Exam #2 Respiratory Flashcards

(134 cards)

1
Q

Before born, lungs are not needed.

A

True

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2
Q

Less than 32-33 weeks of age, baby does not have sufficient amount of surfactant.

A

True

Biggest change in the NICU is the development of surfactant

If less than 34 weeks 1st dose of surfactant is given at birth

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3
Q

Fake surfactant = decrease incidence of -

A

RDS - Respiratory Distress Syndrome

and

BPD- Bronchopulmonary Dysplasia

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4
Q

** Respiratory Distress Syndrome =

A

Lack of Surfactant

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5
Q

Bronchopulmonary Dysplasia

A

hard alveoli due to constant ventilator causing positive pressure.

lung tissues get pounded until gets tough

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6
Q

Treatment for Apnea of Prematurity

A

Caffeine

Can give through IV or Orally

Monitor for Toxicity

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7
Q

What is Apnea of Prematurity?

A

pause longer than 20 seconds

Apnea spells can last so long that they cause bradycardia

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8
Q

Sudden Infant Death Syndrome (SIDS) interventions:

A

sleep on back

No Bumpers

pacifier after 1 month

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9
Q

A + B Spells =

Apnea and Brady

A

How premie tells us they are sick

If alarm goes off, ASSESS FIRST

Infants breathe periodically- breathe, Breathe, Pause
—-Happens in Premature babies

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10
Q

Preterm babies =

A

Bronchiolitis

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11
Q

** S/S of Caffeine Toxicity

A

vomit

Irritable

Tachycardia

Jittery/ excitability

Tremors

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12
Q

Toddlers and Preschoolers breathe abdominally

A

True

use of abdominal muscles to breathe until 5 years old

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13
Q

Infants have irregular breathing patterns and are nose breathers.

A

True

they have smaller, less rigid airways

smaller lung size

horizontal, short eustachian tubes in Infants

Immature immune systems

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14
Q

Surfactant was given in clinical trials and had to stop because was unethical.

A

True….I guess….

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15
Q

What is a late sign of respiratory distress?

A

Cyanosis

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16
Q

Assessing Respiratory Status - VISUAL ASSESSMENT =

A
  • work of breathing such as grunting, flaring, retracting
  • Rate of breathing
  • Chest Movement- is it equal?
  • Posture and activity level - Well Flexed alert and awake**
  • Sensorium (LOC)
  • Level of comfort
  • Color- skin and mucus membranes
    - pink? sats in the 90’s
    - Grey? 80’s or lower
    - Worried about mucus membranes
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17
Q

Assessing Respiratory status - AUSCULTATION

A

Without the stethoscope you can hear grunting, stridor, or wheezing.

With Stethoscope- inspiratory and expiratory- will usually tell us if it is something in the upper or lower airway

Baby- Listen to Axillae and back

Older listen to front and back

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18
Q

While grunting- baby uses abdominal muscles to push air out.

A

True

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19
Q

What are the cardinal signs of respiratory distress?

A

Tachypnea
Restlessness (Huge sign) , Confusion, Anxiety, Irritability
Tachycardia
Diaphoresis

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20
Q

What is the earliest sign of respiratory distress?

A

Tachycardia

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21
Q

What are additional signs of respiratory distress?

A

Wheezing

grunting, flaring, retracting

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22
Q

What are the signs of Impending Respiratory Failure?

A

Depressed or slow respirations (Decreased inspiratory breath sounds)

Dyspnea

Bradycardia

Somnolence

Stupor/coma

Cyanosis (Central (Mucus membranes)

Oxygen desat

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23
Q

What are 2 signs that a baby is about to code?

A

80’s o2 sat

and

Bradycardia

Means about to code…,.

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24
Q

O2 Sats falling are a sign of respiratory failure….

