Visual Flashcards

(49 cards)

1
Q

What is the range of the visual field of each eye? Both eyes?

A

The visual field of each eye extends from 90o laterally to 60o medially, totaling 150o.

All together the visual field covered by two eyes is 180o.

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2
Q

Portion of the field of view that can be seen by both eyes together?

A

The portion of the visual field that can be seen by both eyes together is the binocular segment and covers 120o.

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3
Q

Lateral most FOV of eye can be seen by?

A

The lateral most 30o that can be seen by only the ipsilateral eye is the monocular segment.

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4
Q

How is the visual field segmented?

A

Portions of the visual field are named with reference to the vertical or horizontal meridians (the line dividing right and left or upper and lower halves of the visual field).

We also refer to nasal and temporal segments of the visual field.

Thus, the visual field of each eye is divided into quadrants:
Upper nasal
Upper temporal
Lower nasal
Lower temporal
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5
Q

Images on the retina are?

A

Inverted and reversed

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6
Q

Describe how the half of FOV is transmitted to the visual cortex

A

From each eye the representation of one half of the visual field (bisected by the vertical
meridian) is projected to the contralateral (opposite) hemisphere

Therefore, the representation of the right visual field from both the left and right eyes is
passed on to the left LGN and left cortical hemisphere

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7
Q

The fovea of the eye represents? Packed with what type of cells?

A

The fovea of the eye represents central vision.

When you think foveal or central vision,
think of the center of your eyes’ focus when you read.

Central vision subtends less than
1° of the visual field and is densely packed with cone photoreceptors while containing
no rods

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8
Q

What gives us high acuity vision (cell type)? Visual cortex?

A

Compared to rods, cones provide much greater visual acuity.

A great emphasis is placed on the processing of foveal vision throughout the visual system; this is exemplified by the proportionally greater expanse of cortex devoted to central vision.

This so called cortical magnification is quite prominent in primary visual cortex

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9
Q

Describe the cell structure of the retina

A

Rods and cones are at the back of the retina and respond to the light

They synapse on bipolar
cells which in turn synapse onto retinal ganglion cells.

Amacrine and horizontal cells
are interneurons that provide excitatory and inhibitory synapses across several bipolar
and ganglion cells.

These lateral connections form concentric, center-surround receptive fields

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10
Q

Two major retinal ganglion cell types project to?

A

Lateral geniculate nucleus

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11
Q

What causes the blind spot? Where is it?

A

The optic disc

On the nasal side of each eyeball retina

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12
Q

Where is the fovea? Where does it focus?

A

Temporal side of each eyeball retina

Central fixation point

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13
Q

Where is the blind spot for each eye?

A

Left eye = left FOV

Right eye = right FOV

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14
Q

Two types of retinal ganglion cells? Functions?

A

Large parasol retinal ganglion cells = motion and spatial analysis, project to the magnocellular layers of the LGN

Small midget retinal ganglion cells = important for form and color, project to the parvocellular layers of the LGN

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15
Q

Describe the laminar layers of the LGN

A

Six main layers of LGN

4 parvocellular layers, dorsal, Layers 3-6
2 magnocellular layers, ventral, Layers 1-2

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16
Q

Function of magnocellular and parvocellular layers of the LGN?

A

magnocellular layers of the LGN = motion and spatial analysis

parvocellular layers of the LGN = important for form and color

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17
Q

Where do parvo and magno layers of the LGN project to?

A

The 4C layer of Primary visual cortex (V1) through optic radiations

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18
Q

Describe the sublayers of 4C layer of V1

A
Parvo = 4C-beta
Magno = 4C-alpha
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19
Q

Describe path of magno and parvo fibers past V1

A

Tend to remain segregated in V1

Give rise to dorsal and ventral streams in subsequent visual areas such as V4, MT

Dorsal = parvo
Ventral = Magno
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20
Q

Describe the optic radiations

A

Projections from LGN travel through optic radiations to get to V1

Axons fibers fan out over (lateral to) the temporal horn of the lateral ventricle then back toward primary visual cortex on the occipital pole of neocortex

21
Q

Describe lesion of optic radiation

A

Ipsilateral and contralateral eye fibers from the different LGN layers are mixed, therefore lesions
usually cause homonymous (both eyes) defects affecting the contralateral visual field

E.g. lesion in right optic tract leads to left FOV loss from both eyes

22
Q

What is Meyer’s loop? Where does it begin? End?

A

Fibers of the inferior optic radiations arc through the temporal lobe forming Meyer’s loop and terminate in the lower bank of the calcarine sulcus

23
Q

What portion of vision does Meyer’s loop provide?

A

Meyer’s loop fibers represent the superior (upper) visual field quadrant

24
Q

Describe lesion to Meyer’s loop. What causes and what happens?

