyrexia, mucopurulent nasal discharge, cough, increased RR/depth
258
what can be used to treat pneumonic pasteurellosis (Mannheimia haemolytica)?
oxytetracycline, amoxicillin, macrolides (5-7 days) - long acting is useful
259
how is pneumonic pasteurellosis (Mannheimia haemolytica) diagnosed?
consolidated lungs and culture
260
what can be done to prevent pneumonic pasteurellosis (Mannheimia haemolytica)?
vaccination - heptavac P, ovipast investigating underlying problem/risk factors - stress
261
what are the clinical signs of mycoplasma pneumonia?
(more chronic disease) cough, slight nasal discharge, increased RR/depth low grade pneumonia
262
what group of animals is mycoplasma pneumonia mainly seen in?
housed over stocked lambs
263
what can be used to treat mycoplasma pneumonia?
long acting oxytetracycline and macrolides
264
what time of year is lung worm seen in sheep?
summer/autumn lambs at pasture
265
how is lung worm diagnosed?
larva in faeces (baermans test)
266
at is used to treat lung worm?
anthelmintics (BZ, ML, LV) antibiotics NSAIDs and supportive treated
267
what should the 8 week growth rate of a lamb be?
300-500g/day
268
what efficacy means that there is resistancd to the anthelmintic?
<95% efficacy
269
what are the possible ways in which nematodes can cause disease?
damage lining of guts (reduce absorption) remove nutrients remove blood damage/provoke host immune response
270
what age sheep is Nematodirus battus seen in?
6-12 weeks old
271
what is the main clinical sign of Nematodirus battus?
acute severe diarrhoea
272
what age and time of year is type one Telodorsagia infection seen?
FSG lambs in mid summer onwards
273
what age and time of year is type 2 Telodorsagia infection seen?
yearling in winter months
274
what is the classic clinical sign of Trichostrongylus?
black scour
275
what age and time of year is Trichostrongylus seen?
lambs in later summer to autumn
276
what is the classic clinical sign of Haemonchus contortus?
acute regenerative anaemia and bottle jaw (no diarrhoea)
277
what is the typical Trichostrongyle lifecycle?
eggs passed in dung and develop to L1 and L2, L3 is then dispersed onto pasture and ingested by the sheep where they devlop to L4 and adults to produce eggs
278
what is the prepatent period of a typical trichostrongyle lifecycle?
21 days
279
what parasites have a typical trichostrongyle lifecycle?
Telodorsagia, Trichostrongylus and Haemonchus
280
what does infectivity of pasture with nematodes peak in late summer?
shorter lifecycle due to warmer weather and large output from lambs
281
where the majority of the eggs in Haemonchus come from?
sheep (don't overwinter well on pasture)
282
what is hypobiosis?
L3 in late autumn have arrested development over the winter period
283
how does Nematodirus battus infect the pasture?
eggs shed by lambs remain on the pasture until the following year and then hatched when triggered by prolonged chill and temperature increase to infect the next set of lambs
284
when do lambs begin to develop resistance to nematodes?
5-6 months old
285
what are the five groups of anthelmintic?
benzimadazole levamisole macrocytic lactones amino-acetonitrile spiroindoles
286
what colour are benzimadazole drenches?
white
287
what colour are levamisole drenches?
yellow
288
what colour are macrocytic lactone drenches?
clear
289
what colour are amino-acetonitrile drenches?
orange
290
what colour are spiroindoles?
purple
291
how do benzimadazoles work?
tubulin binding to prevent glucose uptake
292
how do levamisole's work?
ganglionic blocking causing paralysis
293
how do macrocytic lactones work?
block chloride and GABA channels causing paralysis
294
what colour are microcytic lactone drenches?
clear
295
what internal parasites can benzimadazoles be used for?
broad spectrum against roundworms tapeworms albendazole work against fluke
296
what causes anthelmintic resistance?
natural genetics selection pressures
297
what ways can you test for anthelmintic resistance?
faecal egg count reduction test drench test (FEC after drenching)
298
what result on faecal egg count reduction tests would suggest resistance?
a reduction of 95% or less
299
what are the SCOPS principles?
always make sure treatment is fully effective reduce dependance on anthelmintics using management and monitoring avoid bringing in resistant worms/parasites minimise the selection for resistant worms
300
what anthelmintic groups should be used for biosecurity dosing against nematodes?