A

true

dont wait for them to become cyanotic

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25
Why is tachypnea a sign of respiratory distress?
because if you have trouble breathing, you are going to breathe faster to get more air.
26
Acidosis
pH Less than 7.35
27
Respiratory Acidosis causes =
ventilation problem need to be ventilated with oxygen
28
Metabolic Acidosis causes =
Diarrhea, Kidney failure, DKA
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Alkalosis =
pH greater than 7.45
30
Respiratory Alkalosis causes =
test taking, any rapid respiratory rate to little co2, hyperventilating
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Metabolic Alkalosis causes =
Vomiting
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Respiratory Acidosis S/S =
``` Dyspnea Respiratory Distress Shallow respirations Headache Restlessness Confusion Tachycardia Dysrhythmias ```
33
Respiratory Alkalosis S/S =
``` Tachypnea Light Headedness numbness and tingling confusion/ can't concentrate Blurred Vision Dysrhythmia Palpitations / Diaphoresis Dry Mouth, Tetanic spasms of arms and legs ```
34
CPAP-
has prongs gives positive pressure to keep stimulus for breathing….
35
Piaget --- FUNCTIONAL
blow balloons and bubbles in place of Incentive Spirometer.
36
Oxygen Safety?
AVOID NYLON AND WOOL - Avoids materials that generate static electricity, such as wool blankets and synthetic fabrics, Cotton Fabrics and blankets. - Avoid the use of volatile, flammable materials such as oils, greases, alcohol, ether, and acetone (Nail polish remover) near clients receiving oxygen - make sure that electric devices (such as razors, hearing aids, radios, TV, and heating pads) are in good working order to prevent the occurrence of short circuit sparks. - For home oxygen use teach family members to smoke outside away from the client.
37
Respiratory Scoring Sheet
Gives an Idea and can help determine if kids can leave the PICU
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Inspiratory sounds-
Inspiratory Stridor = Upper airway obstruction (Trachea)
39
Wheezing- Exhalation =
Lower airway obstruction (Asthma, Bronchiolitis) Bronchioles and alveoli
40
Upper Airway Obstruction causes :
Foreign body aspiration Swelling of tissues (Croup, tonsils, Epiglotis) Congenital Narrowing of upper airway
41
Clinical Signs of Upper airway Obstruction :
Tachypnea Increased Respiratory Effort Hoarse voice or Cry (seal like cough) Stridor
42
Croup = 4 types of croup
1. larygotracheobronchitis 2. Acute spasmotic croup 3. Epiglotis 4. Bacterial tracheitis
43
Croup General Info-
late autumn / early winter 6 months to 3 years Barking cough, hoarseness, inspiratory stridor respiratory distress
44
What is the most common type of croup?
1. larygotracheobronchitis
45
larygotracheobronchitis LTB or laryngotracheitis
Most common of the croup syndromes. - Sound worse than they look - Abrupt Onset, usually at night Generally Effects children LESS THAN 3 years Organisms responsible : Viral - RSV, Parainfluenza virus, Mycoplasma pneumoniae, Influenza A and B
46
Treatment of Croup LTB - (larygotracheobronchitis)
Dexamethasone - Oral or IM - 0.6 mg/kg - Duration of action is 48 - 96 hours*** Nebulized Epinepherine - Racemic Epinepherine - Used to dilate the airway - Alpha adrenergic Effects = Mucosal Vasoconstriction - Duration 1 - 2 hours *** - Observe the patient for 2 - 4 hours (to make sure airway doesnt collapse again.)
47
Dexamethasone - Oral or IM
- 0.6 mg/kg | - Duration of action is 48 - 96 hours***
48
Nebulized Epinepherine - Racemic Epinepherine
- Used to dilate the airway - Alpha adrenergic Effects = Mucosal Vasoconstriction - Duration 1 - 2 hours *** - Observe the patient for 2 - 4 hours (to make sure airway doesnt collapse again.)
49
Croup LTB Info-
Cough medications and decongestant meds are contranindicated because want baby to cry and cough to keep airway patent. Over the counter medications, esp cough and cold meds should not be administered to CHILDREN UNDER 2 YEARS OF AGE ********* Controversy over whether cool humidified air works. Still may recommend cold water vaporizers, shower vapor, cool night air. Suction out the child
50
Over the counter medications, esp cough and cold meds should not be administered to CHILDREN UNDER 2 YEARS OF AGE *********
true
51
Epiglottitis -
MEDICAL EMERGENCY Sudden swelling and about to swell shut Can't talk Elective intubation Dysphagia cannot swallow
52
Epiglottitis Clinical Manifestations:
- Sudden Onset, Fever and sore throat - Usually in patients 2 - 8 years old ******Drooling, Dysphonia, Dysphagia - Tripod positioning with retractions and flaring - Inspiratory Stridor, mild hypoxia, distress ****** Not hoarse, NO COUGH
53
Epiglottitis Diagnostic Testing :
Lateral neck x-ray Thumb print on x-ray is indicative of swollen epiglottis
54
Epiglottitis positioning =
