Cerebellum Flashcards

exam 1 (29 cards)

1
Q

cerebellum are connected by

A

cerebellar peducles

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2
Q

inferior cerebellum is found in the ______ and mostly contains ____ to cerebellum

A

found in the medulla and mostly contains inputs to cerebellum

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3
Q

Middle cerebellum found in ______

`and contains only ____ to cerebellum

A

found in pons and only input to cerebellum

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4
Q

superior cerebellum found in ______ and contains mostly _____ from cerebellum

A

found in midbrain and mostly contain output from cerebellum

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5
Q

lesions in cerebellar leads to

A

ipsilateral deficits

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6
Q

does the cerebellum contain more neurons than the cerebral cortex?

A

heck yea

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7
Q

what is vulnerable for occlusion?

A

4th ventricle

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8
Q

Mollaret’s triangle

A

is a sel-contained circuit involving the inferior olive, dentate and red nucleus

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9
Q

climbing fibers from the inferior olive go where?

A

direct to the purkinje fibers

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10
Q

what is the order in which cells receives the input in the cerebellum?

A

internal granular (packed with cells) —> Molecular —>Purkinje

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11
Q

most incoming fibers synapse on

A

granular cell dendrites

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12
Q

what are the deep cerebellar nuclei?

A

output nuclei

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13
Q

vermis involved with

A

trunkal control

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14
Q

flocculondular lobe?

A

vestibulocerebellum

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15
Q

vestibulocerebellum receives input t (and its output is to) ________ and when lesioned it affects

A

input from vestibular nuclei and when lesioned it affects balance and gait ataxia

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16
Q

spinocerebellum receives input (and its output is to) _______from and when lesioned it affects

A

receives input from spinocerebellar tract and olives and when lesioned it affects trunkal coordination

17
Q

Neocerebellum input is from _______ and output is ________

A

input: cortex via pontine nuclei
output: back to cortex via VL of thalamus

18
Q

neocerebellum lesioned affect

A

upper extremities dyscoordination

19
Q
vascular syndrome  of the cerebellum that are caused by"
- hemorrhage/infarct
-toxic exposure
- heat stroke
anoxia
A

acute cerebellar disease

20
Q

Subacute cerebellar disease:

  • alcoholic–>
  • Praneoplastic –>
  • Cerebellar tumors –>
  • MS –>
A
  • alcoholic–> vermis
  • Praneoplastic –> antibodies vs. purkinje cells leading to cerebellar ataxia
  • Cerebellar tumors –> occlusion of the 4th ventricle
  • MS –> cerebellum white fibers
21
Q

inherent disease of the cerebellum that can be caused by:

  • congenital cerebellar hypoplasia
  • genetic disorders like spinocerebellar ataxia
  • neurodegenerative like olivopontocerebellar degeneration that has parkisonian like features
  • metabolic diseases
A

chronic cerebellar disease

22
Q

How does the cerebellum differ in its representation of the body?

A

ipsilateral strcuture

23
Q

what does this mean for input and output?

A

remains ipsilateral or crosses twice

24
Q

what is carried in each peduncle?

superior:

middle:

inferior:

A

superior: output
middle: cortico-ponto-cerebellar fibers
inferior: spinocerebellar, vestibulocerebellar

25
How is the cerebellar cortex organized?
input to granular, synapse on purkinje cell in molecular Purkinje cell to deep nuclei
26
what are the functional divisions?
lateral, vermis and paravermis, flocculonodular
27
Clinical symptoms of cerebellum issues
- nystagmus - incoordination, intention tremor - dysdiadochokinesis - hypotonia- flaccid tone
28
dysdiadochokinesis
rapid, altering movements
29
lesion within the mollaret's triangle
palatal myoclonus