4 and 5 (amino-acetonitrile and spiroindoles)
301
when is the best time of year to treat adult sheep for nematodes?
at lambing (reduces pasture contamination) but leave a portion untreated (ideally ones that will have a high egg output)
302
what can be done if haemonchus is an issue in adult sheep?
vaccinate
303
how can reliance on anthelmintics be reduced in grazing lambs?
pasture management - rotation... group ages manage concurrent disease good nutrition breed for resistance vaccinate (haemonchus)
304
what monitoring tools are available to target anthelmintic treatment?
FEC live weight gain forecast FAMANCHA - guide to measuring anaemia for haemonchus
305
how often should groups of lambs have FEC done?
every 2-4 weeks
306
what nematode is FEC not suitable for monitoring?
Nematodirus battus
307
what is the biosecurity strategy in terms of anthelmintic control for brought in sheep?
dose with monopantel/derquantel and hold off pasture for 24-48 hours for treatment to work and then turnout onto contaminated pasture
308
what is the main issues of tapeworm infections in sheep?
carcass condemnations zoonosis (Echinococcus granulosus) gid cysts in brain
309
where are Taenia hydatigena cysts found in sheep?
liver
310
where are Taenia ovis cysts found in sheep?
muscles
311
where are Taenia multiceps cysts found in sheep?
brain
312
where are Echinococcus granulosus cysts found in sheep?
liver and lungs
313
how are tapeworms for which sheep are the intermediate host treated?
praziquantel for intermediate hosts (dogs) don't allow dog to access sheep pasture or carcass
314
why is liver fluke a larger issue in sheep compared to cows?
sheep don't develop immunity
315
what is the intermediate host of liver fluke?
mud snail (Galba truncatula)
316
what are the two factors needed for liver fluke spread?
rain temperature above 10
317
what time of year does the lifecycle of liver fluke begin?
around spring
318
what is the rough lifecycle of liver fluke?
eggs shed in faeces and develops miracidium which hatches and swims to find a snail in the snail they develop into cercaria which exit the snail and develop to metacercariae and are ingested
319
is the prepatent period of liver fluke?
8-12 weeks
320
when is acute liver fluke most commonly seen?
autumn
321
when is chronic liver fluke infection most commonly seen?
winter/spring
322
what causes acute fasciolosis?
large number of metacercariae release onto pasture in late summer causing severe liver damage
323
what causes chronic fasciolosis in sheep?
ingestion of low numbers of fluke over winter/spring
324
how does liver fluke cause acute disease?
migration of immature stages through the liver causes haemorrhage
325
how does liver fluke cause chronic disease?
blood feeding adults in the bile duct leading to fibrosis and cholongitis
326
what are the clinical signs of chronic liver fluke infection?
anaemia, weight loss, submandibular oedema, enlarged liver (secondary clostridial infection - blacks disease)
327
how can fluke be diagnosed?
history/clinical signs post mortem/abattoir reports forecasts faecal egg counts (needs adults) coproantigen ELISA serum ELISA antibody
328
what drug is capable of killing any stage of fluke?
triclabendazole (extensive resistance)
329
what should be done immediately after fluke treatment?
move onto low risk pasture as drugs have no residual activity
330
what drugs can be used to kill adult stages of fluke?
albendazole, oxyclozanide, nitroxynil, closantel
331
what drug has minimal resistance and is also able to treat most stages of fluke?
nitroxynil
332
what dental diseases need to be checked for in sheep?
broken mouth and uneven molar wear (sharp/painful)
333
how many incisors do sheep erupt each year?
2 (1 year has 2 incisors... 4 years has 8 incisors)
334
what is examined externally when doing a sheep dental exam?
jaw, lymph nodes, face, halitosis, pain
335
what possible infections can sheep get in their mouth/pharynx?
Fusobacterium necrophorum - necrotic stomatitis Actinobacillosis lignerisei (facial abscess) - wooden tongue Caseous lymphadenitis
336
what is a common cause of pharyngeal trauma?
dosing gun injuries
337
what are the clinical signs of pharyngeal trauma due to dosing gun injuries?
dull, depressed, inappetent, halitosis, pain, cellulitiis
338
what does Johnes cause?
weight loss, ewe mortality, poor performance
339
what causes Johnes?
