Week 2 for Quiz 2 Flashcards

1
Q

result of overinflation of the lungs, which increases the anteroposterior diameter of the thorax.

A

Barrel Chest

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2
Q

Barrel chest occurs in patients with?

A

aging, emphysema and COPD.

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3
Q

depression in the lower portion of the sternum. This may compress the heart and great vessels, resulting in murmurs

A

funnel chest (pectus excavatum)

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4
Q

funnel chest (pectus excavatum) occurs in patients with?

A

rickets or Marfan syndrome.

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5
Q

result of the anterior displacement of the sternum, which also increases the anteroposterior diameter.
rickets, Marf

A

PIGEON CHEST (pectus CARINATUM)

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6
Q

PIGEON CHEST (pectus CARINATUM) occurs in patients with?

A

rickets, Marfan syndrome, severe kyphoscoliosis

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7
Q

characterized by elevation of the scapula and a corresponding S-shaped spine. This deformity limits lung expansion within the thorax.

A

kyphoscoliosis

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8
Q

kyphoscoliosis occurs in patients with?

A

osteoporosis, thoracic skeletal

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9
Q

Increased responsiveness of lower airways to multiple stimuli. ;Episodic and with reversible obstruction.

A

asthma

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10
Q

ominous signs of asthma

A

Fatigue,
Pulsus paradoxus,
Diaphoresis,
Inaudible breath sounds with diminished wheezing,
Inability to maintain recumbency,
And cyanosis

FPDIIC

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11
Q

what do you call the perfusion pressure occurs in asthmatic patient?

A

Pulsus Paradoxus

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12
Q

medical management: of asthma

A

Albuterol (Ventolin) –
Adrenergic
Theophylline –
Hydration

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13
Q

what happens in a status asthmaticus

A

Severe bronchospasm and does not relieve within 30-60 mins.

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14
Q

Anticholinergics used in asthma

A

Ipratropium bromide (Atrovent, Combivent),

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15
Q

Characterized by airflow obstruction due to chronic bronchitis or emphysema.

A

chronic obstructive pulmonary disease (copd)

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16
Q

main cause of COPD

A

Smoking

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17
Q

According to the American Lung Association, smoking cigarettes causes ________ of all COPD cases

A

85-90%

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18
Q

Umbrella term that include: COPD

A

CRES
Chronic bronchitis
Refractory (irreversible) asthma
Emphysema
Severe bronchiectasis

19
Q

Predisposing and Precipitating factors
of COPD

A

(1) Oxidative stress;
(2) Cytokine release due to inflammation;
(3) impaired activity of anti protease enzymes

20
Q

Triad Symptoms of COPD

A

Cough
Sputum production
Dyspnea on exertion

21
Q

method of assessing lung function by measuring the volume of air that the patient can expel from the lungs after a maximal inspiration.

A

Spirometry

22
Q

Anticholinergic agents for COPD

A

Ipratropium bromide (Atrovent)
Oxitropium bromide (Oxivent)

23
Q

Excessive mucus secretion within the airways and recurrent cough usually related to smoking, pollution, and infection.

A

chronic bronchitis “blue bloaters”

24
Q

Destruction of alveoli, narrowing of small airways and trapping of air resulting in loss of lung elasticity.

A

emphysema “Pink puffers”

25
Q

Alveoli of a patient with emphysema look like?

A

Cauliflower instead of grapes

26
Q

To limit blood flow to hypoxic alveoli, low oxygen leads to pulmonary vasoconstriction, diverting blood to the better-ventilated alveoli

A

hypoxic pulmonary vasoconstriction

27
Q

pathophysiology of cor pulmonale

A

Changes in vascular mediators:
high Endothelin-1
Low Endothelial nitric oxide synthase production: nitric oxide
Low Prostacyclin (vasodilator)

28
Q

Main pathophysiology of Cor Pulmonale

A

hypoxic pulmonary vasoconstriction
pulmonary vascular remodeling

29
Q

Irreversible dilatation of the cartilage containing airways.

A

bronchiectasis

30
Q

Long-term condition where the airways of the lungs become widened, leading to a build-up of excess mucus that can make the lungs more vulnerable to infection.

A

bronchiectasis

31
Q

Infectious Causes: of bronchiectasis

A

Adenovirus and Influenza virus
Staphylococcus aureus, Klebsiella, and Anaerobes
Bordetella pertussis
HIV infection
Tuberculosis

32
Q

Main infectious cause of bronchiectasis

A

Adenovirus and Influenza virus

33
Q

Texture of sputum of patient with bronchiectasis

A

Copious foul-smelling sputum that separates into three layers in a cup.

34
Q

Parts of saliva of patient with bronchiectasis (3 layers)

A

Foam - Frothy partly saliva
Liquid- Thin sero-mucus liquid
Pus/sediment- pus, necrotic, cell debris

35
Q

Treatment #1 for bronchiectasis

A

4 Goals:
Eliminate cause
Improve tracheo bronchial clearance
Control infection
Reverse airflow obstruction

36
Q

Treatment #2 for bronchiectasis

A

Immunoglobulin
Antituberculous drugs
Corticosteroids (ABPA)
Remove aspirated material
Chest physical therapy
Mucolytics
Bronchodilators

37
Q

Treatment #3 for bronchiectasis

A

Lung transplant
Initial empiric
Antibiotics

38
Q

An acute inflammation on lung parenchyma.

A

pneumonia

39
Q

pneumonia with onset in the community or the first 2 days of hospitalization.

A

Cap (community acquired pneumonia)

40
Q

Common Causative Agents: for pneumonia

A

Streptococcus pneumonia (40%) MOST
Klebsiella (20%)
Haemophilus influenzae
Legionella, mycoplasma, Chlamydia

41
Q

3rd most common cause of morbidity among nosocomial infections.

A

Hap (hospital acquired pneumonia) healthcare-acquired

42
Q

A lung infection that develops in a person who is on a ventilator.

A

Vap (ventilator acquired pneumonia)

43
Q

Vaccination for pneumonia

A

Haemophilus influenzae
Streptococcus pneumoniae
Pertussis, varicella, and measles

44
Q

Sputum collection: for TB considerations

A

Best obtained on three consecutive mornings.
Clean and thoroughly rinse the mouth with water.
Breathe deeply 3 times.
After the third breath, cough hard and try to bring up sputum from deep in the lungs