Chapter 8 Flashcards

(64 cards)

1
Q

Adaptation to training is governed by 3 principles

A

Overload
Specificity
Reversibility

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2
Q

Weight training increase

A

Force generating capacity and muscle size

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3
Q

Training regimen must be carefully chosen to

A

Maximize strength with minimal mass gain

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4
Q

Training aims at increasing what proteins

A

Actin and myosin

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5
Q

Low force -low velocity training train which type of muscles first ?

A

Type 1 fibers

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6
Q

Low force -low velocity training train typeI fibers and exception is

A

When type I fibers are fatigued/damaged from other training, then type II will be recruited

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7
Q

Increases muscle size is

A

~ increase cross sectional area

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8
Q

Measurable increase in muscle size ban be seen after

A

~ 15 training sessions

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9
Q

When training for speed muscle mass

A

Increases

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10
Q

When training for speed , anaerobic capacity

A

Increases

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11
Q

When training for speed muscle ATP /Pcr

A

Unchanged

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12
Q

When training for speed key glycolytic enzymes such as phosphofructokinase

A

Increase

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13
Q

When training for speed buffering capacity

A

Increases

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14
Q

When training for speed proportions of fiber types in terms of number of fibers

A

Unchanged

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15
Q

When training for speed proportion of fiber types by weight or size

A

Does change

Enlargement of type II fibers

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16
Q

Sprint training changes

A

Muscle - enzyme activity

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17
Q

Sprint training changes

A

Muscle - enzyme activity

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18
Q

Sprint training changes muscle-enzyme activity these changes are —-dependent

A

Stimulus

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19
Q

T/F

In sprint training, enzymes for anaerobic metabolism can increase

A

T

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20
Q

Elite middle distance needs

A

High capacity lactate production

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21
Q

In training for middle distance what needs to be increased

A

Glycolytic rate limiting step

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22
Q

Enzymes to produce glucose from glycogen

A

Glycogen phosphorylase
Phosphofructokinase
Lactate dehydrogenase

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23
Q

Use of FA as fuel is determined by

A

Rate of delivery to the muscle

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24
Q

What is one of the most important endurance adaptations

A

Capacity to use FAs as fuel

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25
Acts as small store of O2 for times of high demand
Myoglobin
26
The most important function in endurance athletes
Diffusion of O2 Within the muscle cell
27
T/F? | Aerobic-enzyme activity and max. respiratory capacity is higher by 2-3x in trained athletes
T
28
T/F Enzyme activity increases with even small amounts of training
T
29
What is the function of endurance training on intramuscular level of TAG
Increases
30
Effect of endurance training on rate of muscle glycogen
Decreases it 😳
31
T/F Myofibrils can be rebuilt with a different type of myosin
T
32
T/F | Unused muscled dont adapt
T
33
Passive stretch induce
Muscle enlargement
34
In passive stretch mechanical load must be included to activate
Signaling pathways of ribosomal machinery and transcription of genes
35
Activation of this signaling pathway appears crucial in increasing protein synthesis following resistance exercise
MTor
36
Over-training syndrome
Under-performance in spite of continued/ increased training
37
Over-reaching is
Temporary with full recovery or performance improvement tin a few days
38
Over-reaching may be related to
Insufficient recovery of muscle glycogen or exercise induce muscle damage
39
T/F | Heavy training makes one less susceptible to infection
F
40
T/F 2-6 times more likely to upper respiratory infection in long distance runners
T
41
Increase in circulating leukocyte amount related to
Intensity/duration of exercise
42
Hormonal change effect with strenuous exercise
Adrenaline Cortisol Growth hormone Prolactin
43
T/F Phagocytic neutrophils activated during strenuous axe rise but have diminished killing ability for many hours
T
44
T/F NK cells increase during exercise but drop to < 1/2 normal levels for some hours
T
45
Normal level of NK cells return after
24 hrs
46
High training loads are associated with chronic
Immunodepression
47
T/F Overreaching for > 10 days may result in chronically depressed immune function
F > 6 days
48
T/F Training adaptations are reversible
T
49
What can prevent or slow detraining
Short periods of high intensity training
50
T/F High fat diets may improve capacity to oxidize FFA in well trained athletes
T
51
T/F Low CHO training may have stress hormone responses that cancel benefits
T
52
T/F Antioxidant are produced by normal aerobic metabolism
T
53
Singlet O2 has a pair of electron with
Opposite spins
54
Antioxidants prevent H abstraction from
FA side chains
55
Antioxidants bind to metal ion catalysts to
Prevent initiation of free radical reactions
56
Low molecular weight substances that act as free radical scavengers
Vitamin A, C, E Carotenoids - beta carotine Plant polyphenol
57
What are the plant polyphenols
``` Tea Wine Fruit Vegetables Flavonoids -subgroup ```
58
What are the advantages of ROS and RNS ?
Moderate levels of ROS are essential to optimal force production in muscle Also a signaling mechanism
59
In heavy smokers high -----and -----intake increases incidence of lung cancer
Vitamin E | beta-Carotine
60
T/F High dose of antioxidants inhibit apoptosis
T
61
Vitamin A in pregnant women causes
Birth defects
62
Vitamin E can impair absorption of
Vitamin A and K
63
NSAIDs Studies show a post exercise suppression of
Creatine kinase response
64
T/F? | NSAIDs are recommended to treat symptoms of muscle damage
F NOT recommended