Mycobacterium avian paratuberculosis
340
how is Mycobacterium avian paratuberculosis spread?
faeces, colostrum, milk, in utero
341
what is the key risk period for infection of Johnes disease?
first 3-4 months of life
342
where does Mycobacterium avian paratuberculosis replicate in the body?
GI lymph nodes and gut
343
what age do sheep usually show signs of clinical Johnes?
3-4 years old
344
what pathology does Johnes cause?
cellular infiltration and thickening of intestines leads to malabsorption and protein losing enteropathy causing hypoalbuminaemia
345
what are the classic clinical signs of Johnes disease?
weight loss, anaemia, bottle jaw, high parasite burden
346
why is Johnes diagnosis difficult?
tests have a low sensitivity intermittently shed many animals subclinically infected
347
what is found on post mortem of sheep with johnes disease?
enlarged distal mesenteric lymph node histopathology at ileocaecal junction
348
what tests are available for johnes?
serology ELISA antibody PCR test faeces faecal culture
349
how are flock screening tests carried out for johnes?
older thinner ewes selected and PCR faecal antigen is carried out along with culture post mortems of older thinner cull ewes
350
what are the benefits of the johnes vaccine?
reduces clinical cases and excretion of bacteria (doesn't prevent infection)
351
when is the johnes vaccine given to sheep?
4-16 weeks old then repeated annually
352
what is red gut?
torsion of intestines caused by sudden introduction of lush pasture legume diet (often found dead)
353
what are the common respiratory diseases of adult sheep?
chronic suppurative pneumonia maedi visna ovine pulmonary adenomatosis laryngeal chondritis Mannhaemia haemolytica
354
what causes ovine pulmonary adenomatosis?
a retrovirus
355
how is ovine pulmonary adenomatosis spread?
aerosols, lung fluid, milk, colostrum
356
why is ovine pulmonary adenomatosis an iceberg disease?
incubation period of 2-4 years (lot of sheep effected before clinical signs are seen
357
what are the clinical signs of ovine pulmonary adenomatosis?
older thin sheep that have increased respiratory rate and exercise intolerance sudden death
358
how is ovine pulmonary adenomatosis diagnosed?
clinical signs and crackles/wheeze on lungs ultrasound lung consolidation wheelbarrow test (controversial)
359
what is done if there is a positive result on a wheelbarrow test?
euthansia
360
what is done to treat ovine pulmonary adenomatosis?
no treatment
361
what causes maedi visna?
lentivirus
362
what are the two forms of maedi visna?
maedi (chronic respiratory and mastitic disease) visna (neurological)
363
why is maedi visna an iceberg disease?
slow progressive disease (long incubation)
364
how is maedi visna transmitted?
milk, colostrum, lung discharge from mothers
365
how is maedi visna transmitted?
milk, colostrum, lung discharge from mothers
366
what are the clinical signs of respiratory maedi visna?
weight loss increased respiratory effort exercise intolerance
367
what are the clinical signs of neurological maedi visna?
weight loss abnormal gait, ataxia, paralysis circling, head tremor... (much less common)
368
how is maedi visna controlled?
test and cull (good test available)
369
what is the main bacteria causing chronic suppurative pneumonia?
Trueperella pyogenes
370
what are the clinical signs of chronic suppurative pneumonia?
chronic weight loss increased respiratory effort cough nasal discharge pyrexia
371
what breeds is laryngeal chondritis most commonly seen in?
beltex and texels (rams most commonly)
372
what is the pathology seen in laryngeal chondritis?
narrow/swollen larynx, nodules, abscesses
373
why are texels so predisposed to laryngeal chondritis?
shorter larynx and vocal cords are closer and narrows to a funnel
374
how can laryngeal chondritis be treated?
corticosteroids and broad spectrum antibiotics (penicillin) temporary tracheostomy (may never recover or reoccur)
375
what is pink eye also known as?
ovine infectious keratoconjunctivitis
376
what causes ovine infectious keratoconjuntivitis?
Mycoplasma conjunctivae
377
how can ovine infectious keratoconjuntivitis be treated?
isolate sheep and remove risk factors LA oxytetracycline or macrolide
378
what is anterior uveitis also known as?
silage eye
379
what causes anterior uveitis?
