Dysrhythmias Flashcards

(64 cards)

1
Q

automacity function

A

Ability to generate electrical impulse

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2
Q

Excitability function

A

Ability to respond to outside impulse

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3
Q

conductivity function

A

Ability to receive electrical impulse and conduct it

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4
Q

contractility function

A

Ability of myocardial cells to shorten in response to an impulse

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5
Q

when does a P-wave occur?

A

During atrial depolarization

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6
Q

when does QRS complex occur?

A

During ventricular depolarization

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7
Q

when does T-wave occur?

A

During ventricular repolarization

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8
Q

which node initiates atrial depolarization?

A

SA node

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9
Q

T/F, the AV node initiates ventricular depolarization

A

True

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10
Q

normal heart rate

A

60 to 100 per minute

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11
Q

normal P-wave occurrence

A

Upright and rounded
One before every QRS complex
Even regular rhythm

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12
Q

Normal PR interval time

A

12 to 20 seconds

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13
Q

normal QRS time

A

less than 12 seconds
Narrow

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14
Q

what do normal rate, rhythm, P-wave, PR interval, QRS indicate

A

Sinus rhythm

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15
Q

sinus arrhythmia

A

Fluctuation in rate and initiation
Variability in heart rate

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16
Q

is sinus arrhythmia still normal?

A

Yes, no changes to cardiac output

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17
Q

Who is sinus arrhythmia more common in?

A

Younger people
Associated with respiratory and autonomic nervous system fluctuations

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18
Q

dysrhythmia

A

Abnnormal cardiac rhythm
Problem with impulse generation or conduction

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19
Q

do dysrhythmias alter cardiac output?

A

Yes

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20
Q

Inappropriate automaticity cause of dysrhythmia

A

Cell initiates action potential when it is not supposed to

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21
Q

Triggered activity cause of dysrhythmia

A

Extra impulses generated during/right after repolarization

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22
Q

reentry cause of dysrhythmia

A

Cardiac impulse in one part of heart continues to depolarize after main impulse is finished

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23
Q

sinus-

A

Triggered in/around SA node

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24
Q

sinus bradycardia

A

Slow heart rate <60 BPM

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25
is sinus bradycardia still normal?
Yes – regular rhythm, PR, QRS
26
causes of sinus bradycardia
Hyperkalemia – slows depolarization Vagal response Digoxin toxicity late hypoxia meds – beta blockers, CCB MI – ischemia
27
s/sx sinus bradycardia
lightheaded, dizziness Easily fatigued Syncope Dyspnea Chest pain/discomfort Confusion
28
treatment for sinus bradycardia
Assess for symptoms first If symptomatic: atropine If atropine, not effective – pacemaker
29
sinus tachycardia
Fast HR 100-150 bpm
30
is sinus tachycardia still normal?
Yes, but P waves may be partially hidden PR, QRS, rhythm still normal
31
causes of sinus tachycardia
Exercise Pain Strong emotions Fluid volume deficit Fever – increased metabolic rate Meds – Epi, albuterol caffeine, nicotine, cocaine Early hypoxia
32
treatment for sinus tachycardia
based on cause hypovolemia – fluids Fever – antipyretics Pain– analgesics Beta blockers to lower heart rate and myocardial oxygen consumption
33
paroxysmal supraventricular tachycardia
HR 150 to 200 BPM “ feeling like my heart is racing”
34
is PSVT normal?
Usually no P waves, if present is abnormal QRS – normal
35
T/FPSVT usually begins and ends slowly
False, suddenly
36
causes of PSVT
Overexertion Emotional stress Stimulants Digoxin toxicity Rheumatic, heart disease, CAD, R HF Wolf Parkinson, white syndrome
37
s/sx PSVT
Palpitations Chest pain Fatigue Lightheaded, dizzy Dyspnea
38
where does PSVT originate?
Above ventricles, AV node
39
where do sinus bradycardia and tachycardia originate?
SA node
40
which sinus rhythm is caused by early hypoxia?
Sinus tachycardia
41
Which sinus rhythm is caused by late hypoxia?
Sinus bradycardia
42
premature atrial contractions
Early P waves that have morphological changes
43
what should you check with premature atrial contractions?
Electrolytes May need oxygen
44
T/F premature atrial contractions are fatal
false, usually no consequences If frequent, puts patient at more risk for other dysrhythmias
45
does premature atrial contractions have a normal PR and QRS interval?
Yes
46
atrial flutter
Reentry impulse, repetitive and cyclic rate >250, still regular QRS is slower P-wave looks like shark tooth
47
where does atrial flutter originate?
AV node Overrides SA node
48
causes of atrial flutter
CAD, cardiomyopathy Heart valve disease Congenital, heart disease Inflammation of heart – myocarditis HTN Lung disease/overactive thyroid Electrolyte imbalance Heart surgery
49
atrial fibrillation
Multiple irritable spots in atria Irregularly irregular HR 100-175 no identifiable P-wave Fibrillation waves
50
T/F atrial fibrillation is more symptomatic than atrial flutter
True
51
s/sx a fib
palpitations Heart racing Fatigue Dizziness Chest discomfort SOB Can be asymptomatic**
52
causes of a fib
Electrolytes imbalance Hypoxia CBD
53
complications of a fib
Decreased cardiac output Heart failure Embolus – stroke risk
54
what is the most common type of treated dysrhythmia?
Afib
55
treatment for a fib
Control the rate – BB, CCB, digitalis, amiodarone Stroke prevention – anticoagulates antiplatelets
56
Non-medication treatment for a fib
ablation Cardioversion
57
Premature ventricular contractions
ventricular dysrhythmia Contraction coming from ectopic focus in ventricles Comes earlier than QRS complex should Not a normal rhythm or P wave
58
what does the QRS complex look like in PVC?
Wide and distorted shape
59
causes of PVC
Stimulants Electrolytes Hypoxia Exercise Stress Fever CVD
60
ventricular tachycardia
Three or more PVCs together Ectopic focus within ventricles takes control and fires repeatedly rate 150-200 bpm
61
differences with ventricular tachycardia
No atrial contractions No P-wave PR interval not measurable
62
causes of ventricular tachycardia
MI CAD ABNORMAL ELECTROLYTES HEART FAILURE Drug toxicity
63
what is an immediate intervention for ventricular tachycardia and fibrillation?
Check for a pulse
64
ventricular fibrillation
Irregular waveforms of varying shapes and sizes Ventricles are quivering No effective contractions equals no cardiac output