stay with child and parent can keep child calm. Ask for the people to come to them. Let them stay in whichever position they are in.
55
Therapeutic Management of Epiglottitis
Potential for respiratory obstruction Nursing Considerations: - No Tongue Blades - Maintain calm - Emergency Intubation Equipment on hand - No cultures Do a conscious sedation and intubate them….
56
Epiglottitis Prevention -
HIB Vaccine | Influenza Type B
57
Bacterial Tracheitis-
Attacks the Trachea Occurs in the fall and winter months ages 6 months to 6 years URI for several days or classic viral croup Staph aureus, H. Influenzae, Strep Decompensates: high fever, productive cough, respiratory distress Endotracheal intubation, mechanical ventilation, IV antibiotics *******Same problem as epiglottitis but not 3 D's (Dysphagia, Drooling, Dysphonia) Treat with elective intubation
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Lower Airway Obstruction causes:
asthma Bronchiolitis
59
Lower Airway Obstruction clinical Signs:
Typically heard on expiration Wheezing - airway narrows (Rales- crackles) Rhonchi in larger airway Tachypnea Retractions, and nasal flaring Prolonged expiration phase combined with expiratory effort Cough
60
Bronchiolitis
RSV= Respiratory Syncytial Virus- affects small airways NO ANTIBIOTICS ``` #1 Nursing Intervention: Contact Isolation #2 Assess Respiratory Status #3 IV Placement #4 I & O's ```
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Bronchiolitis Diagnostics ;
Culture secretions
62
Bronchiolitis Therapeutic Management :
Hydration Rest Humidification Increased fluid Intake need pulse ox bag mask BP cuff Bulb Syringe
63
Bronchiolitis and RR Greater than 60, baby is at risk for
Aspiration
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RSV Respiratory Syncytial Virus
Born before 33 weeks give synergist - Prevention of RSV: Prophylaxis- Palivizumab- Need once a month during RSV period if under 35 weeks gestation… until 2 Ribavirun - antiretroviral med rarely used. Used for life or death situations only….Not typical treatment for RSV anymore.
65
When is RSV Season In TN
October - March is RSV season in Tennessee
66
Asthma -
Chronic Inflammatory disorder of the airways limited airflow or obstruction that reverses spontaneously or with treatment bronchial hyperresponsiveness Episodic tightening of smooth muscle
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Trach Care-
- if you go past tip, you are suctioning bronchial tissue - Normal saline can wash microbes that are around the mouth area into the lungs. Suction the back of the mouth to get the secretions out. - Non- Sterile cath used to suction mouth first then get sterile cath to suction ET Tube. - Suction for 3-5 seconds - 2 Trachs at bedside.. One same size and one smaller. - Mouth care is as important as trach care.
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Anti Inflammatory Drugs-
Inhaled corticosteroids Oral or Parenteral Corticosteroids Leukotriene Receptor Antagonists
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Inhaled corticosteroids Examples
``` Flucticasone proprionate ( Flonase, Flovent) Budesonide - (Pulmicort) ``` Steroids can cause thrush in mouth, use spacer and rinse out mouth with water or brush teeth after use.
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Leukotriene Receptor Antagonists
Montelukast (Singulair)
71
Corticosteroids-
In low doses for asthma Side effects: Cough, dysphonia, ORAL THRUSH**, Monitoring: Every 3-6 months- growth parameters - Can cause diminished growth
72
Leukotriene Modifiers
Blocks inflammatory and bronchospasm effects of leukotrienes Zafirlucast (Accolate)- for children 7 years and up Montelukast sodium (Singulair) for children 1 year and older ***Can cause aggression and anxiety
73
Montelukast (Singulair) Side Effects:
Headache Can cause increase in SGOT/SGPT- use cautiously in patients with impaired liver function Patients with PKU- Montelukast contains phenyalanine Linked with odd psycho effects such as strange mood changes.
74
Beta 2 Adrenergic Agonists-
allow smooth muscle to relax inhaled form hasa a more rapid onset of action than the oral form side effects: seen mostly in oral form. irritability, tremor, nervousness, and insomnia
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Beta 2 Adrenergic Agonists- examples
Albuterol, levabuterol, (Short acting) Salmeterol - Long acting Action: Relaxes airway smooth muscle Indication: Acute and Chronic treatment of Wheezing Adverse Reactions: Nervousness, tachycardia, jitteriness Nursing Considerations: Inhalation route has fewer side effects
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Albuterol
Sympathomimetic Drug Stimulates beta 2 adrenergic receptors in lungs causing bronchial smooth muscle relaxation CAUTION: May increase risk of arrhythmias in Digoxin patients Side Effects: HA, Nausea, Restlessness, Nervousness, Trembling
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Albuterol Overuse