Listeria monocytogenes
380
what are the clinical signs of silage eye?
blepharospasm cloudy cornea swollen folded iris material in front of eye chamber
381
how is silage eye treated?
subconjunctival oxytetracycline and dexamethasone systemic penicillin
382
what are the main diseases of the foot associated with lameness?
footrot (scald, interdigital dermatitis) CODD - contagious ovine digital dermatitis white line toe granuloma foot abscess
383
how does the foot appear in cases of benign footrot?
interdigital skin inflamed and discharging but there is no separation of the horn (cheesy smell)
384
what are some risk factors for benign footrot?
moisture (main one) - both indoors and outdoors anything causing damage to the skin (frost, thistles...) warmth genetic susceptibility
385
what is the main bacteria associated with benign footrot?
Dichelobacter nodosus
386
what is a secondary bacteria often isolated from benign footrot cases?
Fusobacterium necrophorum
387
what is the classic group of sheep that benign footrot is seen in?
growing lambs
388
what is virulent footrot?
interdigital dermatitis (benign footrot) that progresses to under running of the sole of hoof horn starting medially and progressing laterally
389
how does virulent footrot present?
grey necrotic pungent smelling horn with interdigital dermatitis
390
how long does Dichelobacter nodosus survive in the environment?
7 days
391
is Dichelobacter a opportunistic or obligate pathogen of sheep feet?
opportunistic (found in normal feet)
392
how is interdigital dermatitis (benign footrot) treated?
topical - oxytetracycline, footbath (formalin, zinc sulphate, disinfectant)
393
what factors need to be controlled to ensure footbathing sheep is effective?
appropriate exposure (2 minutes) clean feet prior to bathing correct concentration and depth allow feet to dry afterwards (stand on concrete)
394
how is virulent footrot treated?
isolate lame sheep LA oxytetracylcine or amoxicillin macrolides - tilmicosin, gamithromycin do not trim (unless really necissary)
395
how is virulent footrot treated?
isolate lame sheep LA oxytetracylcine or amoxicillin macrolides - tilmicosin, gamithromycin do not trim (unless really necessary)
396
why shouldn't you trim cases of footrot as a treatment?
delays healing and aids spreading to other sheep
397
how bad is the lameness caused by CODD?
very severe
398
how do the feet of sheep with CODD appear?
ulcerative/proliferative lesion that starts at coronary band progressive underrunning of hoof wall hoof sloughs off
399
what is a grade 1 CODD lesion?
just a coronary band lesion
400
what is a grade 2 CODD lesion?
coronary band lesion with less than 50% of the hoof capsule separated
401
what is a grade 3 CODD lesion?
50-100% of hoof capsule has sloughed off
402
is a grade 4CODD lesion?
a healing but still active lesion (still inflamed...)
403
how extensive is the damage caused by CODD?
very severe inflammation and lameness that can even attack the bone
404
what bacteria is associated with CODD?
Treponeme (same as digital dermatitis of cattle) Dichelobacter nodosus and Fusobacterium necrophorum also found
405
what are the risk factors for CODD?
late summer to early autumn trend large flock size moist, lush, lowland pasture
406
how is CODD treated?
isolate and treat rapidly LA amoxicillin macrolides - tilmicosin, gamithromycin...
407
what is the five point plan for lameness control/management?
vaccinate twice yearly with footvax treat lame sheep quickly biosecurity environmental challenge cull chronically lame sheep
408
why is footvax (footrot vaccine) useful?
treats and prevents footrot (and possibly CODD)
409
what is footvax and vaccine for?
footrot (Dichelobacter nodosus strains)
410
when should footvax not be used?
prior to shearing or lambing (oily injection site lumps and abortion risk) if sheep have had recent moxidectin treatment
411
what is the vaccination protocol for footvax?
initial 2 doses 4-6 weeks apart then 6 monthly boosters
412
what is the culling advice for lame sheep?
if they have had 2-3 cases of lameness they should be culled
413
why should chronically lame sheep be culled?
harbour infection in the feet (spreads to flock) production - less likely to get pregant and rear lambs welfare gets rid of genetically susceptible sheep
414
what is white line disease also known as?
shelly hoof
415
what is white line disease?
separation at the white line of the hoof
416
how lame are sheep with white line disease?
only lame if sensitive tissue is effected (impaction wit stones/mud or abscesses)
417
how is white line disease treated if there is no infection present?
leave and carefully trim area of separation
418
how is white line disease treated if there is no infection?
leave alone and carefully trim area of separation
419
how is white line disease treated if an abscess is present?
trim to release pus but avoid sensitive tissue
420
what is the usual cause of toe granulomas in sheep?
