electrocardiography/reading ecg Flashcards

1
Q

is the sa node acted on by other cells

A

no

Spontaneously active cells

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2
Q

what is the p wave

A

Conducts current down atria & internodal pathways

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3
Q

function of av node

A

Current slows down more time for atrial muscle contraction

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4
Q

how does current travel down the septum

A

His-Purkinje system

Rapid conduction through left & right bundles -> Purkinje fibres -> ventricular contraction

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5
Q

how is that complex shown on an ecg

A

QRS complex of ECG

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6
Q

what is the t wave

A

Cardiac muscle relaxes & membrane potential recovers/repolarises
T-wave of ECG

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7
Q

what does lead II read

A

electrical currents from right shoulder to left leg

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8
Q

which lead would have the largest current (biggest deflection of the qrs)

A

lead II
Electrical current flowing away from the positive electrode results in a negative (downward) waveform display. In Lead II, the R wave is therefore positive. … The closer the mean QRS axis is to the electrical axis of Lead II, the larger the amplitude of the R wave.

qrs is going the exact same direction as the positive component of lead 2

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9
Q

how would the qrs change if lead II wasn’t perfectly parallel with the direction of the current

A

a lower qrs

less deflection in qrs as not parralell

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10
Q

what is axis deviation

A
  • change in heart position
  • change in position of electrical conduction ( one of the right or left bundle branches can de damaged or one more dominant that the other- current goes in a different direction)
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11
Q

where does lead 1 measure

A

horizontal

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12
Q

ii

A

right shouled to left lef

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13
Q

iii

A

left shoulder to right leg

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14
Q

aVF

A

down the middle

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15
Q

aVR

A

right leg to left shoulder

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16
Q

how would the p wave chnage if a pacemaker is producing currents in mixed directions

A

absent p wave

lots of small signal drown our the larger signal to av node

17
Q

what is this condition known as

A

Atrial fibrillation - loss of p-wave

18
Q

what is Atrial flutter

A
  • saw-tooth pattern
19
Q

what would happen if we had a barrier or problem at the av node

A
  • longer pr interval
    Type I Heart Block
    Type II Heart Block
20
Q

from what 2 points is the qr interval measured from

A

from start of p to start of q not R

21
Q

what is a type 1 heartblock

A

delay in conduction
calcium channel heart block
increase p r interval duration

22
Q

what is type 2

and cause

A

loss of conduction
more of an actual block
missing qrs complex ans thus t
due to damage to cells or drugs

23
Q

what is the Wolff-Parkinson-White

A

is caused by an extra electrical pathway in the heart. This allows electrical signals to bypass the usual electrical system route and form a short circuit, resulting in an abnormally fast heart rhythm.
accessory pathway

24
Q

what is it shown on an ecg

A

shorter pr complex
pre excitation
biphasic t wave

25
Q

what causes a biphasic t wave

A

currents are going thru faster

alight depolarisation due to accessory pathway

26
Q

what causes pre excitation

A

accessory pathway allows the current to get from atria to ventrciles faster

27
Q

what is the bottom graph in the ecg

A

rythm strip

so one lead shown for a longer period of time normally lead ii

28
Q

what atrcuture do you use to read ecgs

A

rate ryhthm and axis

29
Q

what is the rate

A

r r interval

30
Q

how to work it out

A

300/ number of large swuares e.g. 3,3

31
Q

what is the rhythm

A

p r interval
qrs duration
st segment

32
Q

what is the st segment

A

the straight part from s to t

33
Q

how do you work them out

A

each small square is 40ms

34
Q

how do you work out axis

A

guestimate using the diagram

35
Q

what is the point of multiple leads

A

greater resalution

more detail abouts whats going on