1. Loss of bronchodilation effects | 2. SEVERE paradoxical Bronchoconstriction
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Nursing Considerations for Albuterol
client family teaching Wait 2 min before inhaling 2nd puff
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Salmeterol Servent
long acting - salmeterol (servent)- Used no more than every 12 hours. Not used in children under 12 years old NEVER USED FOR ACUTE SYMPTOMS
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What are triggers for asthma :
allergens- dust, animal dander, smoke Cold air Weather changes Infection Exercise Fatigue Emotional Distress Environmental changes like starting a new school (ASK ABOUT WHAT THE PATIENTS INDIVIDUAL TRIGGERS ARE)
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Atopy
genetic predisposition for the development of an IgE mediated response to common aeroallergens Strongest predisposing factor for developing asthma Born with more IgE All Children have atopy Hyperresponsiveness to triggers
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Antihistimines
Loratadine (Claritin) &Cetirizine (Zyrtec) Fexofenadine (Allegra)
83
Loratadine (Claritin) &Cetirizine (Zyrtec) MOA=
competes with histamine on H1 receptor sites
84
Fexofenadine (Allegra) MOA=
antagonizes histamine effects
85
What are the side effects of antihistamines?
Side effects: headache, dry mouth, drowsiness (Opposite of muscarinic man) Children may experience a paradoxical reaction of restlessness, insomnia, nervousness
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Aspirin Triad
A subpopulation of asthmatic patients who react with acute dyspnea within 2 hours after ingestion of aspirin. Triad: Chronic rhinosinusitis including polyps, severe bronchial asthma, and intolerance to aspirin and other NSAIDs Client/family education: use acetaminophen
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Drug Therapy for Asthma Long-term meds/ Preventive
``` Corticosteroids Cromolyn sodium Albuterol Salmeterol Leukotriene modifiers ```
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Drug Therapy for Asthma Quick relief/Rescue meds
Albuterol Ipratropium (anticholinergic) Magnesium sulfate IV
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Asthma action plan
…..
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Peak Expiratory Flow Rate
80-100% Green 50 – 80 % Yellow < 50% Red
91
What is red on peak expiratory flow rate?
less than 50%
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What is green on peak expiratory flow rate?
80 - 100%
93
What is yellow on peak expiratory flow rate?
50 - 80 %
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What is the patient teaching for a peak expiratory flow rate?
Done in morning. Standing. Have child “huff and cough” 2 or 3 times to clear airway and set meter gauge on zero before beginning test.
95
Asthma meds and nursing management
Baseline assessment of depth, rate, rhythm, and type of patient respirations Post-treatment assessment of same Monitor the quality and rate of patient’s pulse Assess the patient’s lung sound for crackles, rhonchi, and wheezing Observe fingernails and lips for signs of cyanosis
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Client/Family Teaching For Asthma
Instruct patient and family to increase fluid intake to decrease viscosity of lung secretions Never abruptly discontinue asthma meds Practice good, oral hygiene House at 50 – 60 % humidity No carpet, but if carpet vacuum daily
97
Status Asthmaticus
Respiratory distress continues despite vigorous therapeutic measures Medical emergency Humidified oxygen Aerosolized short-acting Beta 2 –agonist IV access Emergency treatment: epinephrine 0.01 ml/kg SC (maximum dose 0.3 ml) At risk of asphyxia and respiratory arrest
98
Goals of Asthma Management
Avoid exacerbation Avoid allergens Relieve asthmatic episodes promptly Relieve bronchospasm Monitor function with peak flow meter Self-management of inhalers, devices, and activity regulation Participate in sports/exercise when asthma controlled
99
Cystic Fibrosis
Exocrine gland dysfunction that produces multisystem involvement Most common lethal genetic illness among Caucasian children Approximately 3% U.S. Caucasian population are symptom-free carriers Autosomal recessive trait
100
What is the most reliable diagnostic procedure for cystic fibrosis?
Basis of the most reliable diagnostic procedure: sweat chloride test (pilocarpine iontophoresis) Normal sweat chloride is < 40 mEq/L A sweat chloride > than 60 mEq/L is diagnostic Extra salt and fluid required during hot weather
101
What is a normal sweat chloride ?
Normal sweat chloride is < 40 mEq/L
102
What does the level of sweat chloride have to reach in order to be diagnostic for cystic fibrosis?
A sweat chloride > than 60 mEq/L is diagnostic
103
Respiratory Manifestations of CF