over trimming
421
how are toe granulomas treated?
local anaesthetic then cut back granulation tissue and cauterise (ensure granuloma removed)
422
how do sheep with laminitis present?
lame on all four limbs with them all tucked under abdomen digits warm to the touch
423
what is the main cause of laminitis in sheep?
high energy or cereal based feed
424
what causes pedal joint abscess?
extension of interdigital infection into the distal interphalangeal joint structure often seen secondary to white line abscesses
425
what are the clinical signs of pedal joint abscess?
severe lameness foot is swollen widened interdigital space purulent discharging sinus tract
426
how can pedal joint abscesses be treated?
flush and provide antibiotics digit amputation
427
what is strawberry footrot?
proliferative scab lesions on distal limb
428
what is usually the cause of strawberry footrot?
orf and dermatophilus
429
how is strawberry footrot treated?
move to a dry field/housing and give systematic/topical antibiotics
430
what is used to diagnose white muscle disease?
raised glutathione peroxidase in blood
431
what are some infectious causes of arthritis?
neonatal infections (Strep dysgalactiae) tick pyaemia Erysipelothrix rhusiopathie
432
what is the most common joint for degenerative joint disease to occur in?
elbow
433
what areas can you narrow down a neurological condition to?
cerebrum cerebellum vestibular system brain stem spinal cord
434
what are the clinical signs of diffuse damage to the cerebrum?
altered mental state - depressed, hyperexcitable, disorientated blindness seizures opisthotonus - recumbent, extended forelimbs and flexed hindlimbs
435
what is opisthotonus?
extended forelimbs and flexed hindlimbs (recumbent)
436
what are some causes of diffuse cerebrum damage?
bacterial meningitis cerebrocortical necrosis pregnancy toxaemia
437
what are the clinical signs of local cerebral damage?
contralateral blindness circling proprioceptive deficits
438
what are some examples of causes of local cerebral damage?
GID cyst brain abscess trauma
439
what are the clinical signs of cerebellar disease?
altered head carriage ataxia(not weakness) dysmetria - high stepping intention tremors
440
what are some possible causes of cerebellar damage?
congenital - cerebellar hypoplasia border disease (hairy shakers) abscesses
441
what are the clinical signs of vestibular system damage?
head tilt to affected side loss of balance circling falling/rolling to one side spontaneous nystagmus
442
what can cause vestibular system damage?
middle earn infection
443
what are the clinical signs of brainstem damage?
depression cranial nerve deficits ipsilateral hemiparesis
444
what is the main condition that causes brainstem damage?
listeriosis
445
what are some causes of spinal cord lesions?
spinal abscess (joint ill, tick pyaemia...) wobblers syndrome trauma congenital abnormality
446
what needs to be determined when examining lambs with neurological conditions?
congenital or acquired
447
what are some common neurological problem in young lambs?
border disease congenital swayback drunken lamb syndrome bacterial meningitis tetanus
448
how are spinal abscesses of young lambs treated?
dexamethasone (1ml/kg) penicillin - 5 days
449
what transmits louping ill?
ticks
450
what are the clinical signs of louping ill?
head pressing trembling/tremors nystagmus lip twitching louping gait (front and back legs move together)
451
what are common neurological conditions of older lambs?
CCN - cerebrocortical necrosis listeriosis gid trauma louping ill (if in tick area)
452
what causes CCN?
vitamin B1 (thiamine) deficiency
453
what are the clinical signs of CCN?
dull, disorientated, blind, tremors, recumbency, opisthotonus, convulsions
454
what is the classic history of a lamb with CCN?
fats growing lamb with a diet change (weaned and ate a lot of concentrates)
455
how is CCN treated?
vitamin B1 - every 12 hours for 3 days NSAIDs
456
what is listeriosis associated with?
feeding poorly preserved silage with soil contamination (mouldy)
457
what are the clinical signs of listeriosis?
anorexia, depression unilateral hemiparesis trigeminal nerve paralysis (salivation and food impaction) facial nerve paralysis
458
how is listeriosis treated?
benzylpenicillin or procaine penicillin dexamethasone
459
what causes gid?
Taenia multiceps
460
what are the clinical signs of gid?
circling unilateral blindness head tilt skull softening
461
how is gid controlled in sheep?
worm dogs every 6 weeks with praziquantel keep dogs away from sheep carcass
462
how is pregnancy toxaemia diagnosed?