A

Temporal lobe lesions, such as MCA inferior division infarcts, can cause
‘contralateral superior quadrantanopia’

Means that superior quadrant of vision is lost on FOV contralateral to lesion

25
Describe sided-ness of lesions to path of information from retina to V1
Happens before chiasm = monocular and ipsilateral Happens at or after chiasm = homonymous and contralateral
26
Describe the path of the upper optic radiations (beginning and end). What do these fibers represent?
Fibers of the upper (superior) optic radiations beneath the parietal lobe and terminate in the upper bank of the calcarine sulcus These upper optic radiations represent the inferior (lower) visual field quadrant
27
Lesions to the entire optic radiation close to the visual cortex will result in?
Homonymous hemianopia Meaning loss of contralateral FOV from both eyes
28
Damages to the upper and lower banks of the calcarine sulcus in V1 lead to?
Same as upper and lower optic radiation fibers ``` Upper = loss of lower contralateral quadrant Lower = loss of upper contralateral quadrant ```
29
Lesion to superior optic radiation can cause? Caused by?
Lesions involving the parietal lobe, such as MCA superior division infarcts, can cause ‘contralateral inferior quadrantanopia’ Means inferior quadrant of vision is lost on FOV contralateral to lesion
30
Describe the fibers from each eye once in the optic radiations
While fibers from the two eyes are intermingled in the optic radiations, the inputs from the two eyes remain segregated into adjacent columns in the geniculate input layer 4C.
31
Describe the term ocular dominance column
A single neuron in an ocular dominance | column for the right eye responds to visual stimulation of the right eye only.
32
Describe what happens to fibers from each eye once moving past 4C in V1
Beyond layer 4C, in layer 2/3 for example, the inputs from the two eyes mix. That is, a single neuron in layer 2/3 will respond to visual stimulation of either eye.
33
Describe the connection between ocular dominance columns and lazy eye
Failure of one eye to establish cortical territory in the form ocular dominance columns is linked to amblyopia, lazy eye.
34
Describe how monocular visual field loss occurs
(partial or complete) can only result from damage to the visual pathway prior to the optic chiasm At the level of the chiasm and beyond information from the two eyes is mixed (though not in the same neuron until beyond layer 4C of V1).
35
What is amaurosis fugax?
phrase used to describe sudden, transient (~10min) monocular visual loss
36
What causes amaurosis fugax?
It results from transient occlusion of the retinal artery(ophthalmic artery) in one eye caused by emboli. Occlusion of either the superior or inferior branch of the ophthalmic artery results in loss of the inferior or superior visual field, respectively, of the affected eye
37
What is bitemporal hemianopia? Results from?
is loss of the temporal visual field of each eye, resembling ’tunnel vision’. It can only result from damage at the level of the optic chiasm, and is common with pituitary tumors.
38
Retrochiasmal lesions are? What do they cause?
Include lesions of the optic tracts, LGN, optic radiations, or visual cortex ``` Generally cause ‘homonymous’ visual field defects (the same regions of the visual fields for both eyes are affected) ```
39
What is macular sparing?
Macular sparing occurs because of the relatively large representation of the foveal representation throughout the visual pathway. Partial lesions therefore may spare this larger region. Exactly which parts of the visual field are affected and whether one or both eyes are involved tell us where in the visual pathway the lesions have occurred.
40
Describe modes to prevent binocular blindness to a particular vision field.
the segregation of eye inputs can help prevent binocular blindness to a particular visual field. In addition, alternative pathways, for example through the superior colliculus and pulvinar can provide crude visual capabilities, especially related to motion and spatial vision (dorsal stream).
41
Describe the layers of the LGN. Which eye goes to which layer?
Layer 1 is ventral; 6 is dorsal ``` Ipsilateral = 2,3,5 Contralateral = 1,4,6 ```
42
What lobe of brain is important for specific shape recognition?
The ventral temporal cortex
43
Describe the heirarchy from simple to complex shape processing
From LGN (small dots) to V1 (orientation, disparity, and some color) to V4 (color, basic 2D, 3D, curvature) to VTC (complex features and objects)
44
Primary visual cortex is also known as what Broca's area? What about visual association cortex?
Primary = 17 Association = 18
45
V5 is also known as?
MT Higher order visual association cortex
46
Describe magnocellular cells
``` Large cell body •Fat axons •Get input from many photoreceptors •Rapid conduction velocity •Transient Firing ``` Motion information
47
Describe parvocellular cells
* Small cell body * Thin axons * Pool input from fewer neurons * Sustained firing Detail and color
48
Describe visual damage to higher visual areas
Lesions to V2 and higher visual areas result in more functionally specific lesions: the higher the visual area the more general vision is retained and the more specialized the deficit becomes. For example lesions in IT cortex can result in an inability to recognize faces, but one can still see that there is a face.
49
What is visual agnosia?
the inability to recognize particular objects due to lesions of higher order ventral stream areas Example is prosopagnosia