Week 2 for Quiz 2 Flashcards

1
Q

result of overinflation of the lungs, which increases the anteroposterior diameter of the thorax.

A

Barrel Chest

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2
Q

Barrel chest occurs in patients with?

A

aging, emphysema and COPD.

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3
Q

depression in the lower portion of the sternum. This may compress the heart and great vessels, resulting in murmurs

A

funnel chest (pectus excavatum)

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4
Q

funnel chest (pectus excavatum) occurs in patients with?

A

rickets or Marfan syndrome.

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5
Q

result of the anterior displacement of the sternum, which also increases the anteroposterior diameter.
rickets, Marf

A

PIGEON CHEST (pectus CARINATUM)

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6
Q

PIGEON CHEST (pectus CARINATUM) occurs in patients with?

A

rickets, Marfan syndrome, severe kyphoscoliosis

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7
Q

characterized by elevation of the scapula and a corresponding S-shaped spine. This deformity limits lung expansion within the thorax.

A

kyphoscoliosis

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8
Q

kyphoscoliosis occurs in patients with?

A

osteoporosis, thoracic skeletal

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9
Q

Increased responsiveness of lower airways to multiple stimuli. ;Episodic and with reversible obstruction.

A

asthma

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10
Q

ominous signs of asthma

A

Fatigue,
Pulsus paradoxus,
Diaphoresis,
Inaudible breath sounds with diminished wheezing,
Inability to maintain recumbency,
And cyanosis

FPDIIC

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11
Q

what do you call the perfusion pressure occurs in asthmatic patient?

A

Pulsus Paradoxus

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12
Q

medical management: of asthma

A

Albuterol (Ventolin) –
Adrenergic
Theophylline –
Hydration

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13
Q

what happens in a status asthmaticus

A

Severe bronchospasm and does not relieve within 30-60 mins.

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14
Q

Anticholinergics used in asthma

A

Ipratropium bromide (Atrovent, Combivent),

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15
Q

Characterized by airflow obstruction due to chronic bronchitis or emphysema.

A

chronic obstructive pulmonary disease (copd)

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16
Q

main cause of COPD

A

Smoking

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17
Q

According to the American Lung Association, smoking cigarettes causes ________ of all COPD cases

A

85-90%

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18
Q

Umbrella term that include: COPD

A

CRES
Chronic bronchitis
Refractory (irreversible) asthma
Emphysema
Severe bronchiectasis

19
Q

Predisposing and Precipitating factors
of COPD

A

(1) Oxidative stress;
(2) Cytokine release due to inflammation;
(3) impaired activity of anti protease enzymes

20
Q

Triad Symptoms of COPD

A

Cough
Sputum production
Dyspnea on exertion

21
Q

method of assessing lung function by measuring the volume of air that the patient can expel from the lungs after a maximal inspiration.

A

Spirometry

22
Q

Anticholinergic agents for COPD

A

Ipratropium bromide (Atrovent)
Oxitropium bromide (Oxivent)

23
Q

Excessive mucus secretion within the airways and recurrent cough usually related to smoking, pollution, and infection.

A

chronic bronchitis “blue bloaters”

24
Q

Destruction of alveoli, narrowing of small airways and trapping of air resulting in loss of lung elasticity.

A

emphysema “Pink puffers”

25
Alveoli of a patient with emphysema look like?
Cauliflower instead of grapes
26
To limit blood flow to hypoxic alveoli, low oxygen leads to pulmonary vasoconstriction, diverting blood to the better-ventilated alveoli
hypoxic pulmonary vasoconstriction
27
pathophysiology of cor pulmonale
Changes in vascular mediators: high Endothelin-1 Low Endothelial nitric oxide synthase production: nitric oxide Low Prostacyclin (vasodilator)
28
Main pathophysiology of Cor Pulmonale
hypoxic pulmonary vasoconstriction pulmonary vascular remodeling
29
Irreversible dilatation of the cartilage containing airways.
bronchiectasis
30
Long-term condition where the airways of the lungs become widened, leading to a build-up of excess mucus that can make the lungs more vulnerable to infection.
bronchiectasis
31
Infectious Causes: of bronchiectasis
Adenovirus and Influenza virus Staphylococcus aureus, Klebsiella, and Anaerobes Bordetella pertussis HIV infection Tuberculosis
32
Main infectious cause of bronchiectasis
Adenovirus and Influenza virus
33
Texture of sputum of patient with bronchiectasis
Copious foul-smelling sputum that separates into three layers in a cup.
34
Parts of saliva of patient with bronchiectasis (3 layers)
Foam - Frothy partly saliva Liquid- Thin sero-mucus liquid Pus/sediment- pus, necrotic, cell debris
35
Treatment #1 for bronchiectasis
4 Goals: Eliminate cause Improve tracheo bronchial clearance Control infection Reverse airflow obstruction
36
Treatment #2 for bronchiectasis
Immunoglobulin Antituberculous drugs Corticosteroids (ABPA) Remove aspirated material Chest physical therapy Mucolytics Bronchodilators
37
Treatment #3 for bronchiectasis
Lung transplant Initial empiric Antibiotics
38
An acute inflammation on lung parenchyma.
pneumonia
39
pneumonia with onset in the community or the first 2 days of hospitalization.
Cap (community acquired pneumonia)
40
Common Causative Agents: for pneumonia
Streptococcus pneumonia (40%) MOST Klebsiella (20%) Haemophilus influenzae Legionella, mycoplasma, Chlamydia
41
3rd most common cause of morbidity among nosocomial infections.
Hap (hospital acquired pneumonia) healthcare-acquired
42
A lung infection that develops in a person who is on a ventilator.
Vap (ventilator acquired pneumonia)
43
Vaccination for pneumonia
Haemophilus influenzae Streptococcus pneumoniae Pertussis, varicella, and measles
44
Sputum collection: for TB considerations
Best obtained on three consecutive mornings. Clean and thoroughly rinse the mouth with water. Breathe deeply 3 times. After the third breath, cough hard and try to bring up sputum from deep in the lungs