BOHB levels in blood (mainly clinical signs)
463
how is scrapie transmitted?
mother to offspring prenatally, colostrum and milk birthing fluids and placenta
464
what are the clinical signs of scrapie?
2-5 years old skin issues weight loss altered mental state (excitable), trembling, high step ataxia, recumbency
465
what is orf also known as?
contagious pustular dermatitis
466
what type of virus causes orf?
parapox
467
what type of lesions are seen with orf?
painful vesicular/proliferative scabby lesions around mouth, head and teats
468
what is the duration of orf?
4-6 weeks
469
how if orf transmitted?
contact - usually required damaged skin
470
what is required for orf to survive the winter?
a carrier animal (doesn't overwinter)
471
how is orf controlled?
isolate clinical cases thorough cleaning/biosecurity no treatment (secondary infection antibiotics) vaccine
472
when is the orf vaccine used?
only on infected farms
473
why does orf need to be confirmed on the farm before vaccination?
it is a live vaccine so will infect the sheep if uninfected
474
how is the orf vaccine applied?
scratching skin (these scabs will be infection so put in armpit where they won't likely touch other animals/farmer)
475
what are the main mites effecting sheep?
Psoroptes ovis Chorioptes bovis Trombiculus
476
what is the main louse effecting sheep?
Bovicola ovis
477
what tick species effect sheep?
Ixodes ricinus Haemaphyllis punctuata
478
what does Psorptes ovis cause?
sheep scab
479
what type of reaction causes sheep scab?
allergic reaction to the mites
480
what are the clinical signs of sheep scab?
wool loss, ragged moist yellow fleece, kicking/rubbing, weight loss
481
where should you sample for Psorptes ovis?
edge of lesion (on the worst sheep)
482
how is sheep scab diagnosed?
wool pluck and skin scrape ELISA
483
what are the morphological features of Psorptes ovis?
pointed mouth parts and bell shaped suckers
484
how is sheep scab treated?
treat all sheep with organophosphate dip (diazinon)
485
why is reinfection of sheep scab seen after treatment?
mites survive 19 days off the host
486
what are some possible treatment options for sheep scab?
organophosphates (diazanon) macrocylic lactones (ivermectin, doramectin...)
487
how can sheep scab be prevented?
biosecurity (treat all bought in sheep) double fence neighbouring stock cleaning equipment
488
where is chorioptes bovis found on the sheep?
ventral abdomen and scrotum
489
what is used to treat Chorioptes bovis?
organophosphates
490
what type of louse is Bovicola ovis?
chewing
491
what can be done to treat lice?
shearing synthetic pyrethroids and organophosphates
492
when does fly strike occur?
May to October
493
what flies cause fly strike?
Lucilia sericata
494
what is the pathogenesis of Lucilia sericata that causes fly strike?
lay eggs that hatch in about 12 hours that release proteolytic enzymes and hook onto the skin using their mouth causing damage the smell also attracts other flies causing rapid multiplication
495
what is the most common risk factor for fly strike?
faecal soiling (lush grass causing SARA) in warm wet weather
496
what is used to treat fly strike?
synthetic pyrethroids (kills maggots) clean and trim house address other risk factors
497
how can fly strike be prevented/controlled?
tail docking dagging shearning worm control prophylactic treatment
498
what products can be used for fly strike control/prevention?
organophosphate dips pyrethroid pour ons insect growth regulators
499
what is the headfly called?
Hydrotea irritans
500
when does Hydrotea irritans cause the most problems?
when a flock has eye disease - they feed on ocular discharge spreading the disease through the flock (mainly a nuisance fly)
501
what causes ovine sweet itch?
hypersensitivity to midges (mainly thin places of skin)
502
what causes peri-orbital eczema?
Staphylococcus aureus
503
how is peri-orbital eczema spread?
spread through contact (feed troughs...)
504
how is peri-orbital eczema treated?
penicillin
505
what causes lumpy wool?
Dermatophilus congolensi
506
what are the risk factors for Dermatophillus congolensi causing lumpy wool?
wet conditions after shearing thin wooled breeds
507
what are the clinical signs of Dermatophillus congolensi?
crusty lesions with wool loss pruritis (fly strike)
508
how is Dermatophillus congolensi treated?
penicillin
509
what causes caseous lymphadenitis?
