PVD Flashcards

1
Q

PVD

A

Peripheral vascular disease
Umbrella term for arteries and Venus diseases

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2
Q

patho of PVD

A

atherosclerosis in extremities

Ischemia reperfusion

Reduced myofibers, impaired, peripheral nerve function, muscle damage, degeneration

Impaired oxygen consumption

Increase the rate of mobility loss

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3
Q

risk factors for PVD

A

Smoking
Diabetes, high cholesterol
Heart disease
Stroke
>50

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4
Q

cause of PVD

A

Most common – atherosclerosis
Thrombus – clotting
Inflammation of arteries– thromboangitis obliterans
Vasospasm – raynauds disease

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5
Q

s/sx PAD

A

due to local tissue, ischemia an appearance in extremities

calf, buttock pain – numbness, burning, heaviness, intermittent claudication

Wounds that won’t heal
Decreased sensation in extremities
round, smooth sores on toes and feet
Black eschar

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6
Q

what are trophic skin changes in PAD?

A

Shiny and thick skin, toenails
Loss of leg hair
Decreased pulses
Pallor, cyanosis
Reactive hyperemia/dependent Rubor
Erectile dysfunction

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7
Q

intermittent claudication

A

Consistent pain precipitated by a consistent level of exercise

Cramping, Angina of lower extremities

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8
Q

T/F intermittent claudication does not stop with rest

A

False

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9
Q

what is intermittent claudication caused by?

A

Ischemic tissue
Arterial flow obstruction

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10
Q

where is a common site of PAD?

A

Femoral artery

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11
Q

5 P’s of PAD

A

Pain– intermittent claudication
Pulselessness
Palpable, coolness
Paresthesias
Paresis – weakness

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12
Q

dx of PAD

A

ankle brachial index
Compare blood pressure in leg versus arm

Severe – 0.5 ratio.

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13
Q

s/sx PVD

A

dull, achy pain
Lower leg edema
Pulse and drainage present
Sores with a regular borders on ankles
Redskin
Yellow slough

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14
Q

chronic venous insufficiency

A

Occurs when venous wall and/or valves in leg veins are not working effectively

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15
Q

venous stasis

A

Blood pools or collects in veins

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16
Q

s/sx chronic venous insufficiency

A

Lower extremity edema
Achyness, tiredness
Leathery looking skin – dark, hard
Stasis ulcers on ankles
Flaky, itchy skin
New varicose veins

17
Q

valve disease types

A

wear and tear
Calcification
pannus
Endocarditis – inflammation infection
Thrombus – clot

18
Q

stenosis valve

A

Very hard, tight, stiff
Hard for blood to get through

19
Q

regurgitation valve

A

Very loose, blood flows backward and increases pressures

20
Q

aortic stenosis triad

A

Chest pain
SOB
Lightheaded

21
Q

endocarditis

A

Vegetation of valves – infective mass
Septic emboli
Can cause pulmonary occlusion

22
Q

risk factors for endocarditis

A

Prosthetic valve
Pacemaker
IV drug abuse

23
Q

causative agents for endocarditis

A

Strep viridans
Staph aureus
Staph epidermidis

Contaminated needle, dirty skin

24
Q

s/sx endocarditis

A

fever, chills
Anorexia, weight loss
Heart murmur
Myalgias, arthralgias
Ischemia in extremities, spleen, kidney, bowel, brain
septic emboli can cause stroke

Neuro sx– meningitis, seizures, and cephalopathy, abscess of brain

25
septic emboli
Infected blood clots Travel to heart adhere to damaged endothelial tissue attract white blood cells and platelet’s release cytokines and coags
26
patho of septic emboli
simulation of coags– fibrin deposition– develop vegetation Vegetations found in valve, leaflets, cannibalize into circulation Carried by bloodstream – infection/ischemia in tissue
27
s/sx septic emboli
Petechiae – non-blanchable Splinter hemorrhage Janeway lesions olsers nodes roth spots
28
Splinter hemorrhage
Linear streaks and nails
29
Janeway lesions
Red, swollen, nontender on palms and soles
30
olsers nodes
Subcutaneous nodules on fingertip pads
31
roth spots
Oval retinal hemorrhage with pale center
32
why are drug users involved with septic emboli?
Veins are portals of entry with drug use
33
duke criteria of infectious endocarditis
Two Major One major, three minor Five Minor.
34
Treatment for infectious endocarditis
antibiotics – blood culture to treat specific bacteria 4 to 10 weeks, lengthy hospital stay PICC line used