Present in almost all CF patients but onset and extent are variable Stagnation of mucus and bacterial colonization result in destruction of lung tissue Tenacious secretions are difficult to expectorate, obstruct bronchi and bronchioles Decreased O2-CO2 exchange Results in hypoxia, hypercapnia, acidosis Compression of pulmonary blood vessels and progressive lung dysfunction lead to pulmonary hypertension, cor pulmonale, respiratory failure, and death
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Management of CF
Percussion and postural drainage Mucolytic agents Antibiotics – Vancomycin, Tobramycin Pancreatic enzymes Supplement fat soluble vitamins
105
Pulmozyme (dornase alfa)
Action: Enzyme that hydrolyzes the DNA in sputum Indications: Cystic fibrosis Nursing indications: --Given by nebulizer --Monitor for dysphonis and pharyngitis
106
CF Diet
Children with CF have an energy intake of 100% to 200% of standards for healthy persons High protein, high caloric, with unrestricted fat
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Azotorrhea =
increased protein in stool
108
Steatorrhea =
increased fat in stool – greasy stools
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Otitis Media
Otitis Media is primarily a result of a dysfunctioning eustachian tube.
110
AOM
Acute otitis media
111
OME
Otitis media with effusion
112
CSOM
Chronic suppurative otitis media
113
Mastoiditis
...
114
Risk factors for developing AOM or OME
Less than 2 years of age – peak incidence between 6 and 18 mo Atopy Bottle propping Chronic sinusitis Cleft palate Child care attendance Down syndrome Immunocompromising conditions
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Risk factors for developing AOM or OME “Passive smoking increases the risk of persistent middle ear effusion by enhancing attachment of the pathogens that cause otitis to the respiratory epithelium in the middle ear space, prolonging the inflammatory response, and impeding drainage through the eustachian tube.” True or false?
True
116
Signs and symptoms of AOM
Holding or pulling at ears Rolling head side to side Pain Fever Enlarged postauricular, cervical lymph glands
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Diagnosis of AOM
Acute onset of symptoms Evidence of middle ear effusion Signs and symptoms of middle ear inflammation
118
Treatment of AOM
Spontaneous resolution in 80% children Wait up to 72 hours for spontaneous resolution while providing pain control Amoxicillin 90 mg/kg/day divided bid for 10 days Second-line antibiotic: Augmentin (amoxicillin/clavulanate) Third-line: ceftriaxone (Rocephin)
119
What is the second line treatment for AOM?
Second-line antibiotic: Augmentin (amoxicillin/clavulanate)
120
What is the 1st line treatment for AOM?
Amoxicillin 90 mg/kg/day divided bid for 10 days
121
What is the 3rd line treatment for AOM?
Third-line: ceftriaxone (Rocephin)
122
True or False? Steroids, decongestants, and antihistamines are not recommended for treatment of AOM
True
123
True or False? Tonsillectomy does NOT reduce incidence of otitis
true
124
Nursing Care of AOM
Relief of pain ----Acetaminophen, Ibuprofen, acetaminophen with codeine ----Topical --topical benzocaine ----Local heat over ear while child lies on affected side
125
Management of OME
75% of cases resolved within 3 months Temporary hearing deficit Persistent OME past 3 months with hearing or language delays may benefit from tympanostomy tubes
126
Myringotomy and tubes
 “Placement of tympanostomy tubes is recommended after a total of 4 – 6 months of bilateral effusion with a bilateral hearing deficit.” True or False
True
127
Nursing Care For draining ears:
Sterile cotton in ears, still allowing drainage Moisture barrier on skin Speech and hearing evaluation
128
Nursing Care For tubes–
keep bath water and shampoo out of ears – ear plugs Speech and hearing evaluation
129
Pneumococcal immunization
Pneumococcal conjugate vaccine --PCV7-- and currently PCV 13 Reduced the number of otitis media cases dramatically S pneumoniae in 50% of children in daycare
130
Tonsillectomy
Post-op Nursing care: Observe for bleeding (7 – 10 da) Position side-lying Strict intake and output Oral fluids, or popcicles Nothing scratchy or salty
131
Pharyngitis
Gargle with warm saline three times a day Ice chips/popcicles Tylenol q 4 – 6 hours
132
Strep throat
Gargle with warm saline three times a day Ice chips/popcicles Tylenol q 4 – 6 hours Penicillin for 10 days
133
What is the difference in treatment for strep throat vs pharyngitis?
treatment is the same for both but strep throat requires Penicillin for 10 days.
134
Acute Streptococcal Pharyngitis
Treat with Penicillin – usually oral for 10 days -----Erythromycin is allergic to PCN May return to school or day care after have been taking antibiotics for a full 24 hour period Need to discard toothbrush and replace with new one after being on antibiotics for 24 hours