Corynebacterium pseudotuberculosis
510
what is the mopst common lymph node to find caseous lymphadenitis?
parotid
511
how is caseous lymphadenitis spread?
close contact fomites (shearing...) respiratory pathway
512
what conditions is needed for a tick to feed?
above 85% humidity and 7 degrees
513
what are the main UK tick born diseases?
tick borne fever tick pyaemia louping ill (neurological) Q fever (babesiosis in cattle and lyme disease not in sheep)
514
what causes tick borne fever?
Anaplasma phagocytophilia
515
what is the effect of tick borne fever?
profound immunosuppression (gateway for other diseases) fever - abortion and infertility (rams)
516
is there any immunity to tick borne fever?
yes - with age
517
what can be done to control louping ill in areas where it is common?
vaccinate
518
what are the four most common plant poisonings of sheep?
yew acorn laurel rhododendron
519
what causes pneumonic pasturellosis?
Mannheimia haemolytica
520
what is used to treat pneumonic pasteurellosis?
LA oxytetracycline or amoxicillin
521
how can pasturellosis be controlled?
vaccination (reduces severity) control risk factors
522
what causes systemic pasteurllosis?
Biebersteina trahalosi
523
what age lambs are generally effected by systemic pasturellosis?
6-10 months of age
524
what are risk factors for systemic pasturellosis?
change of grazing change of weather concurrent disease stress (handling...)
525
what is used to treat systemic pasturellosis?
LA oxytetracycline
526
wherre are clostridial bacteria found?
farm environment - soil, guts, surfaces...
527
what are clostridial disease risk factors?
poor hygiene endoparasites wounds/trauma/injury changes to diet
528
what causes lamb dysentery?
Clostridium perfingens type B
529
what aged lambs are effected by lamb dysentery?
under 3 weeks old
530
what are the main clinical signs of lamb dysentery?
water profuse diarrhoea with blood in severe abdominal pain
531
what are the main findings on post mortem of a lamb with Clostridium perfingens type B?
haemorrhagic enteritis blood stained fluid in abdominal cavity and pericardium
532
what causes pulpy kidney?
Clostridium perfingens type D
533
what age lambs in pulpy kidney seen in?
4-10 weeks old or 6 months plus
534
what is a major risk factor for pulpy kidney disease?
high concentrate diet (creep fed)
535
what does Clostridium sordelli cause?
abomasitis and toxaemia
536
what sheep is Clostridium sordelli seen to cause disease?
intensively reared concentrate fed 4-10 week old lambs
537
what causes tetanus?
Clostrium tetani
538
what are the clinical signs of tetanus?
stiffness, opisthotonus, recumbency, death
539
what causes braxy?
Clostridium septicum
540
what are the risk factors for braxy?
autumn/winter - frosty root crops
541
what causes blacks disease?
Clostridium novyi type B
542
what is blacks disease often secondary to?
fluke
543
what are the findings of blacks disease on post mortem?
blood stained fluid in body cavity necrotic liver fluke
544
when should a vaccination course for clostridial disease be started?
3 weeks old (given to ewe 4 weeks before lambing)
545
what notifiable diseases have been seen in UK sheep?
anthrax blue tongue contagious agalactiae foot and mouth disease scrapie
546
what causes anthrax?
Bacillus anthracis
547
what are the main clinical signs of foot and mouth in sheep?
lameness, blisters on tongue
548
what are the clinical signs of blue tongue?
fever, mouth ulcers, swelling of head/neck and lameness
549
what are the clinical signs of contagious agalactia?
mastitis, arthritis, keratoconjunctivitis, abortion
550
what causes contagious agalactia?
Mycoplasma agalactiae
551
what are the advantages of a flock health plan?
required for farm assurance schemes up to date advise for farmer improves client relationships improved welfare standards
552
what are good times of year to do a flock health plan?
summer (weaning) - pre-tupping pre-lambing (winter)
553
what are some KPIs for sheep flocks?
scanning % lambing % (born alive, weaned, sold) ewe mortality replacement rate ewe:tup ratio lambing mortality (scanning, birth...)
554
how are KPIs used?
compare to industry standard compare to farms own goals
555
what are the main principles of a biosecurity plan on a flock health review?
impossible to have zero risk business/farm aims discussed identify the disease the flocks at risk of getting identify current flock disease status assess risk of disease introduction quantify the risk devise control measures